Spelling suggestions: "subject:"epithelial cells."" "subject:"pithelial cells.""
361 |
Salmonella enterica serovar enteritidis requires the type three secretion system-1/2 to invade/survive in chicken oviduct epithelial cells and to modulate innate immune responsesLi, Shuhui 03 May 2008 (has links)
Contaminated poultry and egg products are major sources of Salmonella enterica serovar Enteritidis (S. enteritidis, SE) infections in humans. Colonization of SE in chicken reproductive tract results in the production of contaminated commercial shell-eggs and fertilized hatchery eggs. The complex pathogen-host interactions during SE colonization of chicken reproductive tract are largely unknown. This study was aimed at determining the pathogenic roles of the type three secretion systems (TTSS-1 and TTSS-2) in SE infection of chicken oviduct epithelial cells (COEC). A series of SE strains carrying mutations in the genes encoding structure or effector proteins of TTSS-1 and TTSS-2 were constructed. The invasiveness and intracellular survival rate of each SE strain as well as the host innate immune responses induced by the infections were evaluated. The results demonstrate that both TTSS-1 and TTSS-2 are required by SE to invade COEC which involve genes encoding effector proteins SipA, SopB, SopE2, and PipB. In addition to their involvement in host cell invasion, sipA and sipB are also necessary for the survival or replication of SE inside COEC. Inactivation of TTSS-2 genes (ssaV and pipB) resulted in an enhanced bacterial proliferation inside COEC. The data from this study also show that SE infection triggers pro-inflammatory responses in COEC and TTSS-1 is involved in the expression of iNOS and IL-8, a CXC chemokine. TTSS-1 and TTSS-2 are not necessary for induction of K203, MIP-1β, and IL-10 or suppression of TGF-β3 in COEC.
|
362 |
Disulfide bond formation between dimeric immunoglobulin A and the polymeric immunoglobulin receptor in cultured epithelial cells and rat liverChintalacharuvu, Koteswara Rao January 1991 (has links)
No description available.
|
363 |
Investigations Into the Mechanisms of Vitamin C Uptake in Rodent and Human Lens Epithelial CellsObrenovich, Mark E. 17 June 2008 (has links)
No description available.
|
364 |
Cellular Changes and Effects of Cytokines in the Transformation of Human Mammary Epithelial CellsKan, Charlene E. 07 October 2009 (has links)
No description available.
|
365 |
Transcriptional Regulation of Antioxidant and DNA Repair Transcript Abundance in Human Bronchial Epithelial CellsMullins, D'Anna N. January 2006 (has links)
No description available.
|
366 |
Characterization of Inadequate Host Responses to Intracellular Gram-negative Bacterial PathogensGillette, Devyn Dior January 2013 (has links)
No description available.
|
367 |
"Induction of Autophagy-Mediated Lens Epithelial Cell Death Using Cyclosporine A to Prevent Posterior Capsule Opacification"Hydeman, Laura Rosemary January 2014 (has links)
No description available.
|
368 |
Characterization of C/EBPs in Mammary Epithelial Cell BiologyDearth, Lawrence 20 December 2002 (has links)
No description available.
|
369 |
Effects of exogenous ATP and phenothiazines on ion transport in isolated rat intestinal epithelial cells /Richards, Neil William January 1984 (has links)
No description available.
|
370 |
The effect of Lactobacilli and female sex hormones on the innate immune responses of vaginal epithelial cells.Lam, Jeffrey H.Y. January 2019 (has links)
The female genital tract represents the first line of defence against HIV. Biological factors such as female sex hormones, and the vaginal microbiota are known to affect HIV susceptibility at this site. The female sex hormone estradiol is known to play a protective role, whereas the progestin based contraceptive medroxyprogesterone acetate increases HIV susceptibility HIV. In addition, a Lactobacilli dominant vaginal microbiota is generally protective against HIV. Therefore, in this study, we aimed to elucidate the effects of female sex hormones, and Lactobacilli on the innate immune response of vaginal epithelial cells. / Thesis / Master of Science (MSc)
|
Page generated in 0.1114 seconds