<p>This paper presents verbal working memory test results towards establishing the effects of menstrual cycle on working memory of women. The study comprised of a subject-set of twenty healthy young women with a regular 28 – 32 day menstrual cycles. Subjects were tested twice, once during their menstrual phase and second during their ovulation phase (on approximately day 12). Working memory tests were performed in a random sequence i.e. for some subjects during the menstrual phase (low estrogen level) working memory test occurred before their ovulation phase (high estrogen level) memory test and vice versa for other subjects. Study revealed that the test scores in the ovulatory phase were significantly higher than those in the menstrual phase. These findings suggest that higher levels of estrogen may improve working memory. Moreover, effects of estrogen on mood were also considered during both phases of menstruation. The fluctuation in estrogen levels seems to have an effect on women’s mood during menstrual and ovulation phases.</p>

This paper presents verbal working memory test results towards establishing the effects of menstrual cycle on working memory of women. The study comprised of a subject-set of twenty healthy young women with a regular 28 – 32 day menstrual cycles. Subjects were tested twice, once during their menstrual phase and second during their ovulation phase (on approximately day 12). Working memory tests were performed in a random sequence i.e. for some subjects during the menstrual phase (low estrogen level) working memory test occurred before their ovulation phase (high estrogen level) memory test and vice versa for other subjects. Study revealed that the test scores in the ovulatory phase were significantly higher than those in the menstrual phase. These findings suggest that higher levels of estrogen may improve working memory. Moreover, effects of estrogen on mood were also considered during both phases of menstruation. The fluctuation in estrogen levels seems to have an effect on women’s mood during menstrual and ovulation phases.

Animal research has elucidated the neurobiological substrates and environmental determinants of pain modulation. Despite these advances, relatively little is known about how psychological processes activate pain modulatory systems. One psychological process that is thought to play an important role in regulating pain sensitivity is emotion. In addition, previous research into the human menstrual cycle and the animal estrous cycle have determined that either the presence of certain gonadal hormones or the fluctuations of these hormones may lead to changes in how females perceive pain, regulate emotion, and modulate pain. The present study examines both the role of emotion and the human menstrual cycle in pain modulation. Participants were 39 female undergraduate students with a mean age of 18.7 years (SD=1.46). Results are consistent with prior studies indicating that progesterone has antiinflammatory effects. Specifically, significant effects were observed primarily in the luteal phase. Subjects in the luteal phase demonstrated less sympathetic arousal during the experiment but greater autonomic arousal during the noise stressor. Participants in the luteal phase also demonstrated an analgesic/anti-inflammatory response evidenced by an observed decrease in secondary hyperalgesia for those that did not receive the noise stressor. No such changes in pain perception were discovered in the ovulation and follicular phases. Finally, in response to the noise stressor, an inhibition of the analgesic/anti-inflammatory effects was observed in the luteal phase. No such evidence of stress-induced pain modulation was discovered in the ovulation and follicular phases. Although the specific mechanisms of this action still remain unclear, prior evidence points to the role of centrally-mediated pain modulation. It is likely that the stressor worked to inhibit the anti-inflammatory effects commonly observed in the luteal phase to persistent inflammatory pain through centrally-mediated pain modulatory mechanisms. It is hypothesized that hormone-mediated effects at the level of the amygdala influenced the impact of affective pain modulation.

Thesis (M.S.)--Florida State University, 2004. / Advisor: Dr. David Pargman, Florida State University, College of Education, Dept. of Educational Psychology and Learning Systems. Title and description from dissertation home page (viewed 6/16/04). Includes bibliographical references.

published_or_final_version / Zoology / Master / Master of Philosophy

The effects of one night of 3h sleep on cortisol levels were assessed in two groups of women at different points in their menstrual cycles: mid-follicular and mid-luteal. Eighteen women (age: 21.8 ± 0.54; BMI: 22.6 ± 0.63, mean ± SEM) were studied. Salivary samples were collected at six times during two consecutive days: first after a 10 h overnight sleep opportunity and then after a night with a 3 h sleep opportunity. Secondary analysis examined the impact of sleep restriction on self-reports of hunger ratings and mood. Women in the follicular phase showed a significant decrease (p =0.004) in their cortisol awakening responses after sleep restriction and an elevation in afternoon/evening cortisol levels (p =0.008), whereas women in the luteal phase showed no change. Overall group increases in hunger and deterioration in fatigue and vigour were observed. Menstrual cycle phase dramatically altered the responses of women to a single night of sleep restriction.

Unlike men, women’s genital arousal is category-nonspecific with respect to sexual orientation, such that their genital responses do not differentiate stimuli by gender. A possible explanation for women’s nonspecific sexual response is the inclusion of women at different phases of the menstrual cycle or women using hormonal contraceptives in sexual psychophysiology research, which may be obscuring a specificity effect. The present study employs the ovulatory-shift hypothesis – used to explain a shift in women’s preferences for masculine traits during peak fertility – as an explanatory model for women’s nonspecific sexual arousal. Twenty-nine naturally-cycling women were tested at two points in their menstrual cycles (follicular and luteal) to determine the role of hormonal variation, as estimated by fertility status, on the specificity of genital (using vaginal photoplethysmograph) and subjective sexual arousal. Cycle phase at the time of first testing was counterbalanced; however, no effect of order was observed. Inconsistent with the ovulatory-shift model, the predicted mid-cycle shift in preferences for masculinity or sexual activity at peak fertility was not obtained. Category-specificity of genital arousal did not increase during the follicular phase. A statistical trend was observed for higher genital arousal to couple sex stimuli during the follicular phase compared to the luteal phase, suggesting that women’s genital arousal may be sensitive to fertility status with respect to sexual activity (specifically, couple sex), but not gender. Subjective arousal was not influenced by fertility status. This study is the first to provide evidence that women’s genital arousal may be influenced by the probability of conception. / Thesis (Master, Psychology) -- Queen's University, 2011-09-29 08:51:26.973

Diss. (sammanfattning) Uppsala : Univ., 2004. / Härtill 4 uppsatser.

Diss. (sammanfattning) Stockholm : Karol. inst., 2004. / Härtill 4 uppsaatser.

Thesis (M.S.)--Indiana University, 2004. / Includes bibliographical references (leaves 67-88). Also available online (PDF file) by a subscription to the set or by purchasing the individual file.