Global ETD Search

141	Defect study of zinc oxide bulk materials by positron lifetime spectroscopy 蘇振強, So, Chun-keung. January 2008 (has links) published_or_final_version / abstract / Physics / Master / Master of Philosophy Zinc oxide. Materials - Defects. Positrons - Spectra.
142	Studies of electron irradiation induced deep level defects in p-type 6H-SIC Luo, Jiaming, 羅佳明 January 2009 (has links) published_or_final_version / Physics / Master / Master of Philosophy Silicon carbide - Defects. Deep level transient spectroscopy.
143	Visual assessment of children with, or at risk of, neurological impairment Mackie, Roisin T. January 1995 (has links) No description available. 571.4
144	Atomic level diffusion mechanisms in silicon De Souza, Maria Merlyne January 1993 (has links) No description available. 530.41
145	Cosmological phase transition phenomena Martin, Adrian Peter January 1995 (has links) No description available. 539.72
146	The effects of intraluteal infusion of prostaglandin-synthesis inhibitors on the function of the primate corpus luteum. Sargent, Eva Lee. January 1988 (has links) Exogenous prostaglandins (PGs) have been reported to suppress or to promote the function of the primate corpus luteum in vitro and in vivo, but the role of endogenous ovarian prostaglandins in regulating luteal function during the menstrual cycle is unknown. Infusion (via osmotic pump) of the prostaglandin-synthesis inhibitor sodium meclofenamate into the corpus luteum, but not via the jugular vein, during the midluteal phase of the menstrual cycle resulted in a decline in progesterone levels and premature menses in rhesus monkeys (Macaca mulatta). These results suggest that meclofenamate suppresses the production of an obligatory luteotropic prostaglandin or other metabolite of arachidonic acid. We were unable to confirm that ovarian prostaglandin synthesis was diminished during treatment, since we could not consistently measure a gradient in PGE or PGF₂(α) across the ovary. Dispersed cells from the macaque corpus luteum produced PGF₂(α) in vitro. Production was stimulated by exposure to arachidonic acid and was inhibited by meclofenamate and another prostaglandin-synthesis inhibitor, flurbiprofen. Although the two drugs were potent inhibitors of prostaglandin synthesis in vitro, intraluteal infusion of flurbiprofen in monkeys did not mimic the luteolytic effects of meclofenamate. These studies provide the first evidence of an obligatory luteotropic role for a metabolite of arachidonic acid during the primate luteal phase. However the data suggest that the luteolytic effect of meclofenamate in vivo is not mediated entirely by the inhibition of local prostaglandin synthesis. Further studies are needed to determine the mechanism(s) of meclofenamate-induced luteolysis and to identify the putative obligatory luteotropin. Prostaglandins -- Synthesis. Corpus luteum. Luteal phase defects.
147	COMPARATIVE STUDY OF DISLOCATION BEHAVIOR IN SINGLE-CRYSTAL AND RIBBON-TO-RIBBON SILICON. Pinamaneni, Subba Rao. January 1983 (has links) No description available. Silicon crystals. Semiconductors -- Defects. Dislocations in crystals.
148	Transition-metal dichalcogenides and the scanning tunnelling microscope : the creation and imaging of vacancy defects Caulfield, John Christopher January 1998 (has links) No description available. 530.41 Motatomic defects; Single-atom vacancies
149	The evolution of gauged cosmic string networks Vincent, Graham Richard January 1998 (has links) No description available. 523.01
150	Equilibrium and non-equilibrium aspects of early universe phase transitions Antunes, Nuno Dias January 1998 (has links) No description available. 530.1

Search results