Global ETD Search

71	Advanced MRI for cardiac assessment in mice Buonincontri, Guido January 2014 (has links) No description available. 612.8
72	Pathogenic mechanisms of cigarette smoking on ulcerative colitis-associated neoplasia in mice 廖兆霖, Liu, Shiu-lam, Edgar. January 2003 (has links) published_or_final_version / Pharmacology / Doctoral / Doctor of Philosophy Colon (Anatomy) - Cancer - Etiology. Rectum - Cancer - Etiology. Tobacco - Physiological effect. Mice as laboratory animals.
73	Effects of excretory/secretary molecules of trichinella spiralis andT. pseudospiralis (nematoda) on host immune response 孫建維, Sun, Kin-wai. January 2002 (has links) published_or_final_version / Zoology / Master / Master of Philosophy Trichinella spiralis. Nematoda. Host-parasite relationships. Immune response. Mice as laboratory animals.
74	Genetic and pharmacological approaches to study the role of the polyolpathway enzymes in diabetic and ischemic retinopathy Cheung, Kwok-ho, Alvin, 張國豪 January 2007 (has links) published_or_final_version / abstract / Anatomy / Doctoral / Doctor of Philosophy Polyols. Diabetic retinopathy. Eye - Diseases - Genetic aspects. Eye - Diseases - Pathogenesis. Mice as laboratory animals.
75	Indian hedgehog stimulates chondrocyte hypertrophic differentiation inendochondral bone formation Li, Jun January 2007 (has links) published_or_final_version / abstract / Biochemistry / Doctoral / Doctor of Philosophy Cartilage cells. Endochondral ossification. Bone - Hypertrophy. Genes. Mice as laboratory animals.
76	Investigating the molecular mechanisms of campomelic dysplasia in a mouse with a Sox9 gene mutation Au, Y. K., Tiffany. January 2007 (has links) published_or_final_version / abstract / Orthopaedics and Traumatology / Doctoral / Doctor of Philosophy Dysplasia. DNA-binding proteins. Gene expression. Molecular genetics. Mice as laboratory animals.
77	Polyol pathway contributes to iron-induced oxidative damage in ischemia-reperfused rat hearts 鄧偉豪, Tang, Wai-ho, Jack. January 2007 (has links) published_or_final_version / abstract / Physiology / Master / Master of Philosophy Polyols. Reperfusion injury. Ischemia. Iron - Physiological effect. Mice as laboratory animals.
78	Targeted disruption of exchange protein directly activated by cAMP 1 in mice leads to altered glucose homeostasis Kai, Ka-lun, Alan., 奚家麟. January 2008 (has links) published_or_final_version / Anatomy / Master / Master of Philosophy Cyclic adenylic acid. Cellular signal transduction. Pancreatic beta cells. Hyperglycemia. Mice as laboratory animals.
79	INFLUENCE OF CYCLOPHOSPHAMIDE ON THE RESPONSE OF MICE TO BOVINE AND PORCINE TRANSFER FACTOR. CLARK, PAUL DOUGLAS. January 1982 (has links) Cyclophosphamide (CY) was injected intraperitoneally into mice at varying dosage levels 48 hours prior to intravenous inoculation of constant amounts of dialysable leucocytic transfer factor (TF). Bovine TF to coccidioidin and Brucella abortus and porcine TF to Mycobacterium avium were used. Twenty-four hours after TF injections, mice were tested for delayed hypersensitivity by footpad inoculation with specific antigens. The results show that CY slightly enhanced the delayed hypersensitivity response to the specific test antigens in all three systems. This was not true at every CY dose level, however, since there was a correlation between the dosage of CY and the time of peak footpad swelling. Cyclophosphamide at a low level (20 mg/kg body wt) caused the maximal reading at 24 hours after test, whereas the high level dose (200 mg/kg) showed greatest thickening of the footpad at 48 hours. Transfer factor dosage differences appeared to change the response relatively little, if at all. The cyclophosphamide dose response, on the other hand, showed disparities among various antigen systems and at the different observation times. Under these conditions, no consistent, striking effects of CY on transfer could be noted. Transfer factor (Immunology) Immune response. Cyclophosphamide. Mice as laboratory animals. Immunosuppressive agents.
80	Early embryos of dams of heat stress Johnsen, Suzanne Louise, 1960- January 1989 (has links) Increased environmental heat causes early embryonic death before implantation. This study was designed to examine tissues of dams exposed to environmental temperatures of 36°C and to examine 72 hour old embryos from these dams. Results showed adult mice exposed to heat stress had significant changes in liver morphology with hepatocyte swelling and vacuolization of the cytoplasm, organelles in the hepatocytes were displaced next to the cell membrane. After 48 hours of recovery from heat stress, liver morphology appeared normal. Embryos from heat stressed dams had delayed development indicated by increased 2alpha helical cellular inclusions. Embryos responded differently to different fixation techniques indicated permeability changes in either the zona pellucida or cellular membranes. Litter size or pup survivability from heat stressed dams allowed to recover indicated changes seen at this point were reversible Heat -- Physiological effect. Fetus -- Growth. Fetal death. Mice as laboratory animals.

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