Résumé of Thesis (Sc. D.)--University of Michigan, 1940. / Reproduced from type-written copy. eContent provider-neutral record in process. Description based on print version record. Bibliography: leaf 6.

Thesis (M.P.H.)--University of Wisconsin -- La Crosse, 2009. / Includes bibliographical references (leaves 57-63)

Thesis (Ph. D.)--University of Texas at Austin, 2001. / Vita. Includes bibliographical references (leaves 180-194). Available also in a digital version from Dissertation Abstracts.

Title page, contents and abstract only. The complete thesis in print form is available from the University of Adelaide Library. / In human populations, perturbed growth in early life and ageing have been identified as risk factors for the development of Insulin resistance syndrome (IRS). The consequences of restricted prenatal growth on postnatal function have been investigated using numerous experimental models of intrauterine growth retardation, mainly in the rat. These studies have shown that some, but not all, aspects of postnatal function that are programmed in humans are also programmed in the rat. This study was designed to determine whether IRS develops with increasing age in the guinea pig as it does in the human and whether the development of the syndrome is more pronounced in aged offspring which have undergone spontaneous fetal growth restriction and accelerated growth in the neonatal period. It appeared that the guinea pig is a suitable animal model of ageing, displaying many of the metabolic, cardiovascular and anthropometric changes seen in humans. Furthermore, the effects of perturbed prenatal and early postnatal growth on the development of IRS in the aged guinea pig exhibit a sexually dimorphic pattern. / http://proxy.library.adelaide.edu.au/login?url= http://library.adelaide.edu.au/cgi-bin/Pwebrecon.cgi?BBID=1297545 / Thesis (Ph.D.) -- University of Adelaide, School of Molecular and Biomedical Science, 2007

Title page, contents and abstract only. The complete thesis in print form is available from the University of Adelaide Library. / In human populations, perturbed growth in early life and ageing have been identified as risk factors for the development of Insulin resistance syndrome (IRS). The consequences of restricted prenatal growth on postnatal function have been investigated using numerous experimental models of intrauterine growth retardation, mainly in the rat. These studies have shown that some, but not all, aspects of postnatal function that are programmed in humans are also programmed in the rat. This study was designed to determine whether IRS develops with increasing age in the guinea pig as it does in the human and whether the development of the syndrome is more pronounced in aged offspring which have undergone spontaneous fetal growth restriction and accelerated growth in the neonatal period. It appeared that the guinea pig is a suitable animal model of ageing, displaying many of the metabolic, cardiovascular and anthropometric changes seen in humans. Furthermore, the effects of perturbed prenatal and early postnatal growth on the development of IRS in the aged guinea pig exhibit a sexually dimorphic pattern. / http://proxy.library.adelaide.edu.au/login?url= http://library.adelaide.edu.au/cgi-bin/Pwebrecon.cgi?BBID=1297545 / Thesis (Ph.D.) -- University of Adelaide, School of Molecular and Biomedical Science, 2007

Thesis (M.Sc. (Hons)) -- University of Western Sydney, 2002. / "Thesis submitted to fulfil requirements for M. Sci. (Hons)" "January 2002" Bibliography: p. 193 - 214.

Thesis (Ph. D.)--Auburn University, 2008. / Abstract. Vita. Includes bibliographical references.

Thesis (M.S.)--Brigham Young University. Dept. of Physiology and Developmental Biology, 2007. / Includes bibliographical references (p. 33-47).

Thesis (M.S.)--West Virginia University, 2010. / Title from document title page. Document formatted into pages; contains vii, 46 p. : ill. (some col.). Includes abstract. Includes bibliographical references (p. 42-46).

Thesis (Ph.D.)--Texas Christian University, 2007. / Title from dissertation title page (viewed Sept. 11, 2007). Includes abstract. Includes bibliographical references.