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Thoracic Spinal Cord Stimulation Protects the Hippocampus in a Canine Model of Heart FailureSzebeni, Katalin, Szebeni, Attila, DiPeri, T., Davis, N., Ordway, Gregory A., Ardell, J. L. 01 January 2013 (has links)
No description available.
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Growth Factors Involved in the Regulation of Neurons and Glial Cells in the Rat Spinal CordMcCartney, Annemarie McMillan 09 May 2007 (has links)
No description available.
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c-Fos Expression in Rat Brain Stem and Spinal Cord in Response to Activation of Cardiac Ischemia-Sensitive Afferent Neurons and Electrostimulatory ModulationHua, Fang, Harrison, Theresa, Qin, Chao, Reifsteck, Angela, Ricketts, Brian, Camel, Charles, Williams, Carole A. 01 December 2004 (has links)
The purpose of this study was to identify central neuronal sites activated by stimulation of cardiac ischemia-sensitive afferent neurons and determine whether electrical stimulation of left vagal afferent fibers modified the pattern of neuronal activation. Fos-like immunoreactivity (Fos-LI) was used as an index of neuronal activation in selected levels of cervical and thoracic spinal cord and brain stem. Adult Sprague-Dawley rats were anesthetized with urethane and underwent intrapericardial infusion of an "inflammatory exudate solution" (IES) containing algogenic substances that are released during ischemia (10 mM adenosine, bradykinin, prostaglandin E2, and 5-hydroxytryptamine) or occlusion of the left anterior descending coronary artery (CoAO) to activate cardiac ischemia-sensitive (nociceptive) afferent fibers. IES and CoAO increased Fos-LI above resting levels in dorsal horns in laminae I-V at C2 and T4 and in the caudal nucleus tractus solitarius. Dorsal rhizotomy virtually eliminated Fos-LI in the spinal cord as well as the brain stem. Neuromodulation of the ischemic signal by electrical stimulation of the central end of the left thoracic vagus excited neurons at the cervical and brain stem level but inhibited neurons at the thoracic spinal cord during IES or CoAO. These results suggest that stimulation of the left thoracic vagus excites descending inhibitory pathways. Inhibition at the thoracic spinal level that suppresses the ischemic (nociceptive) input signal may occur by a short-loop descending pathway via signals from cervical propriospinal circuits and/or a longer-loop descending pathway via signals from the nucleus tractus solitarius.
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Management of thoracoabdominal aortic aneurysms and dissections : with emphasis on spinal cord protection in aneurysm repair and non-surgical treatment of type-B dissection /Winnerkvist, Anders, January 2006 (has links)
Diss. (sammanfattning) Stockholm : Karol. inst., 2006. / Härtill 6 uppsatser.
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