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  • About
  • The Global ETD Search service is a free service for researchers to find electronic theses and dissertations. This service is provided by the Networked Digital Library of Theses and Dissertations.
    Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
1

Measurement of temporomandibular joint pain, miscellaneous pain dysfunction, and endodontic pain using the McGill Pain Questionnaire a thesis submitted in partial fulfillment ... dental hygiene education ... /

Killins, Kathleen S. January 1981 (has links)
Thesis (M.S.)--University of Michigan, 1981.
2

Participação da endotelina-1 na sinovite induzida por carragenina na articulação temporomandibular de ratos. / Endothelin-1 participation on the rats temporomandibular joint synovitis induced by carrageenan.

Paula, Marco Aurélio Vieira de 29 May 2012 (has links)
Disfunções temporomandibulares são secundárias à inflamação e dor. Estudos mostram a participação da ET na nocicepção. Investigamos o papel da ET1 na sinovite induzida pela injeção de CGN na ATM de ratos. Injeções intra-articulares de CGN, ET1 ou do agonista do receptor ETB BQ3020 foram realizadas na ATM. Após 4h, a hiperalgesia foi avaliada usando um analgesímetro digital e as cavidades articulares foram lavadas para contagem total e diferencial de leucócitos. Antagonistas dos receptores ETA e ETB foram co-injetados com CGN e ET1, e o antagonista de TRPV1 capsazepina com a ET1. A ET1, o BQ3020 e a CGN induziram hiperalgesia que foi inibida pela injeção simultânea (mas não individual) dos antagonistas ETA e ETB. A capsazepina não afetou o efeito nociceptivo da ET1. Nem a ET1, o BQ3020 nem os antagonistas dos receptores da ET afetaram a infiltração de leucócitos. Com base nestes resultados concluímos que a ET1 induz hiperalgesia e está envolvida na nocicepção induzida por CGN através dos receptores ETA e ETB, sem qualquer efeito sobre a migração de leucócitos. / Temporomandibular disorders are secondary to inflammation and pain. Studies show the involvement of ET in nociception. Weve investigated the role of ET1 in synovitis induced by CGN injection on the rats TMJ. Intra-articular injections of CGN, ET1 or ETB receptor agonist BQ3020 were performed at the TMJ. After 4h, hyperalgesia was assessed using a digital analgesimeter and the joint cavities were washed for total and differential leukocyte counting. ETA and ETB receptors antagonists were co-injected with CGN and ET1, and the TRPV1 antagonist capsazepine with ET1. ET1, BQ3020 and CGN induced hyperalgesia, which was inhibited by simultaneous (but not single) antagonists ETA and ETB injection. The capsazepine did not affect the ET1 nociceptive effect. Neither ET1, BQ3020 nor antagonists of ET receptors affect leukocyte infiltration. Based on these results we conclude that the ET1 induces hyperalgesia and its involved in CGN induced nociception through ETA and ETB receptors, without any effect on the leukocytes migration.
3

Participação da endotelina-1 na sinovite induzida por carragenina na articulação temporomandibular de ratos. / Endothelin-1 participation on the rats temporomandibular joint synovitis induced by carrageenan.

Marco Aurélio Vieira de Paula 29 May 2012 (has links)
Disfunções temporomandibulares são secundárias à inflamação e dor. Estudos mostram a participação da ET na nocicepção. Investigamos o papel da ET1 na sinovite induzida pela injeção de CGN na ATM de ratos. Injeções intra-articulares de CGN, ET1 ou do agonista do receptor ETB BQ3020 foram realizadas na ATM. Após 4h, a hiperalgesia foi avaliada usando um analgesímetro digital e as cavidades articulares foram lavadas para contagem total e diferencial de leucócitos. Antagonistas dos receptores ETA e ETB foram co-injetados com CGN e ET1, e o antagonista de TRPV1 capsazepina com a ET1. A ET1, o BQ3020 e a CGN induziram hiperalgesia que foi inibida pela injeção simultânea (mas não individual) dos antagonistas ETA e ETB. A capsazepina não afetou o efeito nociceptivo da ET1. Nem a ET1, o BQ3020 nem os antagonistas dos receptores da ET afetaram a infiltração de leucócitos. Com base nestes resultados concluímos que a ET1 induz hiperalgesia e está envolvida na nocicepção induzida por CGN através dos receptores ETA e ETB, sem qualquer efeito sobre a migração de leucócitos. / Temporomandibular disorders are secondary to inflammation and pain. Studies show the involvement of ET in nociception. Weve investigated the role of ET1 in synovitis induced by CGN injection on the rats TMJ. Intra-articular injections of CGN, ET1 or ETB receptor agonist BQ3020 were performed at the TMJ. After 4h, hyperalgesia was assessed using a digital analgesimeter and the joint cavities were washed for total and differential leukocyte counting. ETA and ETB receptors antagonists were co-injected with CGN and ET1, and the TRPV1 antagonist capsazepine with ET1. ET1, BQ3020 and CGN induced hyperalgesia, which was inhibited by simultaneous (but not single) antagonists ETA and ETB injection. The capsazepine did not affect the ET1 nociceptive effect. Neither ET1, BQ3020 nor antagonists of ET receptors affect leukocyte infiltration. Based on these results we conclude that the ET1 induces hyperalgesia and its involved in CGN induced nociception through ETA and ETB receptors, without any effect on the leukocytes migration.
4

Demographics of Ohio Licensed Dentists and Treatment Preferences for Temporomandibular Joint Disorders

Noll, Sean William 18 August 2022 (has links)
No description available.

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