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Thesis (MHSc--Health Science) -- Auckland University of Technology, 2003. / Also held in print (73 leaves, 30cm.) in Akoranga Theses Collection (T 615.82 HOE)

Thesis (M.D.)--Boston University

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Thesis (Ph. D.)--University of Texas at Austin, 2000. / Vita. Includes bibliographical references (leaves 119-126). Available also in a digital version from Dissertation Abstracts.

Cerebral palsy (CP) is a heterogeneous disorder in which movement and posture are affected. Increased excitation of the central nervous system leads to neural symptoms, which can cause spasticity and muscle weakness. These neural abnormalities result in secondary CP-related mechanical adaptations of muscles and tendons, which can lead to muscle contracture, joint deformities and pain. Therapeutic interventions are therefore essential to treat CP-induced abnormalities. Passive stretching in particular is a popular treatment method in clinical practice. However, due to a lack of scientific evidence, clinicians often have to make assumptions about the mechanical adaptability of muscles and tendons. Currently, the mechanical properties of muscles and tendons in children with CP and their adaptability are not well understood, which makes it difficult to implement evidence-based practice in clinical settings. Therefore, the overall purpose of this research was to examine the mechanical properties of the medial gastrocnemius muscle and Achilles tendon in children with spastic CP, and the adaptations of the muscle and tendon to acute and long-term passive stretching. The first experimental Chapter (3) was carried out in healthy adults, to assess the agreement between two methods of deriving Achilles tendon stiffness (i) active contraction of the triceps surae muscles to elongate the Achilles tendon, or (ii) passive rotation of the ankle joint. Taking into consideration the tendon’s viscoelastic response, the effects of strain-rate on Achilles tendon stiffness were also described. Results revealed that tendon stiffness measured using the “active method” was 6% greater than the “passive method”. There was also a significant increase in Achilles tendon stiffness in response to increased strain-rate. As the more commonly used active method is problematic to be used in children with CP, due to muscle weakness and excessive co-contraction, the passive method of deriving tendon stiffness was used in subsequent experimental studies. In experimental Chapter 4, differences in the mechanical properties of the Achilles tendon and triceps surae muscles between children with CP and their typically developing (TD) peers, were investigated. The results revealed that estimates of triceps surae muscle stiffness were significantly greater in children with CP compared to TD children. The results also showed that despite a smaller tendon cross-sectional area in children with CP, Achilles tendon stiffness was not different between groups. In addition, children with CP had a steeper tendon stiffness-strain-rate relationship compared to TD children. These results have significant clinical implications regarding the diagnosis of spasticity using the current clinical methods. Experimental Chapters 5 and 6 examined the muscle’s and tendon’s response to stretch. Passive stretching, implemented by a clinician or by the children themselves, is a commonly used intervention for children with CP with the aim of inducing structural alterations in muscles and tendons to improve function. In order for these alterations to take place, elongation of the muscle and fascicles would presumably need to occur with acute stretching. To date, this assumption has not been tested. Thus, the purpose of Chapter 5 was to investigate the medial gastrocnemius and muscle fascicle response to acute stretching, using two commonly used stretch techniques. Results of this study revealed that 100 s of stretching caused a transient increase in tendon (1.0 cm), muscle (0.8 cm) and fascicle lengths (0.6 cm). This effect was independent of stretch technique. These results provide evidence that the muscle and fascicles are capable of elongating in response to stretch in children with spastic CP. They provide a basis for the hypothesis that the spastic muscle may be able to adapt in response to long-term stretching. Thus, the purpose of the final experimental Chapter (6) was to assess the effects of a six week passive stretching intervention (four days per week, 15 minutes per day) on muscle and tendon properties, and gait parameters in children with CP. Results revealed there was a significant reduction in joint stiffness in the experimental group following six weeks of stretching. This was accompanied by a reduction in muscle stiffness, but with no alterations in Achilles tendon stiffness. Additionally, there were no positive effects of passive stretching on gait parameters. Together, the results of the present series of investigations demonstrates how fundamental knowledge of muscle and tendon mechanics in children with spastic CP, can be implemented to support evidence-based clinical practice.

Title: The Use of Physiotherapy in Spasticity Management Objectives: The purpose of this thesis is to provide a summary of present findings on spasticity, with particular regard to its pathophysiological mechanisms and clinical manifestations, and to present an overview of medical and physiotherapeutic approaches applied in its treatment. Furthermore, it aims at making a research in physiotherapeutic methods and techniques used for reduction of spasticity. The thesis shall present relevant studies, provide their comparison and critically evaluate the effect of methods and techniques examined in them. Methods: This thesis is a descriptive analysis based on a literature review. Results: Based on the research findings, it may be concluded that physiotherapy has proven to be effective in reducing spasticity. However, it is not possible to determine whether the techniques to reduce spasticity are more effective than techniques without this primary purpose. The examined studies have shown that the choice of physiotherapeutic practices to reduce spasticity was not influenced by disease etiology. Moreover, the same techniques and methods were applied in pediatric and adult patients, regardless of diagnosis. The relationship between reduced spasticity and change in motor function is unclear and our research...

The stiff-knee gait pattern of children diagnosed with cerebral palsy (CP) is assumed to be caused by spasticity of the quadriceps which interferes with knee flexion normally occurring during the swing phase of walking. In current clinical practice, the ability to assess quadriceps spasticity is limited by the lack of an objective and reliable test that discriminates the role spasticity plays in functional limitations. The primary purpose of this series of studies was to assess the pendulum test as an objective measure of quadriceps spasticity. The first study assessed the reliability of the pendulum test. Moderate to very high between day reliability for all thirteen measures of the pendulum test were found. The second study assessed the discriminant ability of the pendulum test to correctly identify a stiff-knee gait pattern. Because most clinicians do not have access to a three dimensional motion analysis system, the third study assessed the reliability and accuracy of visual observation of the pendulum test. Sixty-eight children with a primary diagnosis of cerebral palsy participated. A three-dimensional motion analysis system was used to measure the subjects knee motion while walking, and performing the pendulum test. Spasticity of the quadriceps was also assessed using traditional clinical measures i.e., the modified Ashworth scale (MAS), and the Ely tests. Forty-seven percent of the variance in the stiff-knee gait pattern was explained by a regression model using the pendulum test and traditional clinical measures. The only significant measure in the regression model was the magnitude of knee motion occurring during the first swing of the pendulum test (A1). Discriminant analysis revealed the A1 measure correctly classified 77% of the subjects knee-gait pattern. Four observers demonstrated moderate accuracy and repeatability in estimating A1 value. The visual assessment of A1 correctly classified 72-76% of the subjects knee-gait pattern with no prior knowledge of the subjects gait. This series of studies demonstrated the pendulum test is an objective, repeatable measure of quadriceps spasticity. A negative pendulum test (indicated by an A1 value greater than 45 degrees) is more useful for ruling out a stiff-knee gait pattern compared to the traditional clinical measures.

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