- About
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The Global ETD Search service is a free service for researchers
to find electronic theses and dissertations. This service is
provided by the
Networked Digital Library of Theses and Dissertations.
Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
Search results
Showing 1 to 6 of 6 (0.018 seconds)Spelling suggestions: "subject:"or1"" "subject:"mor1""
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Noncanonical Wnt signaling in breast cancer initiation and progressionBorcherding, Nicholas 01 July 2014 (has links)
No description available.
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Preclinical development of a non-immunosuppressive FTY720 derivative OSU-2S forchronic lymphocytic leukemia and other B-cell malignanciesMani, Rajeswaran 07 October 2014 (has links)
No description available.
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Developing Methods and Targeted Therapeutics to Address Complications of Ibrutinib Treatment in Chronic Lymphocytic LeukemiaHu, Eileen Yifan 07 October 2020 (has links)
No description available.
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Wnt-Signalwegsanalysen während der Metastasierung von Mamma-Karzinomen / Wnt-Signaling in Breast Cancer and Tumor ProgressionSchubert, Antonia 21 November 2016 (has links)
No description available.
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ROR1 Targeted Therapy in Small Cell Lung CancerWang, Walter Z. 11 August 2022 (has links)
No description available.
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FGF4 Induced Wnt5a Gradient in the Limb Bud Mediates Mesenchymal Cell Directed Migration and DivisionAllen, John C 01 December 2013 (has links) (PDF)
The AER has a vital role in directing embryonic limb development. Several models have been developed that attempt to explain how the AER directs limb development, but none of them are fully supported by existing data. I provide evidence that FGFs secreted from the AER induce a gradient of Wnt5a. I also demonstrate that limb mesenchyme grows toward increasing concentrations of Wnt5a. We hypothesize that the changing shape of the AER is critical for patterning the limb along the proximal to distal axis. To better understand the pathway through which Wnt5a elicits its effects, we have performed various genetic studies. We demonstrate that Wnt5a does not signal via the Wnt/β-catenin pathway. However, we show that Wnt5a mutants share many common defects with Vangl2 mutants suggesting that Wnt5a signals through the Wnt/planar cell polarity (PCP) pathway.
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