Thesis (D.Psych.)--University of Melbourne, Dept. of Psychology, 2003. / Typescript (photocopy). Includes bibliographical references (leaves 124-146).

Thesis (Ph.D.)--University of Adelaide, Dept. of Anthropology and Social Inquiry, 2002. / Includes bibliographical references (leaves [287]-309).

Thesis (M.A.)--University of Canterbury, 2009. / Typescript (photocopy). Includes bibliographical references (leaves 44-62). Also available via the World Wide Web.

Thesis (M.S.)--University of Wisconsin--Madison, 1985. / Typescript (photocopy). eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 66-74).

Thesis (Ph. D.)--West Virginia University, 2002. / Title from document title page. Document formatted into pages; contains vii, 219 p. Includes abstract. Includes bibliographical references (p. 161-184).

Thesis (M.Div. with Concentration : Christian Ministries)--Emmanuel School of Religion, 2005. / Vita. Includes bibliographical references (leaves 76-79).

Classificationo f individualsa s high and low restrainede aters, accordingt o their relative score on one of the several restraint assessment questionnaires currently in use, has been shown to predicta n anomalouse ating pattern,r eferredt o as counter-regulationo r disinhibition which, appears to be cognitively controlled. Two main sources of cognitive bias which may characterize dietary restraint In female college students have been investigated in this thesis; attentional and memory biases for foodrelated Information. Experiments I-VI assessed selective attention for food-related words. Median split of subjects on scores from a restraint assessment scale yielded contradictory results. When Middle scorers on the restraint assessment scale were excluded from the analyses, It became clear that aftentional biases for food information do not characterize dietary restraint In female college students. Experiment VI replicated this finding In an adolescent population of school girls. However in Experiment III, after consumption of a sweet drink (either high or low calorie), significant Interference effects in colour-narrdng sweet food words emerged for both the high and low restraint groups. The second series of studies examined memory biases for names of foods which are generally considered to be lorbidden' to dieters but which they may still crave. Heightened recall of 'forbidden'f ood words by the high restrainersw as Indicatedi n both ExperimentsV and VI. The dependence of this memory bias upon the subjects knowing that the experiment is concerned With food and eating style was examined in Experiment Vill and the differential effect emerged in both unprimed and primed recall sessions. it was not found In Experiment VII In which subjectsw ere na7ve.T he final study examinedt he effect of self- versus other- referencing during encoding on recall of 'forbidden'and 'healthy'food names by high and low restralners. The experimentsa re discussedi n terms of schematicp rocessingo f emotionally-relevant Information with reference to Beck's Schema Theory of negative affect (1976, Cognitive Therapy and the Emotional Disorders. Int. Uni. Press: NY) and Williams, Watts, MacLeod, & Mathews's model of biased Information processing in emotional disorders (1988, Cognitive Psychology and Ernotional Disorders. Wiley: Chichester). It Is concluded that dietary restraint may be characterized by a memory bias for food names which dieters attempt to avoid. These biases, although statistically significant, were not substantial. it is suggested that future research takes into account the possibility that distinct categories exist within high restraint groups.

No description available.

Objectives: This thesis aimed to investigate whether a history of childhood trauma was indirectly associated with eating psychopathology through mediation by dissociation and/or emotion dysregulation. Method: Firstly, a systematic review was conducted to appraise the current level of evidence within the literature that supported dissociation as a potential mediator. To this end, studies that assessed the variables of childhood trauma, dissociation and eating psychopathology within a single study were identified and systematically reviewed. Secondly, an empirical cross-sectional study was undertaken to investigate a multiple mediation model of the association between childhood trauma and eating psychopathology which included dissociation and emotion dysregulation as potential mediators. 165 undergraduate Psychology students took part in this study. Participants completed measures of childhood trauma, eating psychopathology, dissociation and emotion dysregulation. Experiences of multiple forms of childhood trauma were assessed, including emotional abuse (CEA), physical abuse (CPA), sexual abuse (CSA), emotional neglect (CEN) and physical neglect (CPN). Results: The results of the systematic review were inconclusive regarding the potential role of dissociation as a mediator in the relationships between childhood trauma and eating psychopathology. Findings within the reviewed studies generally offered more support for associations between childhood trauma and dissociation and dissociation and eating psychopathology. Studies reported more inconsistent findings regarding the association between childhood trauma and eating psychopathology. The results from the empirical study indicated that CEA and CEN were both significantly associated with increased eating psychopathology within the whole sample. These relationships were significantly mediated by both dissociation and emotion dysregulation. A separate analysis with female participants only, indicated that CPA and CPN in addition to CEA and CEN were significantly associated with increased eating psychopathology. The associations between CEA, CEN, CPN and eating psychopathology were all significantly mediated by both dissociation and emotion dysregulation. Dissociation and emotion dysregulation did not mediate the association between CPA and eating psychopathology. Conclusions: The studies included within the systematic review offered tentative support for an indirect relationship between childhood trauma and eating psychopathology through dissociation. Firm conclusions were limited, however, due to a number of methodological shortcomings identified within the included studies. The main methodological shortcomings concerned the definition and measurement of childhood trauma and the failure of a number of studies to address theoretical models within their research design. Addressing both of these methodological limitations, the results of the empirical study provided support for the growing consensus that emotional maltreatment may be an important risk factor for the development of eating psychopathology. Further, the results of this study indicate that childhood trauma impacts indirectly on eating psychopathology through an enduring effect on both dissociative and emotion regulation processes.

Background The Ravello Profile is a battery of standardised neuropsychological measures of areas of functioning that evidence indicates are impaired in Anorexia Nervosa (AN), namely visuo-spatial functioning, central coherence and executive functioning. The neuropsychological profile of individuals with Bulimia Nervosa (BN) is less well established. The current study aimed to examine differences in cognitive performance between people with BN, AN and non-eating disordered controls on the Ravello Profile. Methods The AN group (N=60) comprised participants from an existing database (Frampton et al. 2009). The BN group (N=22) largely comprised participants from NHS adult out-patient services. The non-eating disordered control group (N=20) comprised of colleagues and acquaintances of the researcher. Differences between AN, BN and control samples on visuo-spatial functioning, central coherence, executive functioning and error rates were examined. Results The AN group performed significantly worse than the BN group on a measure of central coherence and on some measures of executive function, but the BN group did not perform worse than the control group. There was no significant difference between the groups on three measures of visuo-spatial functioning. However, the AN group was significantly slower than both the BN and control group to copy the figure. The results showed some evidence of increased error rates in BN relative to AN, which may reflect greater impulsivity in BN. Conclusions The results indicate separate patterns of neuropsychological performance between AN, BN and controls, with AN demonstrating poorer performance on measures of executive function and central coherence, whilst BN participants showed higher rates of errors. The BN group were also generally faster to complete some tasks, indicative of a preference for speed over accuracy or impulsivity. Those working with individuals with AN or BN should take into consideration possible effects of their respective cognitive limitations and adapt interventions accordingly.