Spelling suggestions: "subject:"renal failure"" "subject:"renal ailure""
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The biology of galectin-3 in normal and cystic renal developmentJohnson, Tanya Michelle January 2002 (has links)
No description available.
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Could a calcium antagonist be the ideal drug treatment of elderly hypertensives?Chellingsworth, Miriam Claire January 1992 (has links)
No description available.
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Nitric oxide modulates the effect of RHIGF-I on the accumulation of collagen and kidney growthAli, Abdulgader Saleh January 1997 (has links)
No description available.
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Effect of IGF-I receptor antagonists on the kidney in STZ-induced diabetesHickling, Helen Marie January 1999 (has links)
No description available.
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Recombinant human erythropoietin therapy for anaemia in patients maintained by continuous ambulatory peritoneal dialysis (CAPD)Stevens, Judith Mary January 1990 (has links)
No description available.
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Immune responses in mice with chronic renal failureGibb, Iain Alasdair January 1983 (has links)
No description available.
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The aetiology and management of peritonitis in the continuous ambulatory peritoneal dialysis patientMawhinney, William Michael January 1992 (has links)
No description available.
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Regulation of mammalian gene expression by oxygenGleadle, Jonathan M. January 1997 (has links)
No description available.
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Plasminogen activators and their inhibitor synthesized by human mesangial cells and other cell typesReid, Fiona Jane January 1994 (has links)
The plasminogen activator synthesized by human mesangial cells was identified as tissue-type plasminogen activator (t-PA). Glomerular epithelial cells synthesized t-PA and urokinase (u-PA). Both mesangial cells and glomerular epithelial cells synthesized plasminogen activator inhibitor-1 (PAI-1) into culture supernatant. In this study experimental techniques were developed to allow quantification of matrix-associated PAI-1. PAI-1 associated with the matrix of mesangial cells was less than 1% of the total PAI-1 synthesized by the cells. Similar amounts were detected in the matrix of human umbilical vein endothelial cells and Hep G2 cells. When cultured in the presence of transforming growth factor- (TGF-), PAI-1 synthesized by mesangial cells was significantly increased and this up-regulation was shown to occur at the mRNA level. PAI-1 associated with the matrix of mesangial cells also significantly increased, though matrix-associated PAI-1 still constituted less than 1% of the total PAI-1 synthesized by the cells. TGF- stimulated the synthesis of PAI-1 by whole glomeruli, epithelial cells and epithelial-mesangial cell co-cultures, while decreasing their overall plasminogen activator synthesis. When cultured in the presence of platelet-derived growth factor-BB (PDGF-BB) there was no effect on either PA or PAI-1 synthesized by the glomerular cells examined. TGF- has been implicated in the development of glomerulonephritis via an effect on matrix production. Our results suggest tht TGF- also plays a role in the proteolytic balance within the glomerulus, leading to an environment favouring its deposition.
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The prevalence and nutritional causes of hypoglycaemia in patients with end-stage renal failure (ESRF) on maintenance haemodialysis (MHD) at Kenyatta National Hospital Nairobi, Kenya /Kariuki, Anastacia Wanjiku. January 2008 (has links)
Thesis (MNutr)--University of Stellenbosch, 2008. / Bibliography. Also available via the Internet.
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