Pauline Melville’s The Ventriloquist’s Tale is an intricately layered novel in which the myths and folktales of the Amerindians of Guyana, as they are represented in Melville’s novel, are engaged in a dialogue with their reality. This narrative/mythical dialogue results in enactments and re-enactments of the myths and folktales, not only retelling them, but also recycling them, resulting in the Amerindians interpreting their myths and folktales nonmetaphorically. Mikhail Bakhtin’s concept of settings as chronotopes, “timespaces” in which time and space are inseparable from each other and from the theme, is used to define the distinct thematic qualities of the three narrative layers in the novel. I label these three chronotopes unfixed space, the juncture, and the interior. The interior is established as the chronotope in which the enactments and reenactments of myths and folktales primarily take place, re/enactments which add yet another layer to the novel. I argue that the reason the chronotope of the interior is the nexus of these myths and folktales is largely because the Amerindians adhere to a concept of time which is cyclical rather than linear. The enactments and reenactments are then unfolded as intentionally complex and contradictory threads, which are then untangled to show how the myths and folktales are recycled in the novel. This untangling reveals how the threads interconnect, and how they can all be traced back to the narrator, the trickster deity Macunaima, suggesting he is as unbound by temporal and spatial limitations as the narrative layer of myths and folktales from which he has emerged.

The function of CABP1, a novel cAMP-binding protein in Dictyostelium discoideum, has been investigated. Previously, it has been shown that the molecular weight of CABP1 varies in different strains and that this variation is correlated with differences in response to cAMP. Sequence analysis showed that compared to its counterpart in strain AX2, the gene encoding CABP1 of strain V12M2 is missing 27 bp. AX2 transformants expressing the capA gene of V12M2 exhibited no change in phenotype, suggesting that CABP1 is not involved in the features examined or that the V12M2 variant is recessive. Disruption of capA by homologous recombination was attempted with no success, suggesting that this gene might be essential. Proteins interacting with CABP1 have been identified by immunoprecipitation using specific antibodies, CABP1 was also found to copurify with annexin VII, a calcium-dependent phospholipid-binding protein, suggesting that CABP1 and annexin VII might interact.

Thesis (Ph. D.)--Washington State University, May 2008. / Includes bibliographical references.

Thesis (Ph. D.)--Cornell University, May, 2008. / Vita. Includes bibliographical references (leaves 133-142).

Thesis (Honors)--College of William and Mary, 2008. / Includes bibliographical references (leaves 78-79). Also available via the World Wide Web.

Thesis (Ph. D.)--Ohio State University, 2004. / Title from first page of PDF file. Document formatted into pages; contains xxiv, 256 p.; also includes graphics (some col.). Includes bibliographical references (p. 228-256).

Thesis (M.Phil.)--Hong Kong University of Science and Technology, 2004. / Includes bibliographical references (leaves 148-151). Also available in electronic version. Access restricted to campus users.

Thesis (Ph. D.)--University of Wisconsin--Madison, 1971. / Typescript. Vita. Description based on print version record. Includes bibliographical references (leaves 92-107).

Thesis (Ph.D.) - University of Queensland, 2003. / Includes bibliography.

Thesis (Ph. D.)--University of Wisconsin--Madison, 1966. / Vita. Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.