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  • About
  • The Global ETD Search service is a free service for researchers to find electronic theses and dissertations. This service is provided by the Networked Digital Library of Theses and Dissertations.
    Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.

The complex genetics of experimental autoimmune neuroinflammation /

Jagodić, Maja, January 2004 (has links)
Diss. (sammanfattning) Stockholm : Karol. inst., 2004. / Härtill 4 uppsatser.

On the possible use of oxysterols for the diagnosis and evaluation of patients with neurological and neurodegenerative diseases /

Leoni, Valerio, January 2005 (has links)
Diss. (sammanfattning) Stockholm : Karol. inst., 2005. / Härtill 5 uppsatser.

Neurogenesis, neural stem cells and nitric oxide in neuroinflammation /

Danilov, Alexandre I., January 2005 (has links)
Diss. (sammanfattning) Stockholm : Karolinska institutet, 2005. / Härtill 6 uppsatser.

Hormones and dendritic cells influences on the initiation of the autoimmune disease experimental autoimmune encephalomyelitis /

Papenfuss, Tracey L. January 2007 (has links)
Thesis (Ph. D.)--Ohio State University, 2007. / Full text release at OhioLINK's ETD Center delayed at author's request

Genetic analysis of IL7R and other immune-regulatory genes in multiple sclerosis /

Lundmark, Frida, January 2007 (has links)
Diss. (sammanfattning) Stockholm : Karolinska institutet, 2007. / Härtill 5 uppsatser.

Quantitative magnetic resonance in diffuse neurological and liver disease /

Dahlqvist Leinhard, Olof, January 2010 (has links)
Diss. (sammanfattning) Linköping : Linköpings universitet, 2010. / Härtill 6 uppsatser.

Rescue therapy with alemtuzumab in B cell/antibody-mediated multiple sclerosis

Akgün, Katja, Metz, Imke, Kitzler, Hagen H., Brück, Wolfgang, Ziemssen, Tjalf 05 November 2019 (has links)
Alemtuzumab exerts its clinical efficacy by its specific pattern of depletion and repopulation of different immune cell subsets. Recently, single cases of multiple sclerosis patients who developed severe exacerbation after the first alemtuzumab application, accompanied by re-appearance of peripheral B cells, were reported. Here we present a case with underlying B cell-driven multiple sclerosis that impressively improves after alemtuzumab, although peripheral B cell repopulation took place. Our detailed clinical, histopathological, imaging and immunological data suggest that alemtuzumab can act as an effective rescue treatment in highly active B cell-driven and antibody/complement-mediated multiple sclerosis type II patients.

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