Digitized by Kansas Correctional Industries

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by Wong Wah Fung. / Thesis (M.Phil.)--Chinese University of Hong Kong, 1996. / Includes bibliographical references (leaves 53-54). / Introduction --- p.2 / Chapter 1 --- Preliminaries --- p.3 / Chapter 1.1 --- A Summary on C*-algebras --- p.3 / Chapter 1.2 --- Hereditary C*-subalgebras --- p.5 / Chapter 1.3 --- C*-Inductive Limit --- p.7 / Chapter 1.4 --- Basic K-Theory of C*-algebras --- p.9 / Chapter 2 --- C*-algebras of Real Rank Zero --- p.14 / Chapter 2.1 --- Basic Properties of C*-algebras with Real Rank Zero --- p.14 / Chapter 2.2 --- Strongly Morita Equivalence and Extensions --- p.30 / Chapter 3 --- Simple C*-algebras --- p.42 / Chapter 3.1 --- Basic Properties --- p.42 / Chapter 3.2 --- Cuntz Algebras --- p.51 / Bibliography --- p.53

by Chau Man Pan. / Thesis (M.Phil.)--Chinese University of Hong Kong, 2003. / Includes bibliographical references (leaves 74-75). / Abstracts in English and Chinese. / Chapter 1 --- Preliminaries --- p.6 / Chapter 1.1 --- General Theory of C* algebras --- p.6 / Chapter 1.2 --- Hilbert Modules and Induced representations --- p.11 / Chapter 1.3 --- Crossed Products --- p.15 / Chapter 2 --- Twisted crossed products --- p.18 / Chapter 2.1 --- Basic definition --- p.18 / Chapter 2.2 --- Iterated twisted crossed products --- p.24 / Chapter 3 --- Induced representations --- p.27 / Chapter 3.1 --- Construction of Imprimitivity bimodule --- p.27 / Chapter 3.2 --- Basic theory about induced representations --- p.32 / Chapter 4 --- Ideal Theory --- p.38 / Chapter 4.1 --- Induction and Restriction processes --- p.38 / Chapter 4.2 --- Sub-quotients of twisted crossed products --- p.48 / Chapter 5 --- Mackey Machine --- p.53 / Chapter 5.1 --- Quasi regular systems --- p.53 / Chapter 5.2 --- First Step of Mackey Machine --- p.57 / Chapter 5.3 --- Second step of Mackey Machine --- p.59 / Chapter 6 --- Abelian systems --- p.64 / Chapter 6.1 --- Dual spaces of Abelian systems --- p.64 / Chapter 7 --- Appendix --- p.69 / Chapter 7.1 --- Classical version of induced representation --- p.69

Made available in DSpace on 2014-06-12T15:52:55Z (GMT). No. of bitstreams: 2 arquivo5108_1.pdf: 4356374 bytes, checksum: 821d83f4d72de4ed3c01344036390c23 (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue date: 2004 / O presente trabalho estudou o efeito da deficiência em ácido ascórbico (AA) em cobaios machos adultos, durante 21 dias, sobre o peso e a histoarquitetura (histologia e histometria) do testículo e cauda do epidídimo, sobre a qualidade (número, motilidade e morfologia) dos espermatozóides e sobre os níveis plasmáticos de testosterona e a resposta secretória das células intersticiais testiculares ao estímulo gonadotrófico com hCG e ao tratamento in vitro com AA. A deficiência em AA causou diminuição de 32 % no peso corporal O peso absoluto do testículo foi reduzido em 30,5 % enquanto o seu peso relativo não foi modificado pela deficiência em AA. O peso absoluto e relativo da cauda do epidídimo diminuiram em 44,8 % e 25,2 %, respectivamente. A histoarquitetura dos dois órgãos foi alterada pela carência em AA. O parênquima do testículo apresentou alterações do epitélio germinativo dos túbulos seminíferos. A cauda do epidídimo apresentou diminuição na altura do epitélio colunar de revestimento e do número de espermatozóides na luz tubular. O número e motilidade dos espermatozóides diminuiram (73 % e 27,5 %, respectivamente), como também sua morfologia foi alterada pela deficiência em AA. Os níveis plasmáticos de testosterona foram reduzidos em 72,6 % nos animais deficientes em AA. A produção de testosterona pelas células intersticiais testiculares estimulada com uma dose elevada de hCG (10 mUI/ml) in vitro foi menor nos animais deficientes em AA. O tratamento com uma dose alta de AA (100 μM) in vitro não modificou a secreção basal de testosterona em nenhum dos grupos de animais, mas inibiu a secreção estimulada pelohCG no grupo controle, não modificando a secreção no grupo deficiente em AA. Nossos estudos confirmam prévias referências e fornecem evidências adicionais da importância do AA na manutenção da integridade estrutural e funcional do testículo e epidídimo. Os resultados também indicam que o efeito primário da deficiência em AA parece estar no testículo, reduzindo a produção de testosterona e, conseqüentemente, privando desse hormônio os órgãos da reprodução

馬嘉域. / "2008年2月". / 論文(哲學碩士)--香港中文大學, 2008. / 參考文獻(leaves 116-118). / "2008 nian 2 yue". / Abstract also in English. / Ma Jiayu. / Lun wen (zhe xue shuo shi)--Xianggang Zhong wen da xue, 2008. / Can kao wen xian (leaves 116-118). / 緒論 --- p.5 / Chapter 第一章 --- 規則式道德爲道德建立基礎的嘗試及其失敗 --- p.10 / Chapter 1.1 --- 啓蒙運動的努力一一休謨的嘗試 --- p.10 / Chapter 1.2 --- 啓蒙運動的努力一一康德的嘗試 --- p.14 / Chapter 1.3 --- 爲何啓蒙運動以來的道德哲學必定失敗 --- p.19 / Chapter 1.4 --- 啓蒙運動後的努力一一效益主義及其他 --- p.21 / Chapter 1.5 --- 直覺主義與情感主義一一道德根據的徹底喪失 --- p.23 / Chapter 1.6 --- 小結道德衰退的三階段 --- p.26 / Chapter 第二章 --- 麥肯泰爾論德性的根據和基礎 --- p.28 / Chapter 2.1 --- 德性與實踐活動的關係 --- p.29 / Chapter 2.1.1 --- 內在價値與外在價値 --- p.30 / Chapter 2.1.2 --- 卓越標準 --- p.33 / Chapter 2.1.3 --- 必須依靠他人的協作性活動 --- p.36 / Chapter 2.1.4 --- 對德性初步理解的不足 --- p.39 / Chapter 2.2 --- 個人生活的敘述性整體 --- p.40 / Chapter 2.2.1 --- 個人主義式自我與整體性自我 --- p.41 / Chapter 2.2.2 --- 個別行爲的可理解性 --- p.43 / Chapter 2.3.3 --- 敘述、對話與個人身份 --- p.47 / Chapter 2.3.4 --- 個人生活的敘述式整體 --- p.50 / Chapter 2.3 --- 傳統的槪念 --- p.51 / Chapter 2.4 --- 小結德性論三階段的聯繫 --- p.55 / Chapter 第三章 --- 道德相對主義問題的消解及其困難 --- p.57 / Chapter 3.1 --- 傳統的合理性 --- p.57 / Chapter 3.2 --- 知識論危機 --- p.59 / Chapter 3.3 --- 不同傳統之間的互相了解及相對主義的超越 --- p.60 / Chapter 3.3.1 --- 真理的槪念 --- p.62 / Chapter 3.3.2 --- 可理解性的問題 --- p.64 / Chapter 3.4 --- 小結 --- p.68 / Chapter 第四章 --- 後期麥肯泰爾一一德性倫理學的社會生物學基礎 --- p.69 / Chapter 4.1 --- 人與動物相同的地方 --- p.70 / Chapter 4.1.1 --- 意圖的前語言認識 --- p.72 / Chapter 4.1.2 --- 行爲的理由 --- p.73 / Chapter 4.2 --- 我們如何由動物成長爲人類? --- p.75 / Chapter 4.2.1 --- 價値與成長的關係 --- p.75 / Chapter 4.2.2 --- 人類成長的三個階段 --- p.77 / Chapter 4.2.3 --- 成長三階段的關係 --- p.79 / Chapter 4.3 --- 個人成長的依賴性 --- p.80 / Chapter 4.4 --- 施予及受惠的社群關係 --- p.84 / Chapter 4.4.1 --- 施予及受惠的社群關係與權力分配 --- p.85 / Chapter 4.4.2 --- 施予受惠關係的具體運作 --- p.86 / Chapter 4.5 --- 維持社群關係的德性 --- p.88 / Chapter 4.5.1 --- 正直地慷慨的特徵 --- p.89 / Chapter 4.5.2 --- 德性根據的改變 --- p.90 / Chapter 4.6 --- 小結一一德性與成長的整體圖像 --- p.91 / Chapter 第五章 --- 麥肯泰爾德性論的一些問題 --- p.93 / Chapter 5.1 --- 對待有缺陷人士的應有態度 --- p.94 / Chapter 5.2 --- 德性根據轉變一一社群內親疏關係的問題 --- p.98 / Chapter 5.3 --- 價値衝突的處境 --- p.101 / Chapter 5.4 --- 社群建立的可能性 --- p.105 / 結語 --- p.109 / 參考書目 --- p.116

Chan, Wai-Kit. / Thesis (M.Phil.)--Chinese University of Hong Kong, 2007. / Includes bibliographical references (leaves 52-53). / Abstracts in English and Chinese. / Abstract --- p.ii / Acknowledgement --- p.iii / Introduction --- p.iv / Chapter 1 --- Preliminaries --- p.1 / Chapter 1.1 --- C*-algebras --- p.1 / Chapter 1.2 --- Topological groups --- p.8 / Chapter 2 --- Property T for topological groups --- p.18 / Chapter 2.1 --- Definitions and some basic properties --- p.18 / Chapter 2.2 --- Hereditary properties --- p.23 / Chapter 2.3 --- A characterization for property T --- p.26 / Chapter 2.4 --- Examples --- p.32 / Chapter 3 --- Property T for C*-algebras --- p.34 / Chapter 3.1 --- Countable discrete groups and their group C*- algebras --- p.34 / Chapter 3.2 --- Property T and nuclearity --- p.46 / Bibliography --- p.52

Fanconi Anämie ist eine seltene rezessiv vererbte Erkrankung, deren zu Grunde liegende Enzymdefekte in ein Netzwerk unterschiedlichster DNA-Reparaturproteine eingewoben sind. Phylogenetisch sind uns Vögel relativ nahe verwandt, was sie zu einem guten Modellorganismus jenseits der Säugetiermodelle macht. Eine von Hühnerzellen abgeleitete Zelllinie (DT40) wurde bereits schon breit eingesetzt um die Funktion des FA-Signalwegs zu erforschen. Nachdem auch das Hühnergenom vollständig entschlüsselt wurde, konnten zu fast allen FA-Genen Orthologe gefunden werden. Unter den zahlreichen FA-Genen sind für diese Arbeit vor allem FANCC und -G von Bedeutung, da beide Gene auf dem Z-Geschlechtschromosom des Huhns liegen und eine Inaktivierung des zweiten Z-Chromosoms beim Hahn äquivalent zur X-Inaktivierung beim Menschen nicht stattfindet. Somit sollte es ein ´natürliches´ Gendosisungleichgewicht zwischen den Geschlechtern geben. Im durchgeführten Southern Blot konnte keine geschlechtsspezifisch weibliche Bande (für FANCC und -G) gefunden werden. Somit ist davon auszugehen, dass die FA-Gene C und G ausschließlich auf dem Z-Chromosom lokalisiert sind. Dies wurde auch nochmals mittels FISH bestätigt - beide Gene fanden sich auf dem kurzen Arm des Z-Chromosoms (FANCC zentromernah, FANCG zentromerfern). Aus Studien mit DT40 Zellen ist bereits bekannt, dass FA defiziente Zellen ähnlich wie humane FA-Zellen eine Hypersensitivität gegenüber Substanzen zeigen, die DNA-crosslinks verursachen. In Anlehnung an die humane FA-Diagnostik wurden die neu etablierten embryonalen Fibroblasten mit unterschiedlichen Konzentrationen und Einwirkzeiten von MMC behandelt und die Schäden ausgewertet. In allen Untersuchungen trugen die weiblichen Zellen mehr Schäden davon als die männlichen. Bei niedrigen Konzentrationen zeigte sich dies nur als Trend, bei höheren MMC-Konzentrationen und längeren Einwirkzeiten fanden sich bei fast allen durchgeführten Untersuchungen auch statistisch signifikante Unterschiede. Somit ergibt sich aus dieser Arbeit ein deutlicher Hinweis auf ein funktionelles Ungleichgewicht zwischen Henne und Hahn was die DNA-Reparatur nach Schädigung durch MMC angeht. / Fanconi anemia is a rare recessive disorder whose underlying enzyme deficiencies are woven into a network of various DNA repair proteins. Phylogenetically related birds are relatively close to us, which makes it a good model organism beyond the mammalian models. A cell line derived from chicken cells (DT40) has already been widely used to study the FA pathway. Even after the chicken genome was completely decoded, orthologs could be found for almost all FA genes. Among the numerous FA genes are mainly FANCC and G of importance since both genes are located on the Z sex chromosome of chicken. An inactivation of the second Z chromosome as the inactivation of the x chromosom in human does not take place. Thus there should be a 'natural' imbalance of the gen dose between the sexes. Southern blot showed no gender female band (for FANCC and G). Thus it can be assumed that the FA genes C and G are exclusively localized on the Z chromosome. This was also confirmed by FISH, both genes were located on the short arm of the Z chromosome. From studies in DT40 cells is already known that FA deficient cells, similar to a human FA cell show hypersensitivity to substances that cause DNA crosslinks. Based on the human FA diagnosis, newly established embryonic fibroblasts were treated with different concentrations of MMC and chromosomal the damage was evaluated. In all studies, the female cells contributed more damage than the male. At low concentrations, this was only shown as a trend, at higher MMC concentrations and longer exposure times there was a significant differences. Thus, results from this study, a clear indication of a functional imbalance between hen and rooster in terms of DNA repair after damage by MMC.

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