Nineteenth-Century Pets and the Politics of Touch examines texts of the era in which both humans and animals find empowerment at the point of physical encounter. I challenge contemporary perceptions of human-pet relationships as sweetly affectionate by focusing on touch. I uncover an earlier interest in the close reciprocal relationships between human and nonhuman animals, arguing that these nineteenth-century thinkers presented what I call a “politics of touch,” in which intimate and often jarring physical encounters allow for mutuality and autonomy. I first turn to Charlotte Brontë’s Shirley (1849) and protective violence, a condoned ferocity that frequently unites and guards pet and pet keeper against unwanted amorous intrusions, while also showcasing animal agency and the possibility of deviation from the pet keeper’s wishes. Brontë’s animals simultaneously preserve and rework the traditional form of the marriage plot, allowing for powerful animal-centric possibilities. In chapter 2, I analyze the affective maternal and erotic bonds between women and their pets in Olive Schreiner’s novels. While this touch was frequently seen by both protofeminists and people antagonistic to women’s rights as a cause for disdain because affection was supposedly misplaced, it is a crucial part of Schreiner’s feminist project in that it provides forms of maternity outside of the socially mandated wifehood and motherhood that Schreiner so resents for stripping women of their autonomy. For chapter 3, I seek to complicate readings of Count Fosco, the compelling villain of Wilkie Collins’s The Woman in White (1860), to show the disquieting sympathy that privileges odd women and animals. Heeding Count Fosco shows that valuable sympathy is not a pretty picture of a lovely woman walking with her purebred dog, but rather the excessively grotesque images of an unattractive woman holding a dying dog in her arms and mice and birds erotically clamoring over a fat man’s body. The final chapter considers the violent sympathetic touch evidenced in the practice of mercifully killing grieving dogs in Frances Power Cobbe’s animal advocacy texts. I argue that Cobbe’s schema recognizes gender fluidity as she posits a feminized animal grief marked by excess, while she concurrently masculinizes human sympathy by making it violent through mercy killings that complicate our accepted understandings of nineteenth-century sentiment. In contrast to other scholars of nineteenth-century animal studies who look at how humans understand and treat animals, my focus on the reciprocity of human-animal touch keeps animals at the center of my analysis. I argue that nineteenth-century sympathetic and sentimental texts, often dismissed as trite or as creating distance between the sympathizing subject and object of sympathy, demonstrate theoretical and political complexity through representations of shockingly intimate touch. In doing so, Victorian writers anticipated and even transcended recent theoretical conversations in the field of feminist animal studies.

The increasing prevalence of allergic diseases in affluent societies has been associated with changes in microbial exposure early in life and a less diverse gut flora. The objective of this thesis was to assess the development of allergic sensitisation and symptoms during the first four years of life in a non-selected birth cohort in relation to environmental factors, family history, gut microbiota and salivary IgA antibodies. The cohort comprised all 1,228 infants living in a Swedish county who were born over a one-year period. The parents replied to questionnaires, and 817 children (67 %) were skin prick tested both at 1 and 4 years of age. Saliva (n=279), faecal (n=139) and blood (n=253) samples were collected at 1 year of age from children with a positive skin prick test at 1 year and from a sample of children with a negative skin prick test. Faecal samples were also obtained from 53 children at 4 years of age. Dog keeping during infancy was associated with a decreased risk of sensitisation to pollen and late-onset wheezing at age 4, and the reduced odds ratios persisted after adjustment for heredity and avoidance measures, OR 0.3, 95% CI 0.1-0.9 and OR 0.5, 95% CI 0.2-1.0, respectively. In contrast, early dog keeping was associated with an increased risk of earlyonset transient wheezing but only in children with parental asthma (OR 2,8, 95% CI 1.3-6.4). Levels of short chain fatty acids (SCFAs) in faeces were assessed both at 1 and 4 years of age and related to the development of sensitisation and symptoms. The levels of acetic (p<.01) and propionic (p<.01) acids decreased from one to four years of age, whereas valeric acid (p<.001) increased which is in line with a more complex gut microbiota with age. Allergic children, compared with non-allergic children, had lower levels of i-butyric, i-valeric and valeric acid in faeces both at 1 and 4 years of age. Low levels of secretory IgA (SIgA) in saliva were associated with wheezing but only in sensitised children. In children with positive SPT to at least one allergen both at 1 and 4 years of age and in children with circulating IgE antibodies to egg or cat at one year of age, those who developed late-onset wheezing had lower levels of SIgA than those who did not, p=.04 and p=.02 respectively. Of 9 children with levels of SIgA in the upper quartile and persistent sensitisation, none developed wheezing, compared with 10/20 children with lower levels, (p=. 01). Having older siblings, more than three infections during infancy, at least one smoking parent and male gender were all associated with high levels (in the upper quartile) of total IgA and SIgA. The findings in this thesis indicate that the microbial load early in life could affect the development of allergy. A functional assessment of the gut flora demonstrated differences between allergic and non-allergic children both at 1 and 4 years of age. Salivary IgA was associated with infections during infancy, and high levels of secretory IgA protected from symptoms in sensitised children. Finally, dog keeping in infancy may offer protection from allergy, but the mechanism is uncertain.

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