- About
-
The Global ETD Search service is a free service for researchers
to find electronic theses and dissertations. This service is
provided by the
Networked Digital Library of Theses and Dissertations.
Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
Search results
Showing 241 to 250 of 7198 (0.046 seconds)Spelling suggestions: "subject:"7molecular biology"" "subject:"bimolecular biology""
| 241 |
Ligand chemistry of telluriumPathirana, Hema M. K. K. January 1985 (has links)
No description available.
|
| 242 |
Novel polymeric controlled release systemsHolland, Simon J. January 1986 (has links)
No description available.
|
| 243 |
The gastrointestinal absorption of leadPartridge, Stephen January 1986 (has links)
No description available.
|
| 244 |
Insulin release from cultured B-cellsLambert, David G. January 1987 (has links)
Established RlNm5F and lN111 R1 and newly available HlT-T15 and UMR 407/3 B-cell lines have been successfully maintained in vitro. With the exclusion of UMR 407/3 cells, all lines were continuously propagable. Doubling times and plating efficiencies for HlT-T15, RlNm5F, lN111 R1 and UMR 407/3 cells were 20 hours and 85%, 31 hours and 76%, 24 hours and 80% and 38 hours and 94% respectively. All the cell lines were anchorage dependent, but only UMR 407/3 cells grew to confluence. Only HlT-T15 and UMR 407/3 cells produced a true insulin response to glucose but glucose markedly increased the rate of D-[U14C]glucose oxidation by all the cell lines. Glucose induced insulin release from HlT-T15 cells was biphasic with an exaggerated first phase. Insulin release from HlT-T15, RlNm5F and IN111 R1 cells was stimulated by amino acids and sulphonylureas. Glucagon stimulated insulin release from HlT-T15 and RlNm5F cells while somatostatin and pancreatic polypeptide inhibited release. These observations suggest that net insulin release from the whole islet may be the result of significant paracrine interaction. HlT-T15 and RlNm5F cell insulin release was stimulated by forskolin and inhibited by imidazole. Ca2+ channel blockade and calmodulin inhibition suppressed insulin release from HlT-T15, RlNm5F and IN111 R1 cells. In addition phorbol esters stimulated insulin release from RlNm5F cells. These data implicate cAMP, Ca2+ and protein kinase-C in the regulation of insulin release from cultured B-cells. Acetylcholine increased insulin release from HlT-T15 and RlNm5F cells. Inhibition of the response by atropine confirmed the involvement of muscarinic receptors. HlT-T15 cell insulin release was also inhibited by adrenaline. These observations suggest a possible role for the autonomic nervous system in the modulation of insulin release. Preliminary studies with a human insulinoma maintained in monolayer culture have demonstrated a limited life span of some seven weeks, a continuous low level of insulin release but no insulin response to glucose challenge.
|
| 245 |
The ovarian cycle of rainbow trout with special reference to atresiaCumaranatunga, Palawinnege R. T. January 1985 (has links)
No description available.
|
| 246 |
Glucose stimulus insulin secretion coupling in the pancreatic islets of lean and obese miceHussain, Masharaf January 1985 (has links)
No description available.
|
| 247 |
Potential inhibitors of dihydrofolate reductase: synthesis and NMR spectroscopyYap, Chee H. January 1985 (has links)
No description available.
|
| 248 |
Structural analysis of humic materialsAl-Ahmadi, Abdullah January 1986 (has links)
No description available.
|
| 249 |
Dihydropteridine reductaseCutler, Paul January 1986 (has links)
No description available.
|
| 250 |
The effect of non-steroid growth promotants on the growth of common carpAhmad, Tagried S. January 1986 (has links)
No description available.
|
Page generated in 1.7014 seconds