No description available.

Doctor of Clinical Neuropsychology/Master of Science / Introduction: Parkinson’s disease (PD) is known to cause detrimental effects to motor function and cognition. The motor effects of the disease in turn impact emotion expression in patients with PD. There is conflicting evidence in research, however, as to whether PD also affects emotion comprehension, and if so, what emotions in particular are affected and across what modalities. This study aimed to investigate the effects of PD on a broad range of skills involved in basic and complex emotion comprehension. Whether these effects extend into other areas associated with emotion processing, such as social cognition and autobiographical memory, was also explored. Methods: Sixteen patients with PD participated in the study along with sixteen control subjects who were matched for age, gender, education level and estimated premorbid intelligence. The PD participants, on average, were in the moderate phase of the disease and taking PD medication, including dopamine. Participants were tested on a range of recognition measures including prototypical and morphed facial expressions with reduced intensity (40 and 80%), emotion prosody, written emotion vignettes, emotional imagery, pictures of emotion, social cognition, and a cued autobiographical memory task. A mood inventory was given, and disease severity and duration were noted. Results: The PD group did not show pervasive deficits in emotion recognition overall. Deficits were demonstrated in prosody recognition, specifically with fearful tones, and in an incongruent prosody task, specifically with angry and neutral tones. The PD group was not able to recognise facial expressions of disgust (mixed intensities) as well as controls, with the result showing a trend toward significance. PD participants were also significantly worse in Theory of Mind (TOM) reasoning but not at another social cognition measure involving recognising social emotions through expressions from the eyes only. There were no differences between the groups across all other tests. Discussion: PD is thought to cause subtle deficits in emotion comprehension which are only elucidated through complex tasks. The effects of PD on complex processing also impact TOM performance, which relies on skills involved in complex emotion recognition. Effects of mood and disease factors on performance were circumscribed. Evidence suggested that the basal ganglia and fronto-striatal connections play a role in emotion comprehension.

published_or_final_version / Public Administration / Master / Master of Public Administration

No description available.

No description available.

No description available.

Doctor of Clinical Neuropsychology/Master of Science / Introduction: Parkinson’s disease (PD) is known to cause detrimental effects to motor function and cognition. The motor effects of the disease in turn impact emotion expression in patients with PD. There is conflicting evidence in research, however, as to whether PD also affects emotion comprehension, and if so, what emotions in particular are affected and across what modalities. This study aimed to investigate the effects of PD on a broad range of skills involved in basic and complex emotion comprehension. Whether these effects extend into other areas associated with emotion processing, such as social cognition and autobiographical memory, was also explored. Methods: Sixteen patients with PD participated in the study along with sixteen control subjects who were matched for age, gender, education level and estimated premorbid intelligence. The PD participants, on average, were in the moderate phase of the disease and taking PD medication, including dopamine. Participants were tested on a range of recognition measures including prototypical and morphed facial expressions with reduced intensity (40 and 80%), emotion prosody, written emotion vignettes, emotional imagery, pictures of emotion, social cognition, and a cued autobiographical memory task. A mood inventory was given, and disease severity and duration were noted. Results: The PD group did not show pervasive deficits in emotion recognition overall. Deficits were demonstrated in prosody recognition, specifically with fearful tones, and in an incongruent prosody task, specifically with angry and neutral tones. The PD group was not able to recognise facial expressions of disgust (mixed intensities) as well as controls, with the result showing a trend toward significance. PD participants were also significantly worse in Theory of Mind (TOM) reasoning but not at another social cognition measure involving recognising social emotions through expressions from the eyes only. There were no differences between the groups across all other tests. Discussion: PD is thought to cause subtle deficits in emotion comprehension which are only elucidated through complex tasks. The effects of PD on complex processing also impact TOM performance, which relies on skills involved in complex emotion recognition. Effects of mood and disease factors on performance were circumscribed. Evidence suggested that the basal ganglia and fronto-striatal connections play a role in emotion comprehension.

Thesis (Ph. D.)--Dept. of Economics and Social Science Consortium. University of Missouri--Kansas City, 2006. / "A dissertation in economics and social science consortium." Advisor: L. Randall Wray. Typescript. Vita. Title from "catalog record" of the print edition Description based on contents viewed Oct. 31, 2007. Includes bibliographical references (leaves 184-201). Online version of the print edition.

This study investigates the validity of the twin deficits hypothesis for the Turkish quarterly data over the 1988:1-2005:2 periods. To this end, we consider a VAR variable space containing budget deficits, current account deficits, real output, real interest rates and real exchange rates and employ cointegration, equilibrium/error correction mechanism techniques along with Granger-non-causality tests and impulse response analyses. The empirical results from decompositions of the budget and current account deficits into their cyclical and structural components suggest that both CAD and BD are counter-cyclical. The twin deficit hypothesis, consistent with the conventional Mundell-Flemming framework, postulates that current account and budget deficits move together in the long run and the causality runs from the former to the latter. The results from Engle-Granger and Johansen cointegration procedures support either the twin divergence or the Ricardian equivalence postulations but not the twin deficits hypothesis. Current account deficits and budget deficits are also found to be jointly endogenous. The short-run impacts of budget deficits on current account deficits are found to be mainly through the real exchange rate and real interest rate channels.

Στην παρούσα εργασία εξετάζουμε τους προσδιοριστικούς παράγοντες των δημοσιονομικών ελλειμμάτων καθώς και των ελλειμμάτων στο ισοζύγιο τρεχουσών συναλλαγών. Επίσης, προσπαθούμε να ερευνήσουμε την επίδραση των δημοσιονομικών ελλειμμάτων στα ελλείμματα του ισοζυγίου τρεχουσών συναλλαγών (Υπόθεση δίδυμων ελλειμμάτων). Για το σκοπό αυτό χρησιμοποιούμε ένα δείγμα 27 χωρών του ΟΟΣΑ καλύπτοντας την περίοδο 1994-2006. / In this paper, we examine the determinants of budget deficits and current account deficits. Furthermore, we try to investigate the effect of budget deficits to current account deficits (Twin deficits hypothesis). For this purpose, we use a sample of 27 OECD countries covering the period 1994-2006.