Thesis (Ph. D.)--University of Hong Kong, 1998. / Includes bibliographical references (leaves 282-293).

Thesis (M.S.W.)--University of Hong Kong, 1999. / Includes bibliographical references (leaves 122-129).

Thesis (Ph.D) -- University of Western Sydney, 2007. / A thesis submitted to the University of Western Sydney, College of Health and Science, School of Nursing, in fulfilment of the requirements for the degree of Doctor of Philosophy. Includes bibliographical references.

Thesis (M.S.)--Springfield College, 2004. / Includes bibliographical references. Also available online (PDF file) by a subscription to the set or by purchasing the individual file.

Thesis (Ph.D.)--Victoria University (Melbourne, Vic.), 2009.

Thesis (M.S.)--Springfield College, 2004. / Includes bibliographical references.

Thesis (MSc)--University of Stellenbosch, 2006. / Bibliography.

The statistical correlations between habitual diet and plasma risk factors for coronary heart disease CHD were analyzed using multiple regression. Thirty-one male subjects between 30-56 years kept complete dietary records for 7 days. Daily means of nutrient consumption were calculated using a computerized data base. Fourteen independent variables (total kilocalories, protein %, carbohydrate %, fat %, ethanol %, caffeine, P/S ratio, cholesterol, age, weight, height , weight**.75, Body Mass Index, kilocalories/weight**.75) were created. Plasma samples were analyzed and the following simple (total plasma cholesterol (TC), VLDL-C, LDL-C, HDL2-C, HDL3-C, apo A-I, apo A-II, apo B) and derived (VLDL-C+LDL-C, LDL-C/TC, LDL-C/HDL-C, HDL-C, HDL2-C/HDL3-C, HDL-C/TC, apo B/apo A-I, apo B/apo A-II, apo A-Il/apo A-I) dependent variables were created. Dependent variables were individually regressed against the entire set of independent variables. An F-value of 4.00 to enter an independent variable in the model and of 3.99 to remove one were used to achieve significance of p<05. Age appeared in 5 regression models (TC, apo B, apo B/apo A-I, apo B/apo A-II, apo A-Il/apo A-I) and was positively correlated with increased risk for CHD. Total kilocalories appeared in 4 models (LDL-C, apo B, VLDL-C+LDL-C, LDL/TC) and was negatively correlated with risk. Fat % appeared in 4 models (VLDL-C+LDL-C, LDL/TC, LDL-C/HDL-C, HDL-C/TC) and was associated with increased risk. Body Mass Index was entered in 2 models (HDL2-C, HDL-C) and was positively correlated with risk. P/S ratio was negatively correlated with risk in the three models (HDL2, apo A-I, HDL-C) in which it appeared. The independent variable carbohydrate % was negatively associated with risk in 2 models, LDL-C and apo B. Kcal/wt**.75 was also negatively correlated with risk in the VLDL-C, LDL-C/TC and HDL-C/TC models. One independent variable, EtOH %, was positively associated with risk in the apo A-II and apo A-II/apo A-I models. Two dependent variables did not have any independent predictors (HDL3-C, HDL2-C/HDL3-C) entered in their regression models. Six independent variables did not appear in any regression model (protein %, caffeine, cholesterol, weight, height, weight**.75). Independent variables positively correlated with increased risk for CHD were therefore fat %, ethanol %, age, and Body Mass Index. Independent variables correlated with decreased risk for CHD were total kilocalories, carbohydrate %, P/S ratio, and kilocalories/weight**.75. / Graduation date: 1985

No description available.

No description available.