Poon Lok-man. / Thesis (M.Phil.)--Chinese University of Hong Kong, 2001. / Includes bibliographical references (leaves 126-128). / Abstracts in English and Chinese. / ABSTRACT --- p.ii / ACKNOWLEDGEMENT --- p.v / LIST OF TABLES --- p.vi / LIST OF FIGURES --- p.vii / GLOSSARY --- p.viii / Chapter CHAPTER 1: --- INTRODUCTION --- p.1 / Chapter CHAPTER 2: --- AIR POLLUTANTS AND ENVIRONMENTAL MONITORING --- p.8 / Chapter CHAPTER 3: --- PRINCIPLE OF LEAD FINGERPRINTING --- p.15 / Chapter CHAPTER 4: --- INSTRUMENTATION AND THEORY --- p.20 / Chapter CHAPTER 5: --- OPTIMIZATION OF ANALYTICAL PERFORMANCE IN LEAD ISOTOPE ANALYSIS --- p.29 / Chapter CHAPTER 6: --- DETERMINATION OF LEAD CONTENTS AND ISOTOPIC RATIOS IN POTENTIAL AIRBORNE LEAD SOURCES --- p.55 / Chapter CHAPTER 7: --- DETERMINATION OF LEAD CONTENT IN PARTICULATES COLLECTED ONTO HV-FILTERS --- p.69 / Chapter CHAPTER 8: --- ENVIRONMENTAL SURVEY --- p.74 / Chapter CHAPTER 9: --- "CONCLUSIONS, LIMITATIONS AND FURTHER DIRECTION" --- p.97 / APPENDICES --- p.105 / BIBLIOGRAPHY --- p.126

Chan Pui-Ling. / Thesis (M.Phil.)--Chinese University of Hong Kong, 2003. / Includes bibliographical references (leaves 169-182). / Text in English; abstracts in English and Chinese. / Chan Pui-Ling. / Acknowledgements --- p.i / Abstracts --- p.ii / Table of Contents --- p.v / List of Plates --- p.ix / List of Figures --- p.xii / List of Tables --- p.xiv / Abbreviations --- p.xviii / Chapter 1 --- Introduction --- p.1 / Chapter 1.1 --- Indoor Air Quality (IAQ): An overview --- p.1 / Chapter 1.1.1 --- Importance of indoor air quality --- p.2 / Chapter 1.1.2 --- Common indoor air pollutants --- p.2 / Chapter 1.1.3 --- Airborne bacteria --- p.4 / Chapter 1.1.3.1 --- Possible sources of airborne bacteria --- p.4 / Chapter 1.1.3.2 --- Health effects of the airborne bacteria --- p.5 / Chapter a. --- Sick building syndromes --- p.5 / Chapter b. --- Building-related illness --- p.7 / Chapter 1.1.4 --- Importance of studying airborne bacteria --- p.12 / Chapter 1.2 --- Situation in Hong Kong --- p.13 / Chapter 1.2.1 --- Outdoor air quality --- p.14 / Chapter 1.2.2 --- Indoor air quality --- p.14 / Chapter 1.2.2.1 --- Hong Kong studies --- p.16 / Chapter 1.2.3 --- Air quality objectives in Hong Kong --- p.18 / Chapter 1.3 --- Different sampling methods --- p.18 / Chapter 1.4 --- Identification of bacteria --- p.24 / Chapter 1.5 --- Site selection --- p.26 / Chapter 2 --- Objectives --- p.28 / Chapter 3 --- Materials and methods --- p.29 / Chapter 3.1 --- Samples collection --- p.29 / Chapter 3.1.1 --- Sampling site --- p.29 / Chapter 3.1.2 --- Complete Biosampler System --- p.29 / Chapter 3.1.3 --- Sampling preparation --- p.33 / Chapter 3.1.4 --- Sampling procedures --- p.33 / Chapter 3.2 --- Recovery of the airborne bacteria --- p.36 / Chapter 3.2.1 --- Cultural medium --- p.36 / Chapter 3.2.2 --- Recovery procedures --- p.36 / Chapter 3.2.3 --- Frozen stocks --- p.37 / Chapter 3.3 --- Indentification of bacterial strains --- p.37 / Chapter 3.3.1 --- Gram stain --- p.37 / Chapter 3.3.1.1 --- Chemical reagents --- p.37 / Chapter 3.3.1.2 --- Gram stain procedures --- p.38 / Chapter 3.3.2 --- Oxidase test --- p.38 / Chapter 3.3.2.1 --- Chemical reagents --- p.38 / Chapter 3.3.2.2 --- Oxidase test procedures --- p.41 / Chapter 3.3.3 --- Midi Sherlock® Microbial Identification System (MIDI) --- p.41 / Chapter 3.3.3.1 --- Culture medium --- p.41 / Chapter 3.3.3.2 --- Chemical reagents --- p.41 / Chapter 3.3.3.3 --- MIDI procedures --- p.41 / Chapter 3.3.4 --- Biolog MicroLogTM system (Biolog) --- p.41 / Chapter 3.3.4.1 --- Culture medium --- p.41 / Chapter 3.3.4.2 --- Chemical reagents --- p.44 / Chapter 3.3.4.3 --- Biolog procedures --- p.44 / Chapter 3.3.5 --- DuPont Qualicon RiboPrinter® Microbial Characterization System (RiboPrinter) --- p.46 / Chapter 3.3.5.1 --- Culture medium --- p.46 / Chapter 3.3.5.2 --- Chemical reagents --- p.46 / Chapter 3.3.5.3 --- RiboPrinter procedures --- p.46 / Chapter 4 --- Results --- p.50 / Chapter 4.1 --- Sample naming system --- p.50 / Chapter 4.2 --- Interpretation of results --- p.50 / Chapter 4.2.1 --- Midi Sherlock® Microbial Identification System (MIDI) --- p.51 / Chapter 4.2.2 --- Biolog MicroLog´ёØ System (Biolog) --- p.51 / Chapter 4.2.3 --- DuPont Qualicon RiboPrinter® Microbial Characterization System (RiboPrinter) --- p.52 / Chapter 4.3 --- Sample results --- p.53 / Chapter 4.3.1 --- Sample 1 (Spring) --- p.53 / Chapter 4.3.2 --- Sample 2 (Summer-holiday) --- p.62 / Chapter 4.3.3 --- Sample 3 (Summer-school time) --- p.71 / Chapter 4.3.4 --- Sample 4 (Autumn) --- p.81 / Chapter 4.3.5 --- Sample 5 (Winter) --- p.90 / Chapter 4.4 --- Bacterial profile of the student canteen --- p.100 / Chapter 4.5 --- The cell and colony morphology of the dominant bacteria --- p.100 / Chapter 4.6 --- Comparison between samples --- p.121 / Chapter 4.6.1 --- Spatial variation --- p.121 / Chapter 4.6.1.1 --- Spatial effect on bacterial abundance --- p.121 / Chapter 4.6.1.2 --- Spatial effect on species diversity --- p.121 / Chapter 4.6.2 --- Daily variation --- p.126 / Chapter 4.6.2.1 --- Daily effect on bacterial abundance --- p.126 / Chapter 4.6.2.2 --- Daily effect on species diversity --- p.126 / Chapter 4.6.3 --- Seasonal variation --- p.126 / Chapter 4.6.3.1 --- Seasonal effect on bacterial abundance --- p.126 / Chapter 4.6.3.2 --- Seasonal effect on species diversity --- p.130 / Chapter 4.7 --- Temperature effect on individual airborne bacterial population --- p.130 / Chapter 4.7.1 --- Gram positive bacteria --- p.130 / Chapter 4.7.2 --- Gram negative bacteria --- p.130 / Chapter 4.8 --- Effect of relative humidity on individual airborne bacterial population --- p.137 / Chapter 4.8.1 --- Gram positive bacteria --- p.137 / Chapter 4.8.2 --- Gram negative bacteria --- p.137 / Chapter 5 --- Discussion --- p.143 / Chapter 5.1 --- Bacterial profile --- p.143 / Chapter 5.1.1 --- Bacterial diversity --- p.143 / Chapter 5.1.2 --- Information of the identified bacteria from the student canteen --- p.144 / Chapter 5.1.3 --- Pathogenicity --- p.153 / Chapter 5.1.4 --- Summary on the bacterial profile --- p.153 / Chapter 5.2 --- Comparison between samples --- p.160 / Chapter 5.2.1 --- Spatial variation (Sampling point 1 against Sampling point 2) --- p.160 / Chapter 5.2.2 --- Daily variation (Morning against Afternoon) --- p.161 / Chapter 5.2.3 --- Seasonal variation --- p.162 / Chapter 5.2.4 --- Summer holiday against Summer school time --- p.163 / Chapter 5.2.5 --- Summary on the factors affecting the bacterial content --- p.164 / Chapter 5.3 --- Summary on indoor air quality of the student canteen in terms of bacterial level. --- p.166 / Chapter 6 --- Conclusions --- p.168 / Chapter 7 --- References --- p.169 / Appendix 1 --- p.183 / Appendix 2 --- p.187

published_or_final_version / Urban Design / Master / Master of Urban Design

published_or_final_version / Environmental Management / Master / Master of Science in Environmental Management

published_or_final_version / Environmental Management / Master / Master of Science in Environmental Management

Lau Wing-Ngar Vivian. / Thesis (M.Phil.)--Chinese University of Hong Kong, 2002. / Includes bibliographical references (leaves 135-144). / Abstracts in English and Chinese. / Acknowledgements --- p.i / Abstract --- p.ii / Abbreviations --- p.vi / List of figures --- p.viii / List of tables --- p.xi / Contents --- p.xiii / Chapter 1 --- Introduction --- p.1 / Chapter 1.1 --- General introduction --- p.1 / Chapter 1.2 --- Roadside air pollution worldwide and in Hong Kong --- p.2 / Chapter 1.2.1 --- Air quality in Hong Kong --- p.3 / Chapter 1.3 --- Characteristics of particulate matter --- p.9 / Chapter 1.4 --- Composition and sources of particulate matter --- p.11 / Chapter 1.5 --- Toxic effects of particulate matter --- p.12 / Chapter 1.5.1 --- Lung injury --- p.12 / Chapter 1.5.2 --- Cardiovascular injury --- p.15 / Chapter 1.5.3 --- Mutagenesis and carcinogenesis --- p.16 / Chapter 1.6 --- Aims of my study --- p.16 / Chapter 2 --- Toxic Effects of Heavy Metals Ions on Selected Cultured Cell-lines --- p.18 / Chapter 2.1 --- Introduction --- p.18 / Chapter 2.1.1 --- Metals --- p.18 / Chapter 2.1.1.1 --- Cadmium --- p.22 / Chapter 2.1.1.2 --- Chromium --- p.23 / Chapter 2.1.1.3 --- Lead --- p.25 / Chapter 2.1.1.4 --- Zinc --- p.26 / Chapter 2.1.2 --- Metallothioneins --- p.28 / Chapter 2.1.3 --- p53 --- p.31 / Chapter 2.1.4 --- Tumor Necrosis Factor-alpha (TNF-α) --- p.32 / Chapter 2.1.5 --- Aims of this chapter --- p.32 / Chapter 2.2 --- Materials and methods --- p.35 / Chapter 2.2.1 --- Reagents --- p.35 / Chapter 2.2.2 --- Cultured Cell lines --- p.35 / Chapter 2.2.2.1 --- PU5-18 --- p.36 / Chapter 2.2.2.2 --- LL24 --- p.36 / Chapter 2.2.2.3 --- HBE4-E6/E7 --- p.37 / Chapter 2.2.3 --- Cytotoxicity assays --- p.37 / Chapter 2.2.4 --- ELISA assays --- p.40 / Chapter 2.2.4.1 --- ELISA assay ofp53 levels --- p.41 / Chapter 2.2.4.2 --- ELISA assay of TNF-α levels --- p.43 / Chapter 2.2.5 --- MT gene expression studies by Luciferase assay --- p.44 / Chapter 2.2.5.1 --- PCR amplification --- p.44 / Chapter 2.2.5.2 --- 5´ة End modification of PCR amplified DNA --- p.44 / Chapter 2.2.5.3 --- Ligation of DNA fragment to linearized vector --- p.46 / Chapter 2.2.5.4 --- E. coli. transformation by heat shock --- p.46 / Chapter 2.2.5.5 --- PCR sequencing --- p.47 / Chapter 2.2.5.6 --- Transfection of plasmid into HBE4-E6/E7 cells --- p.49 / Chapter 2.2.5.7 --- Data analysis --- p.50 / Chapter 2.3 --- Results and discussion --- p.51 / Chapter 2.3.1 --- Cytotoxicity assays --- p.51 / Chapter 2.3.2 --- Combination effects of metals on cytotoxicity --- p.61 / Chapter 2.3.3 --- p53 --- p.65 / Chapter 2.3.4 --- TNF-α --- p.68 / Chapter 2.3.5 --- MT gene expression studies by Luciferase assay --- p.69 / Chapter 2.4 --- Conclusion --- p.74 / Chapter 3 --- Effects of Polycyclic Aromatic Hydrocarbons (PAHs) on Cultured Cell-lines --- p.75 / Chapter 3.1 --- Introduction --- p.75 / Chapter 3.2 --- Materials and methods --- p.79 / Chapter 3.2.1 --- Reagents --- p.79 / Chapter 3.2.2 --- Cell culture --- p.79 / Chapter 3.2.3 --- AlamarBlue assay --- p.80 / Chapter 3.2.4 --- EROD assay --- p.80 / Chapter 3.3 --- Results and discussion --- p.84 / Chapter 3.4 --- Conclusion --- p.88 / Chapter 4 --- Chemical and Biological Assays on Roadside Dust --- p.89 / Chapter 4.1 --- Introduction --- p.89 / Chapter 4.1.1 --- Composition of particulate matter in Hong Kong --- p.89 / Chapter 4.1.2 --- Metal contents of particulate matter in Hong Kong --- p.91 / Chapter 4.1.3 --- Possible adverse health impacts of particulate matter --- p.94 / Chapter 4.1.3.1 --- In vitro studies using different cell models --- p.94 / Chapter 4.1.3.2 --- In vivo studies using rodents --- p.97 / Chapter 4.1.3.3 --- Epidemiological studies --- p.98 / Chapter 4.1.4 --- Aims of this chapter --- p.100 / Chapter 4.2 --- Materials and methods --- p.101 / Chapter 4.2.1 --- Sampling of roadside dust --- p.101 / Chapter 4.2.2 --- Chemical analysis of roadside dust --- p.104 / Chapter 4.2.2.1 --- Reagents --- p.104 / Chapter 4.2.2.2 --- Total metal contents --- p.105 / Chapter 4.2.2.3 --- Extractable metal contents --- p.105 / Chapter 4.2.3 --- Biological assays --- p.105 / Chapter 4.2.3.1 --- Cell models --- p.106 / Chapter 4.2.3.2 --- Pretreatment of roadside dust --- p.106 / Chapter 4.2.3.3 --- AlamarBlue assay --- p.106 / Chapter 4.2.3.4 --- ELISA assays --- p.108 / Chapter 4.2.3.5 --- Luciferase assay --- p.108 / Chapter 4.3 --- Results and discussion --- p.110 / Chapter 4.3.1 --- Total metal contents --- p.110 / Chapter 4.3.2 --- Extractable metal contents --- p.113 / Chapter 4.3.3 --- AlamarBlue assay --- p.116 / Chapter 4.3.4 --- p53 --- p.122 / Chapter 4.3.5 --- TNF-α --- p.122 / Chapter 4.3.6 --- Luciferase assay --- p.126 / Chapter 4.4 --- Conclusion --- p.129 / Chapter 5 --- General discussion and conclusion --- p.130 / Chapter 6 --- References --- p.135

Positive associations between short-term exposure to ambient particulate matter (PM) pollution and cardio-respiratory morbidity and mortality have been established in epidemiologic studies. However, scientific uncertainties remain regarding which PM constituents and sources are most harmful to the exposed population. While tentative evidence of positive links between certain PM constituents and sources with specific health outcomes exists, significant heterogeneity in study findings remains. The chemical composition and emission source of air pollution vary not only temporally, but also geographically. Thus, substantial research on these characteristics under different atmospheres (e.g., Asian atmosphere) is warranted to enhance our understanding of PM-related health effects. / In Hong Kong, air pollution levels often exceed the World Health Organization’s air quality guidelines, posing serious public health threat. Although two decades of active research have associated PM mass (weights) concentration in Hong Kong with elevated risk of daily mortality and emergency hospital admissions, individual chemical constituents and sources responsible for the adverse health effects associated with PM mass have rarely been examined. This thesis attempted to reduce the current scientific uncertainty by making use of the speciation data for PM with aerodynamic diameter ≤ 10 μm (PM₁₀) to examine the associations between chemical constituents and daily cardio-respiratory emergency hospital admissions in Hong Kong between 2001 and 2008. It also capitalized on Positive Matrix Factorization (PMF) source apportionment model to create an alternative measure of PM concentrations that quantified the relative contribution of PM₁₀ sources. This enabled the assessment of health risks associated with exposure to particle mixture from specific sources. / Time-series analyses conducted in this work showed evidence of positive links of emergency hospitalizations with multiple PM₁₀ constituents for various exposure lags examined. In multipollutant models adjusting for gaseous co-pollutants, three groups of constituents were significantly associated with increased risk of cause-specific hospitalizations. They included: combustion-related constituents (i.e., elemental carbon, organic matter, potassium ion, manganese, nitrate ion, arsenic, lead), sea salt-related constituents (i.e., sodium ion, chloride ion, magnesium), and constituents related to soil/road dust (i.e., aluminum, iron). Significant associations were most evident between November to April. PMF model identified eight PM₁₀ sources: 1) vehicle exhaust, 2) soil/road dust, 3) regional combustion, 4) residual oil, 5) fresh sea salt, 6) aged sea salt, 7) secondary nitrate, and 8) secondary sulfate. Health effect analysis demonstrated that all PM₁₀ sources, except fresh sea salt, were positively associated with emergency hospitalizations. Combustion-related sources were positively associated with risk of cardiovascular hospitalizations at shorter cumulative lags than with respiratory hospitalization. Sources that primarily generate coarser particles (i.e., soil/road dust and aged sea salt) were linked to respiratory hospitalizations at shorter cumulative lags than combustion sources that emit fine particles, which were associated with respiratory hospitalization at delayed cumulative lags. / This work may help prioritize future toxicological research on the biologic mechanisms linking PM pollution to cardio-respiratory health. It also stresses the importance of regulation and reduction of traffic and other combustion-related emissions, reconfiguration of urban environment to reduce personal exposure to traffic emissions, as well as establishment of a coordinated and robust regional-scale air quality management plan. Constituent- and source-based air quality standards and policy strategies should be considered, supplementary to standards for total PM mass (e.g., PM₁₀ and PM₂.₅), to effectively protect the population from air pollution mixture. For example, policy measures aiming at controlling anthropogenic sources of coarse particles (e.g., soil/road dust, precursor gases for atmospheric conversion of aged sea salt) should be advisable. / 以往流行病學研究已經建立大氣懸浮顆粒（PM）污染短期暴露和心血管﹑呼吸系統疾病的發病率以及死亡率之間的關聯，但仍然不能確定究竟那種PM化學成分和排放源對暴露人群最有害。初步證據已經顯示了個別PM化學成分和排放源與某些疾病的相關聯，但是，現今研究結果之間依然存在顯著差異。此外，顆粒污染物的化學成分和排放源會隨地理空間不同而變化，因此有必要在不同地域的大氣環境下（如亞洲）進行研究，從而提高我們對PM影響健康的認識。 / 香港的空氣質量常超過世界衛生組織規定的可接受空氣質量標準，空氣污染已經成為一個嚴重的公共衛生問題。香港二十年的空氣污染研究已顯示PM質量濃度會增加死亡率和緊急入院的風險，然而仍然缺乏PM化學成分和排放源對健康影響的研究。本港環境保護署擁有十多年連續測量的可吸入懸浮粒子（PM₁₀）成分數據，因此本論文利用這數據與公共衛生數據來填補這個學術空白。具體的，本論文探索香港2001年至2008年間的PM化學成分與期間每日心血管和呼吸系統緊急入院的關聯。本論文還利用正交矩陣因子分解法（PMF），即大氣顆粒物的源解析技術，進行顆粒源分離，已建立替代單元以分別代表PM₁₀不同排放源的相對貢獻建立一個替代措施量化PM₁₀的成分中不同來源相對的貢獻，最終評估暴露於特定來源的PM₁₀混合成分的健康風險。 / 本文中的時間序列分析研究結果表明緊急入院率與多個PM化學成分在不同的暴露滯後時間上均存在正關聯。本論文的多種污染物時間序列的模型對氣體的共污染性進行了控制，研究發現以下三种顆粒物對某些具體疾病的緊急入院風險有顯著增加︰燃燒有關的顆粒（即︰碳元素，有機物，鉀離子，錳，硝酸根離子，砷，鉛），與海鹽有關的顆粒（即︰鈉離子，氯離子，鎂），以及與土壤/道路粉塵有關的顆粒（即︰鋁，鐵），這種相關性在寒冷的季節更為明顯（即每年的11月至4月）。PMF鑑定了八個PM₁₀的排放源或排放組合，包括︰1）汽車排氣，2）土壤/道路粉塵，3）區域燃燒產物，4）渣油，5）鮮海鹽，6）陳海鹽，7）二次硝酸鹽，和8）二次硫酸鹽。分析發現，除鮮海鹽外，其他PM₁₀的排放源與緊急入院呈正關係。研究顯示與燃燒有關的排放源能增加即時因心血管疾病緊急入院的風險，而主要含有粗顆粒的排放源能增加即時呼吸系統疾病緊急入院的風險，且在作用時間上比燃燒排放源（主要含有細顆粒）的危害更快。 / 本論文所提供的研究成果有助於重新定位未來有關空氣污染與心肺健康的生物學機制毒理學研究方向。研究結果強調監管和降低交通以及其他有關燃燒排放的重要性，利用合理城市環境結構及配置來減少居民暴露交通空氣污染的風險，以及建立區域性的空氣質量管理計劃。此外，本研究還建議除了PM質量濃度（如PM₁₀和PM₂.₅）的空氣質量標準以外，針對PM化學成分和排放源的空氣質量標準也應加以考慮，以有效地保護居民免受空氣污染的危害，例如，應考慮有關粗顆粒（如土壤/道路麈和前驅氣體）的人為來源的政策措施。 / Pun, Chit. / Thesis Ph.D. Chinese University of Hong Kong 2014. / Includes bibliographical references (leaves 108-133). / Abstracts also in Chinese. / Title from PDF title page (viewed on 18, October, 2016). / Pun, Chit. / Detailed summary in vernacular field only. / Detailed summary in vernacular field only. / Detailed summary in vernacular field only. / Detailed summary in vernacular field only.

There are two parts in this study. The first part is to test the validity of the assumption of thermodynamic equilibrium between fine particulate (PM2.5) nitrate and ammonium and gas-phase nitric acid (HNO3(g)) and ammonia (NH3(g)). A rough estimation of the characteristic time to achieve thermodynamic equilibrium is first carried out, which suggests that PM2.5 and gas-phase species are in thermodynamic equilibrium. Then equilibrium is tested by calculating the equilibrium concentrations of HNO3(g) and NH3(g) implied by the PM2.5 inorganic composition, temperature and relative humidity observed at the Atlanta Supersite 1999 using ISORROPIA model. The second part of this study is to analyze the ground-level ozone pollution precursor relationships in Hong Kong area. Characteristics of O3 precursors are explored. Trace gases NO and CO, VOCs, absorption coefficient, temperature and solar radiation are associated with the O3 formation. Specific VOC and VOC-sources that contribute most to the formation of photochemical smog are identified. The accuracy of pollutant emission inventories for Hong Kong and PRD region is also assessed. Combined with back trajectory information, dCO/dNOy is used to define whether O3 is locally or regionally occurred. An OBM is used to investigate the relative benefits of various emission-control strategies. Generally the formation of O3 throughout much of Hong Kong area is limited by VOC, in which reactive aromatics are dominant.

Kam Wai-ming. / Thesis (M.Phil.)--Chinese University of Hong Kong, 2001. / Includes bibliographical references (leaves 153-162). / Abstracts in English and Chinese ; questionnaire in Chinese. / List of Tables --- p.x / List of Figures --- p.xiii / List of Abbreviation --- p.xvi / Chapter Chapter One --- Introduction --- p.1 / Chapter 1.1 --- Introduction --- p.1 / Chapter 1.2 --- The Research Problems --- p.2 / Chapter 1.3 --- Objectives of the Study --- p.4 / Chapter 1.4 --- Study Area --- p.4 / Chapter 1.5 --- Significance of the Study --- p.8 / Chapter 1.6 --- Organization of Thesis --- p.9 / Chapter Chapter Two --- Literature Review --- p.11 / Chapter 2.1 --- Pedestrianization: Basic Ideas and History --- p.11 / Chapter 2.1.1 --- Definition of Pedestrianization --- p.11 / Chapter 2.1.2 --- Motivation of pedestrianization --- p.13 / Chapter 2.1.3 --- Learning from the Development of Pedestrianization in Other Cities --- p.15 / Chapter 2.1.4 --- Impacts of Pedestrianization on Environment --- p.19 / Chapter 2.2 --- Pedestrianization in Hong Kong --- p.21 / Chapter 2.2.1 --- Development --- p.21 / Chapter 2.2.2 --- Ways for Successful Pedestrianization in Hong Kong --- p.22 / Chapter 2.3 --- Human Perception and Response on Air Pollution and Pedestrianization --- p.24 / Chapter 2.3.1 --- Introduction of Human Perception --- p.24 / Chapter 2.3.2 --- Human Perception of Air Pollution --- p.27 / Chapter 2.3.3 --- Changes of Human Perception in Response to the Changes of Environmental Quality After Pedestrianization --- p.31 / Chapter Chapter Three --- Methodology --- p.34 / Chapter 3.1 --- Research Design --- p.34 / Chapter 3.2 --- BACIPR Approach --- p.35 / Chapter 3.3 --- Objective Assessment of the influence of pedestrianization on air quality --- p.38 / Chapter 3.3.1 --- Model Prediction --- p.39 / Chapter 3.3.2 --- Physical Measurement of Particulates --- p.46 / Chapter 3.4 --- Subjective Assessment: Human Perception of Roadside Air Pollution --- p.56 / Chapter 3.4.1 --- Guidelines Used for Subjective Assessment --- p.57 / Chapter 3.4.2 --- Development of the Questionnaire --- p.58 / Chapter 3.4.3 --- Statistical Analysis --- p.59 / Chapter Chapter Four --- Model Simulation of the Effect of Pedestrianization on Air Quality --- p.61 / Chapter 4.1 --- Introduction --- p.61 / Chapter 4.2 --- Air Quality in Causeway Bay Before Pedestrianization --- p.64 / Chapter 4.2.1 --- Overall Spatial Variation of Air Quality --- p.64 / Chapter 4.2.2 --- Respiratory Suspended Particulate (RSP) --- p.67 / Chapter 4.2.3 --- Nitrogen Dioxide (N02) --- p.68 / Chapter 4.2.4 --- Carbon Monoxide (CO) --- p.69 / Chapter 4.3 --- Air Quality in Causeway Bay After Pedestrianization --- p.70 / Chapter 4.3.1 --- Overall Spatial Variation of Air Quality --- p.70 / Chapter 4.3.2 --- Respiratory Suspended Particulate (RSP) --- p.70 / Chapter 4.3.3 --- Nitrogen Dioxide (N02) --- p.73 / Chapter 4.3.4 --- Carbon Monoxide (CO) --- p.75 / Chapter 4.3.5 --- Effect of Government's Pedestrianization Scheme in Improving Air Quality --- p.77 / Chapter 4.4 --- Air Quality Impact of Pedestrianization in Single Street --- p.77 / Chapter 4.4.1 --- RSP Concentration in Russell Street Section Before Pedestrianization --- p.78 / Chapter 4.4.2 --- RSP Concentration in Russell Street Section After Pedestrianization --- p.81 / Chapter 4.5 --- Summary and Conclusion --- p.83 / Chapter Chapter Five --- Measurement of Particulate Pollution in Causeway Bay --- p.84 / Chapter 5.1 --- Change in Particulate Pollution After Pedestrianization in Russell Street --- p.86 / Chapter 5.1.1 --- Overall Changes in Particulate Pollution after Pedestrianization --- p.86 / Chapter 5.1.2 --- Changes in Particulate Pollution after Pedestrianization under Different Weather Conditions --- p.87 / Chapter 5.1.2.1 --- Effects of Pedestrianization on Fine Day --- p.88 / Chapter 5.1.2.2 --- Effects of Pedestrianization on Rainy Days --- p.89 / Chapter 5.1.3 --- Changes of Particulate Pollution after Pedestrianization under Different Traffic Flow Conditions --- p.90 / Chapter 5.1.3.1 --- Effects of Pedestrianization during Peak Hours --- p.91 / Chapter 5.1.3.2 --- Effects of Pedestrianization during Non-Peak Hours --- p.92 / Chapter 5.1.4 --- Changes in Particulate Pollution As a result of Pedestrianization on Different Days of the Week --- p.93 / Chapter 5.1.4.1 --- Effects of Pedestrianization on Weekdays --- p.94 / Chapter 5.1.4.2 --- Effects of Pedestrianization on Non-Weekdays --- p.94 / Chapter 5.1.5 --- Change in PM 10 I/C ratio After Pedestrianization --- p.95 / Chapter 5.2 --- Variations in Particulate Pollution Characteristics Due to Different Pedestrianization Street Designs --- p.96 / Chapter 5.2.1 --- General Contrasts between an Open and Semi-enclosed Street --- p.97 / Chapter 5.2.2 --- Seasonal Effect on Particulate Pollution in Jardine's Crescent and in Its Control Street --- p.97 / Chapter 5.2.3 --- Climatic Effects on Particulate Pollution --- p.101 / Chapter 5.2.4 --- Effects of Traffic Conditions on Particulate Pollution Pattern in Jardine's Crescent and in Its Control Street --- p.102 / Chapter 5.2.5 --- Effects of Day of the Week on Particulate Pollution --- p.104 / Chapter 5.3 --- The Variation of Particulate Pollution in Causeway Bay --- p.105 / Chapter 5.3.1 --- Spatial Variation --- p.105 / Chapter 5.3.2 --- Seasonal Variation --- p.108 / Chapter 5.3.3 --- Rain as a Cleaning Agent --- p.111 / Chapter 5.4 --- Summary and Conclusion --- p.112 / Chapter Chapter Six --- Pedestrianization and Perception of Air Quality --- p.113 / Chapter 6.1 --- Introduction --- p.113 / Chapter 6.2 --- Effect of Pedestrianization in Improving the Perceived Air Quality --- p.115 / Chapter 6.2.1 --- Overall Changes of PAQ after Pedestrianization --- p.115 / Chapter 6.2.2 --- Changes in Perceived Air Quality As a Result of Pedestrianization --- p.118 / Chapter 6.2.2.1 --- Correlation between PAQ and AQ --- p.118 / Chapter 6.2.2.2 --- Difference in Dose-Response Relationships before and after Pedestrianization --- p.120 / Chapter 6.2.3 --- Summary of Findings on Human Perception --- p.136 / Chapter 6.3 --- Aspects of Pedestrianization Which Improve Perceived Air Quality --- p.136 / Chapter 6.3.1 --- Behavior Constraint Model --- p.137 / Chapter 6.3.2 --- Environmental Stress Model --- p.139 / Chapter 6.3.3 --- Perception of Air Pollution Through Smell and Vision --- p.141 / Chapter 6.4 --- Summary and Conclusion --- p.144 / Chapter Chapter Seven --- Conclusion --- p.146 / Chapter 7.1 --- Summary of Findings --- p.146 / Chapter 7.1.1 --- Model Simulated Air Pollution Levels in Causeway Bay and Possible Effects of the Government Pedestrianization Scheme --- p.147 / Chapter 7.1.2 --- Changes in the Pattern and Characteristics of Particulate Pollution after Pedestrianization --- p.148 / Chapter 7.1.3 --- Effects of Pedestrianization on Human Perception of Air Quality --- p.149 / Chapter 7.2 --- Discussion of Findings --- p.151 / References --- p.153 / Appendix A --- p.163