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Computational models of cerebral hemodynamics : a thesis presented for the degree of Doctor of Philosophy in Bioengineering at the University of Canterbury, Christchurch, New Zealand /Alzaidi, Samara Samir. January 1900 (has links)
Thesis (Ph. D.)--University of Canterbury, 2009. / Typescript (photocopy). "1 April 2009." Includes bibliographical references (leaves [263]-274). Also available via the World Wide Web.
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Long-term patency of a polymer vein valveMidha, Prem Anand. January 2009 (has links)
Thesis (M. S.)--Bioengineering, Georgia Institute of Technology, 2010. / Committee Chair: Ku, David; Committee Member: Gleason, Rudolph; Committee Member: Milner, Ross. Part of the SMARTech Electronic Thesis and Dissertation Collection.
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A comparison of hemodynamic responses in losartan- and enalapril- treated normotensive ratsWang, De, 王得 January 1999 (has links)
published_or_final_version / Medical Sciences / Master / Master of Medical Sciences
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A mathematical model of the dynamics of cytosolic free calcium in cultured vascular endothelial cells responding to shear stressWiesner, Theodore F. 08 1900 (has links)
No description available.
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In vitro hemodynamics of valved cardiac conduitsFaughnan, Patrick Doris 05 1900 (has links)
No description available.
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Stentless mitral valve fixation : impact on hemodynamic performanceJensen, Morten Ølgaard Jegstrup 05 1900 (has links)
No description available.
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Visualization of flow phenomena in a vascular graft modelWhite, Samuel Scott 12 1900 (has links)
No description available.
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Effect of oscillatory flow on the intracellular free calcium concentration of single cultured bovine aortic endothelial cellsFrancke, Eric Ivars 08 1900 (has links)
No description available.
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Hemodynamic environment regulates matrix metalloproteinase-9 expression in vascular endothelial cellsMagid, Richard Allem 08 1900 (has links)
No description available.
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Investigation of Arterial Geometry as a Local Risk Factor for Carotid AtherosclerosisBahman Bijari, Payam 02 August 2013 (has links)
There is little doubt that disturbed hemodynamic forces play a role in the development of focal atherosclerotic lesions; however, these forces are difficult to measure directly. Instead, it has been proposed that artery geometry, as the primary determinant of local hemodynamics, could be a clinically feasible surrogate “local” risk factor for atherosclerosis. To date this hypothesis has not been satisfactorily tested, owing to superficial geometric surrogates of disturbed flow, small sample sizes (effect of systemic factors) and/or confounding effects of disease on geometry. The primary objective of this thesis was to test this “geometric risk hypothesis” via direct association of definitive geometric factors and an early atherosclerosis marker (e.g. wall thickness), made possible through our access to magnetic resonance imaging and risk factor data from the Atherosclerosis Risk in Communities’ Carotid MRI sub-study. First, it was shown that the 3D geometry of the carotid bifurcation could be characterized rapidly and reliably, even for routine clinical acquisitions. Second, two novel individual geometric variables were proposed, inspired by the influence of flare and tortuosity on flow separation, which were shown to improve the prediction of disturbed flow burden compared to “conventional” shape-based geometric variables. Third, these redefined geometric factors, but not their shape-based counterparts, were shown by multiple regression to be independent predictors of wall thickness, but only after thoroughly accounting for the secondary effects of wall thickening on geometry. These findings provide strong evidence for the geometric risk hypothesis of atherosclerosis in humans group study, and provide important guidance for future investigations of geometric risk; however, the incremental value of optimized geometric risk factors is questionable relative to conventional cardiovascular risk factors, which challenges their future clinical usage as additional non-modifiable local risk factors.
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