Thesis (M.S.) -- Central Connecticut State University, 2008. / Thesis advisor: Cynthia Pope. "... in partial fulfillment of the requirements for the degree of Master of Science in Geography." Includes bibliographical references (leaves 85-90). Also available via the World Wide Web.

Thesis (M.A.)--The University of North Carolina at Greensboro, 2008. / Directed by Rebecca Adams; submitted to the Dept. of Sociology. Title from PDF t.p. (viewed Aug. 26, 2009). Includes bibliographical references (p. 147-156).

Thesis (M.A.)--Acadia University, 1998. / Includes bibliographical references. Also available on the Internet via the World Wide Web.

Thesis (M.A.)--Acadia University, 1998. / Includes bibliographical references. Also available on the Internet via the World Wide Web.

published_or_final_version / Medical Sciences / Master / Master of Medical Sciences

No description available.

University of Macau / Faculty of Health Sciences

Thesis (Ph. D.(Psychology))--University of Pretoria, 2003. / Includes bibliographical references (leaves 310-353).

It has been suggested that infection with Chlamydia may play a role in the initiation/progression of Alzheimer’s disease (AD). To evaluate this hypothesis APP/PS transgenic mice (genetically manipulated to express AD pathology) and wild type (Wt) mice were infected with C. muridarum, and magnetic resonance imaging (MRI) and histopathology were used to assess pathological changes. Congo red staining of tissue sections demonstrated no AD plaque pathology in Wt infected and non-infected mice, while clear pathology (neuritic plaques) was seen in transgenic mice, with a trend towards higher plaque counts in the brains in the infected transgenic mice. When MRI was used to evaluate the effects of infection in vivo, hyperintensities in T2 times were observed in APP/PS infected mice compared to APP/PS control mice both at month 5 and month 20. Together these results suggest that infection with Chlamydia may accelerate the development of AD.

Non-family caregivers are healthcare professionals who work with individuals who have Alzheimer’s disease (AD). Focusing on non-family caregiver perspectives, the research in this thesis provided an opportunity to explore ways they perform their jobs and interact with residents in the built environment. Non-family caregiver’s knowledge and experience with spatial advantages and disadvantages provides important information that could be used in the design of future AD care facilities. Unfortunately, there appears to only be a small number of peer reviewed publications about interior design research focusing specifically on design for AD. The literature is limited on the topic of non-family caregivers and how their knowledge and experience may contribute to the design process and impact the outcome of the built environment. In order to respond to these challenges, designers should give consideration to the benefits associated with engaging the input of those who work closely with individuals in AD care environments.