Spelling suggestions: "subject:"anatomy anda physiology"" "subject:"anatomy ando physiology""
231 |
Characterization of Inhbb, Heatr5a, & Cyp2s1 Expression in Dorsal Root Ganglia by In-Situ HybridizationKrech, Joshua D. 03 June 2021 (has links)
No description available.
|
232 |
Lobster (Panulirus interruptus) Striated Muscle SarcomeresExpand Non-Uniformly During Passive LengtheningFender, John Matthew 15 June 2022 (has links)
No description available.
|
233 |
Mechanisms of Estrogen Signaling in the Colonic EpitheliumPereda, Guillermo 23 May 2022 (has links)
No description available.
|
234 |
Fatemapping of Urothelial Cell Lineages During Normal Kidney Development and Renal PathogenesisMosley, Claudia Foulk January 2018 (has links)
No description available.
|
235 |
The Role of IGF-1 In Geriatric SkinCastellanos, Amber 19 May 2020 (has links)
No description available.
|
236 |
The Skeletal Biology of Hibernating Woodchucks (<i>Marmota monax</i>)Doherty, Alison H. 28 March 2013 (has links)
No description available.
|
237 |
The Effects of Two Analgesic Balm Applications on Pain and Psychosocial Factors Related to InjurySpring, Leigh T. 26 September 2013 (has links)
No description available.
|
238 |
Ontogeny of the Brain Endocasts of Ostriches (Aves: Struthio camelus) with Implications for Interpreting Extinct Dinosaur EndocastsRomick, Cheyenne Ariel January 2013 (has links)
No description available.
|
239 |
Growth factor expression and release in the ischemic heartVos, Lynette Christine 01 January 2004 (has links) (PDF)
The angiogenic and cardioprotective effects of basic fibroblast growth factor (FGF-2) and vascular endothelial growth factor (VEGF) in the ischemic myocardium have been studied, but expression and release of endogenous FGF-2 and VEGF during myocardial ischemia are poorly understood. In addition, nitric oxide synthase isoforms eNOS and iNOS may play a role in myocardial ischemia. The purpose of this study was to investigate the release of FGF-2 and expression of FGF-2, VEGF, eNOS, and iNOS in the normal and ischemic heart. In Phase I, serum FGF-2 levels in patients undergoing treadmill stress test were measured to investigate correlation between serum FGF-2 levels and presence of ischemic heart disease. The study found that serum FGF-2 in ischemia-positive and ischemia-negative patients was not significantly elevated after treadmill stress test, and serum FGF-2 levels did not differ significantly between ischemia-positive and ischemia-negative patients. In Phase II, FGF-2 levels in coronary effluent from isolated perfused rabbit hearts subjected to low-flow ischemia was measured. Results suggest that FGF-2 is released into the coronary effluent of isolated perfused hearts over time and that this release may be elevated in ischemic (50% flow) hearts. Furthermore, the present study indicates that FGF-2 is released immediately after surgical isolation and instrumentation of the isolated heart. A linear model was developed to describe the release of FGF-2 from the isolated heart as a function of the coronary flow rate Q : [special characters omitted]where t = time and Q = 1 and 3.01 for normal and 50% flow rates respectively. In Phase III, effect of acute low-flow ischemia on FGF-2, VEGF, eNOS, and iNOS mRNA expression was measured in isolated perfused hearts using RT/PCR. Preliminary results indicate that FGF-2, VEGF, and iNOS mRNA expression is upregulated and eNOS expression is decreased in ischemic hearts suggesting that these growth factors play a role in short-term response of the myocardium to ischemia. The results of this study suggest that FGF-2, VEGF, and iNOS mRNA expression are increased, eNOS expression is decreased, and FGF-2 is released in response to low-flow ischemia in the isolated perfused heart.
|
240 |
The Central Nervous System Aspects of Cardiac Arrest and Resuscitation in a Rat Model of Global IschemiaXu, Kui 06 July 2010 (has links)
No description available.
|
Page generated in 0.3143 seconds