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Characterization of host-bacteria interactions contributing to group B streptococcus colonizationSmith, Jennifer Marie. January 2002 (has links)
Thesis (Ph. D.)--Marshall University, 2002. / Title from document title page. Document formatted into pages; contains x, 147 p. Includes abstract. References at the end of each chapter.
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112 |
Molecular detection of streptococcus sinensis in blood culture samplesfrom hospitalized patients in Hong Kong: aneight-year retrospective studyChan, Shun-kwan., 陳信君. January 2011 (has links)
published_or_final_version / Microbiology / Master / Master of Medical Sciences
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113 |
Characterization of a novel streptococcus species associated with marine environmentLin, Ching-nga., 林靜雅. January 2011 (has links)
published_or_final_version / Microbiology / Master / Master of Medical Sciences
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114 |
Multilocus sequence typing for streptococcus agalactiaeNg, Yi-ting., 吳依婷. January 2011 (has links)
published_or_final_version / Microbiology / Master / Master of Medical Sciences
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115 |
Vancomycin tolerance in streptococcus pneumoniaeKoo, Kun, 古軍 January 2002 (has links)
published_or_final_version / Medical Sciences / Master / Master of Medical Sciences
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116 |
Epidemiological and emm gene analysis of non-m-typeable group A streptococcus isolates from Hong Kong余慧儀, Yu, Wai-yee, Annie. January 2001 (has links)
published_or_final_version / Medical Sciences / Master / Master of Medical Sciences
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117 |
Functional and evolutionary analyses of pneumococcal genome variationCroucher, Nicholas Jason January 2012 (has links)
No description available.
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118 |
Inhibitory effects in vivo of certain plant extracts upon Streptococcus pyogenesHansen, Jo Ann Brown, 1929- January 1954 (has links)
No description available.
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119 |
Platelet GPIb and downstream activation by S. sanguisAbdulrehman, Ahmed Y. 13 December 2010 (has links)
There is increasing evidence suggesting the contribution of bacterial infections in atherothrombotic conditions. Studies have demonstrated that bacteria residing within the oral cavity activate platelets once they enter circulation. S. sanguis 2017-78 is capable of stimulating platelet aggregation in a thromboxane-dependent manner. In the present study, the signaling events associated with S. sanguis have been studied further. S. sanguis 2017-78 caused the phosphorylation of p38 MAP kinase and subsequently cPLA2. The p38 MAP kinase inhibitor, SB203580 inhibited S. sanguis 2017-78-induced platelet aggregation as well as the phosphorylation of both p38 MAP kinase and cPLA2. These data are consistent with cPLA2 as a physiological target of p38.
A second component of the study examined the effects of aspirin, a known inhibitor of cyclooxygenase, on these signalling pathways.
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Acute respiratory infection in young children in Nairobi, Kenya : epidemiological, clinical and microbiological studyKamiya, Yasuhiko January 2001 (has links)
No description available.
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