Spelling suggestions: "subject:"debinding"" "subject:"4ebinding""
161 |
Signalling pathways implicated in the growth factor and cytokine mediated up regulation of gelantinase B, collagenase 1 and stromelysin-1 in rabbit aortic smooth muscle cells in vitroHussain, Shaista January 2000 (has links)
No description available.
|
162 |
Characterisation of the native rat GABAâ†B receptor and the recombinant GABAâ†B receptor transiently expressed in COS cellsKeir, Miranda J. January 1999 (has links)
No description available.
|
163 |
An investigation into the molecular basis of substrate specificity in lactate dehydrogenaseHart, K. W. January 1989 (has links)
No description available.
|
164 |
Recognition of cell wall analogues by glycopeptide antibioticsGroves, Patrick January 1994 (has links)
No description available.
|
165 |
Biochemical properties of caldesmon.Abougou, Jean-Claude January 1988 (has links)
An attempt to develop a short and reliable method of caldesmon purification led to the development of three procedures of caldesmon purification. The first method was seldom used because of its low yield and the lack of caldesmon endogenous kinase activity. However, it allowed us to purify MLCK (myosin light chain kinase). The second and third methods gave respectively, a caldesmon sample with and without kinase activity. We were able to localize the endogenous kinase in the 0-30% ammonium sulfate precipitated DEAE pellet but we were unsuccessful at purifying the kinase to homogeneity. We found that caldesmon can also be phosphorylated by rat brain Ca²⁺-calmodulin-dependent kinase II at sites identical to those of caldesmon endogenous kinase but different to those of kinase C. In addition, caldesmon and its endogenous kinase are two different proteins. Furthermore, our study of caldesmon inhibition of actomyosin ATPase activity showed that further research needs to be done to refute F-actin bundling process as a possible cause of caldesmon inhibition of actomyosin ATPase activity. In addition, our studies of caldesmon inhibition of HMM and S-1 ATPase activity suggest that S-2 might be partially involved in the inhibition mechanism. Finally, caldesmon did not affect the 6S-10S transition of myosin conformation and since caldesmon cannot compete against higher affinity calmodulin-binding protein such as MLCK thus, the flip-flop theory is untenable.
|
166 |
The regulation of the type 5 cyclic nucleotide phosphodiesterase in airway smooth muscle by metal ions and small molecular weight proteinsGrady, Amanda Ellen January 1999 (has links)
No description available.
|
167 |
The purification and characterization of a specific 3-methylcholanthrene-binding protein (SBP)Arnold, P. S. January 1987 (has links)
No description available.
|
168 |
A structure-function analysis of tomato annexin p35Eng-Kiat, Lim January 1999 (has links)
No description available.
|
169 |
Studies on the structure and function of the influenza virus ribonucleoprotein and polymerase complexKlumpp, Klaus January 1997 (has links)
No description available.
|
170 |
Identification of new targets for the Rho and Rac GTPasesTapon, Nicolas Alexandre Marie January 1998 (has links)
No description available.
|
Page generated in 0.0454 seconds