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Eye movement measurement for clinical applications using pattern recognition甄榮輝, Yan, Wing-fai. January 1988 (has links)
published_or_final_version / Electrical Engineering / Master / Master of Philosophy
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OPTIMIZATION AND STOCHASTIC APPROXIMATION TECHNIQUES APPLIED TO UNSUPERVISED LEARNINGMcClellan, Richard Paul, 1944- January 1971 (has links)
No description available.
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AUTOMATIC RECOGNITION OF PICTORIAL-PATTERNS BY DESCRIPTIVE ANALYSIS AND CLASSIFICATIONTodd, Henry Swan, 1933- January 1973 (has links)
No description available.
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Pattern recognition by simulating parallel plane threshold devicesBarr, James Robert, 1936- January 1965 (has links)
No description available.
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Probabilistic second level continuous evaluator of adaptive pattern recognizersStratton, William Roland, 1939- January 1967 (has links)
No description available.
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A comparison of size and shape of certain commercially available dress patternsDisney, Phyllis Ann, 1934- January 1961 (has links)
No description available.
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Feature extraction and evaluation for cervical cell recognitionCahn, Robert L. January 1977 (has links)
No description available.
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Combined top-down and bottom-up algorithms for using context in text recognitionBouchard, Diana C. January 1979 (has links)
No description available.
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Subversion, transcendence, and rejection history in the fiction of contemporary Chinese avant-garde writers Su Tong, Yu Hua, and Ge FeiYu, Zhansui 05 1900 (has links)
This thesis explores the different patterns of history presented in the fiction of the three major contemporary Chinese avant-garde writers Su Tong, Yu Hua, and Ge Fei as well as their respective views of history. Based on detailed case studies of the three writers, the thesis examines the complicated and intertwined relationships of contemporary Chinese avant-garde fiction with previous Chinese traditions—Confucian, the May Fourth, and Communist—and with foreign influences. It also assesses the overall literary achievement of Chinese avant-garde fiction, its position in the history of modern Chinese literature, and its impact on the Chinese writers of later generations.
Unlike most previous research on this subject, which overemphasizes the "alien" nature of Chinese avant-garde fiction or its discontinuity with Chinese tradition, this thesis aims at a more balanced investigation. Not only is the "newness" of Chinese avant-garde fiction deeply explored, its "Chineseness" or its profound continuity with Chinese literary and cultural conventions is also carefully examined. By comparison, the thesis attaches more importance to the "Chineseness" of Chinese avant-garde fiction.
My analysis demonstrates that, while Su Tong aims at the total subversion of the Communist interpretation of the Chinese revolution and history, while Yu Hua attemptsto transcend the Maoist materialistic view of history through reincorporating subjectivity into historical interpretation, Ge Fei totally rejects the conceptualization of history and the underlying rationalistic assumption of human experience as a perceptible and understandable unity.
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A reexamination of the role of the hippocampus in object-recognition memory using neurotoxic lesions and ischemia in ratsDuva, Christopher Adam 11 1900 (has links)
Paradoxical results on object-recognition delayed nonmatching-to-sample (DNMS)
tasks have been found in monkeys and rats that receive either partial, ischemia-induced
hippocampal lesions or complete hippocampal ablation. Ischemia results in severe DNMS
impairments, which have been attributed to circumscribed CA1 cell loss. However,
ablation studies indicate that the hippocampus plays only a minimal role in the performance
of the DNMS task. Two hypotheses have been proposed to account for these discrepant
findings (Bachevalier & Mishkin, 1989). First, the "hippocampal interference" hypothesis
posits that following ischemia, the partially damaged hippocampus may disrupt activity in
extrahippocampal structures that are important for object-recognition memory. Second,
previously undetected ischemia-induced extrahippocampal damage may be responsible for
the DNMS impairments attributed to CA1 cell loss.
To test the "hippocampal interference" hypothesis, the effect of partial NMDAinduced
lesions of the dorsal hippocampus were investigated on DNMS performance in
rats. These lesions damaged much of the same area, the CA1, as did ischemia; but did so
without depriving the entire forebrain of oxygen, thereby reducing the possibility of
extrahippocampal damage. In Experiment 1, rats were trained on the DNMS task prior to
receiving an NMDA-lesion. Postoperatively, these rats reacquired the nonmatching rule at
a rate equivalent to controls and were unimpaired in performance at delays up to 300 s. In
Experiment 2, naive rats were given NMDA-lesions and then trained on DNMS. These
rats acquired the DNMS rule at a rate equivalent to controls and performed normally at
delays up to 300 s. These findings suggest that interference from a partially damaged
hippocampus cannot account for the ischemia-induced DNMS impairments and that they
are more likely produced by extrahippocampal neuropathology. In Experiment 3, rats from
the previous study were tested on the Morris water-maze. Compared to sham-lesioned
animals, rats with partial lesions of the dorsal hippocampus were impaired in the
acquisition of the water-maze task. Thus, subtotal NMDA-lesions of the hippocampus
impaired spatial memory while leaving nonspatial memory intact.
Mumby et al. (1992b) suggested that the ischemia-induced extrahippocampal
damage underlying the DNMS deficits is mediated or produced by the postischemic
hippocampus. To test this idea, preoperatively trained rats in Experiment 4 were subject to
cerebral ischemia followed within 1hr by hippocampal aspiration lesions. It was
hypothesized that ablation soon after ischemia would block the damage putatively produced
by the postischemic hippocampus and thereby prevent the development of postoperative
DNMS deficits. Unlike "ischemia-only" rats, the rats with the combined lesion were able to
reacquire the nonmatching rule at a normal rate and performed normally at delays up to 300
s. Thus, hippocampectomy soon after ischemia eliminated the pathogenic process that lead
to ischemia-induced DNMS deficits. Experiment 5 investigated the role of ischemiainduced
CA1 cell death as a factor in the production of extrahippocampal neuropathology.
Naive rats were given NMDA-lesions of the dorsal hippocampus followed 3 weeks later by
cerebral ischemia. If the ischemia-induced CA1 neurotoxicity is responsible for producing
extrahippocampal damage then preischemic ablation should attenuate this process and
prevent the development of DNMS impairments. This did not occur: Rats with the
combined lesion were as impaired as the "ischemia-only" rats in the acquisition of the
DNMS task. This suggests that the ischemia-induced pathogenic processes that result in
extrahippocampal neuropathology comprise more than CA1 neurotoxicity.
The findings presented in this thesis are consistent with the idea that ischemiainduced
DNMS deficits in rats are the result of extrahippocampal damage mediated or
produced by the postischemic hippocampus. The discussion focuses on three main points:
1) How might the post-ischemic hippocampus be involved in the production of
extrahippocampal neuropathology? 2) In what brain region(s) might this damage be
occurring? 3) What anatomical, molecular, or functional neuropathology might ischemia
produce in extrahippocampal brain regions? The results are also discussed in terms of a
specialized role for the hippocampus in mnemonic functions and the recently emphasized
importance of the rhinal cortex in object-recognition memory.
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