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The effect of ascorbic acid supplementation on the absorption of iron from some vegetable staplesSayers, Merlyn Herbert January 2015 (has links)
No description available.
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A mechanism for ascorbate induced cataract in human lens /Simpson, Gregory L. W. January 2001 (has links)
Thesis (Ph. D.)--University of Missouri--Columbia, 2001. / "December 2001." Typescript. Vita. Includes bibliographical references (leaves 153-159). Also available on the Internet.
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A study of the chemical degradation of ascorbic acid in model systemsDe Salegui, Miriam Nurnberg, January 1957 (has links)
Thesis (Ph. D.)--University of Wisconsin--Madison, 1957. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.
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On the nature of ascorbic acid oxidaseDunn, Frank Jack, January 1949 (has links)
Thesis (Ph. D.)--Columbia University. / Vita. Bibliography: p. 40-41.
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Thermal transitions in wheat glutenHayta, Mehmet January 1999 (has links)
No description available.
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Poly(aniline) composites as bioelectrochemical sensorsWallace, Emma Naomi Kathleen January 1997 (has links)
No description available.
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Vitamin C and milk a dissertation submitted in partial fulfillment ... Master of Science in Public Health ... /Beckerman, Jacob M. January 1939 (has links)
Thesis (M.S.P.H.)--University of Michigan, 1939.
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Vitamin C and milk a dissertation submitted in partial fulfillment ... Master of Science in Public Health ... /Beckerman, Jacob M. January 1939 (has links)
Thesis (M.S.P.H.)--University of Michigan, 1939.
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Inhibition of DNA repair by sodium ascorbate in vitro and in vivoKoropatnick, Donald James January 1981 (has links)
Several short-term assays are in use to assess the carcinogenic hazard of chemicals. While the ability to induce initiating events that may lead to carcinogenesis is measured, compounds and conditions that might modify the ability of chemicals to cause those initiating events are not assessed by such tests. In addition, compounds that affect the ability of cells to react in a normal fashion to the damaging action of carcinogens are not detected by these methods.
Shifts in alkaline sucrose gradient profiles of centrifuged DNA (as an indication of DNA fragmentation) and formation of aryl and alkyl DNA adducts (as an indication of DNA modification) have been used as short-term assays for carcinogenic and mutagenic potential. Repair of DNA damage has been measured by restoration of near-control sedimentation profiles of DNA and the loss of aryl and alkyl adducts over time after damage or modification of DNA by carcinogens and mutagens.
In this study, the ability of sodium ascorbate to modify the DNA fragmenting and adduct-forming action of the carcinogens N-methyl-N'-nitro-N-nitrosoguanidine (MNNG) and benzo(a)pyrene (BP) was investigated. In addition, the ability of cells In vivo and In vitro to repair DNA in the presence of sodium ascorbate was assessed by the two methods described above.
It was found that sodium ascorbate inhibited repair in vivo and in vitro. In addition, sodium ascorbate was found to fragment DNA in vivo and in vitro in the presence of copper, and to inhibit the action of carcinogens _in vivo and in vitro by nucleophilic
scavenging of electrophilic carcinogens.
Sodium ascorbate was also found to inhibit the binding of BP to DNA jLn vivo and In vitro. On the other hand, other reducing agents had other effects. Propyl gallate (a sulphydryl reducing compound) inhibited binding of BP to DNA in vitro, but enhanced binding of BP to DNA in vivo. The sulphydryl reducing agent glutathione enhanced binding of BP to DNA in vivo and in vitro.
Alkaline sucrose gradient analysis of DNA damage and recovery from that damage, and BP adduct formation in DNA and disappearance over time, appear to be suitable methods for assessment of the modifying properties of compounds and conditions on the initiating events that may lead to mutation or carcinogenesis. / Medicine, Faculty of / Medical Genetics, Department of / Graduate
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The effects of a dietary supplement of fresh oranges on the oral health of childrenDilley, Gary J. January 1973 (has links)
Indiana University-Purdue University Indianapolis (IUPUI) / The effects of additional citrus fruit in the diet on the
periodontium have been a debated subject for some time. This
study attempted to measure the effects of eating three additional
oranges per day by 123 children ages six through twenty years and
an equal number of controls over a 23-week period. To measure any
changes that might take place, the following were evaluated clinically,
and the decayed, missing and filled surfaces were also evaluated
radiographically:
1. gingival status
2. plaque formation
3. D.M.F.S. and d.m.f.s.
4. white spots
Results after the 23 week test period showed that the gingival
scores increased significantly in both groups (increased inflammation).
The plaque formation score also increased in both groups, but only
the non-orange eaters' score increased significantly over their
original score and over the orange eaters' score. The decayed,
missing, and filled surfaces and white spots did not change significantly
in either group.
Therefore with this study sample over the 23-week test
period, the additional oranges in the diet had limited measurable
effect on the hard and soft tissues of the oral cavity.
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