To be a fat woman is to experience a prolonged, personal battle with the body. The battle is enacted in a social context which is the site of remarkable consensus about the personal culpability of fat people for their bodily largeness; for women in particular the sanctions are especially powerful. In this research nine large women have engaged in a prolonged dialogue about the experience of being 'obese'. In the course of a feminist research endeavour, with a researcher who is similarly positioned, they have both contributed to and gained from a project which illuminates the experience of largeness alongside a critical examination of the discourses which shape body size.This dissertation critiques a dominant medical discourse which ignores conflicting research and supports a narrow view of health by simplistically linking increased body weight with poor health outcomes. Such is the hegemonic power of medicine that an examination of both nursing and popular literature in the area of study, reveals wide-spread acceptance of the notion that to be thin is to be healthy and virtuous, and to be fat is to be unhealthy and morally deficient. For nursing, the unquestioning obedience to medical teaching, raises serious questions about nursing's autonomy and separateness from medicine. Nurses have perpetuated an unhelpful and reductionist approach to their care of large women, in direct contradiction to nursing's supposed allegiance to a holistic approach to health care. Current strictures on women's body size and continued support for reduction dieting leave large women with the choice between two binary opposites; to diet or not to diet. Either choice has consequences which are traumatic and not health promoting. The experience of largeness emerges as a socially constructed disability in which many women are denied the opportunity to be fully healthy.

Thesis (Ph.D.)--University of Wollongong, 2004. / Typescript. Includes bibliographical references: p. 160-170.

Thesis (Ph. D.)--Auburn University. / Abstract. Vita. Includes bibliographical references (p. 136-145).

Thesis--Auburn University, 2009. / Abstract. Vita. Includes bibliographical references (p. 91-101).

Thesis (M.A.)--Eastern Illinois University, 2009. / Includes bibliographical references (leaves 50-57).

Thesis (Ph. D.)--Oregon State University, 1995. / Typescript (photocopy). Includes bibliographical references. Also available on the World Wide Web.

Thesis (M.S.)--Kutztown University of Pennsylvania, 1996. / Source: Masters Abstracts International, Volume: 45-06, page: 2891. Typescript. Abstract precedes second title page as preliminary pages 2-3. Includes bibliographical references (leaves 28-31).

The effects of intrinsic traits on the diversification dynamics have been extensively investigated, with several traits being associated with increase in diversification. On the other hand, the possible negative effects of traits on the diversification of a lineage have been for the most part overlooked. Here we used both the fossil record and molecular data to study the diversification dynamics of Placentalia, focusing on the orders in decline of diversity, and investigated different mechanisms that might control the evolutionary success of the 21 placental orders. More specifically we: 1- determined which of the 21 orders of Placentalia are in decline of diversity (i.e., Decline model); 2- investigated whether the Decline model has a phylogenetic signal; 3- tested the hypothesis that the differences in body size are related to the Decline model; 4- tested the hypothesis that the orders in Decline have lower morphological disparity; 5- investigated whether the orders in decline of diversity, inferred from the fossil record, are the ones with higher extinction risk nowadays. Our analysis indicate that the majority of the orders of placental mammals have a pattern consistent with the Decline model and, although the Decline model is not equally distributed among the placental superorders, there was no significant phylogenetic signal for the orders in diversity decline. We found a positive correlation between the Decline model and the average body size which is in line with previous studies on body size evolution. We argue that such results suggest a complex evolutionary dynamics: larger body size appears to be an evolutionary attractor with lineages showing a tendency to increase in size, however, the increase in body size would be counterbalanced by a higher propensity to Decline. Moreover, we found a the negative correlation between the Decline model and morphological variation. We suggest that such results could indicate two possible scenario: (i) the low morphological variation would cause lineages to loose diversity; (ii) the low morphological variation would be the product of decrease in diversity through extinction selectivity. Finally, we found no correlation between the extinction risk of extant species and the deep time diversity decline, which suggests that the drivers of the current and the past Decline are not the same / O efeito de caracteres intrínsecos na dinâmica de diversificação foram extensamente investigados e diversos caracteres foram associados com aumentos na diversificação. Contudo, os possíveis efeitos negativos de um caractere sobre a diversificação de uma linhagem foram em grande parte ignorados. No presente trabalho integramos o registro fóssil com dados moleculares para estudar a dinâmica de diversificação de Placentalia, focando nas ordens em declínio de diversidade, e investigamos possíveis mecanismos responsáveis por gerar os padrões de diversificação encontrados. Mais especificamente nós: 1- determinamos quais das 21 ordens de Placentalia estão em declínio de diversidade (i.e., Declínio); 2- investigamos se o Declínio apresenta um sinal filogenético; 3- testamos a hipótese de que o tamanho do corpo está relacionado com o Declínio; 4- testamos a hipótese de que as ordens em Declínio possum menor disparidade morfológica; 5- investigamos se as ordens em Declínio, inferido a partir do registro fóssil, são as mesas com maior risco de extinção na atualidade. Nossas análises indicam que a maioria das ordens de mamíferos placentários apresentam um signal consistente com o Declínio e, embora o Declínio não esteja igualmente distribuído entre as superorderns de Placentalia, não há um signal filogenético significativo para as ordens em Declínio. Nossos resultados indicam uma correlação positiva entre o Declínio e o tamanho corporal médio de cada ordem que está de acordo com estudos prévios sobre evolução do tamanho do corpo. Argumentamos que estes resultados sugerem uma dinâmica de evolução complexa: tamanho corpóreo grande seria um atrator evolutivo que gera a tendência das linhagens aumentarem de tamanho, todavia, o aumento do tamanho do corpo seria contrabalançado pela maior susceptibilidade ao Declínio. Outrossim, encontramos uma correlação negativa entre o Declínio e a variação morfológica. Argumentamos que essa correlação poderia indicar dois possíveis cenários: (i) a baixa variação morfológica seria responsável pela redução no número de linhagens e tornaria as ordens mais susceptíveis ao declínio de diversidade; (ii) a baixa variação morfológica teria sido gerada pela diminuição da diversidade. Por último, o risco de extinção das espécies atuais não está correlacionado com o Declínio, o que sugere que os mecanismos responsáveis pelo Declínio no passado e no presente não são os mesmos.

The four elements of the lower hind foot (calcaneus, metatarsal, naviculo-cuboid, and tibia) were tested for use as age-independent proxies of body size in white-tailed deer using known aged specimens from Ft. Hood Texas. Statistical analysis indicates that the calcaneum and the tibia are good proxies of age-independent body size in white-tailed deer. In addition to expanding the list of elements that can be used for studies of age-independent body size, these elements can also be used to age faunal remains to an ordinal scale of juveniles and adults. This is useful for research regarding prehistoric prey populations; as a single element can be used to determine prey body size and age simultaneously, which are the two variables used to assess changes in human subsistence practices via the archaeological remains of their prey.

Freshwater cyclopoid copepods exhibit at least a fivefold range in somatic genome size and a mechanism, chromatin diminution, which could account for much of this interspecific variation. These attributes suggest that copepods are well suited to studies of genome size evolution. We tested the nucleotypic hypothesis of genome size evolution, which poses that variation in genome size is adaptive due to the 'bulk' effects of both coding and noncoding DNA on cell size and division rates, and their correlates. We found a significant inverse correlation between genome size and developmental (growth) rate in five freshwater cyclopoid species at three temperatures. That is, species with smaller genomes developed faster. Species with smaller genomes had significantly smaller bodies at 22°C, but not at cooler and warmer temperatures. Species with smaller genomes developed faster at all three temperatures, but had smaller bodies only at 22°C. We propose a model of life history evolution that adds genome size and cell cycle dynamics to the suite of characters on which selection may act to mold life histories and to influence the distribution of traits among different habitats.