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  • About
  • The Global ETD Search service is a free service for researchers to find electronic theses and dissertations. This service is provided by the Networked Digital Library of Theses and Dissertations.
    Our metadata is collected from universities around the world. If you manage a university/consortium/country archive and want to be added, details can be found on the NDLTD website.
1

Avaliação da microalbuminuria e da alfa-1 microglobulina em pacientes soro-positivos para hepatite C

Beltrame, Luiz Paulo 15 December 2000 (has links)
Orientador: Maria Almerinda Vieira Fernandes Ribeiro Alves / Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas / Made available in DSpace on 2018-07-27T10:23:52Z (GMT). No. of bitstreams: 1 Beltrame_LuizPaulo_M.pdf: 5682775 bytes, checksum: 7814fb084f0c86a65c60b28361d06fc4 (MD5) Previous issue date: 2000 / Resumo: Avaliamos a microalbuminúria e a alfa-l microglobulina urinária em 104 doadores de sangue, descobertos serem portadores de hepatite C, na triagem sorológica a que foram submetidos no ato da doação. A microalbuminúria foi usada como marcador precoce de lesão glomerular renal, e a alfa-l microglobulina foi usada como marcado r de lesão tubular renal. Todos os indivíduos eram assintomáticos, apresentavam provas de funções hepática e renal normais, sedimento urinário normal, e ausência de macroproteinúria. Enquanto praticamente todos os indivíduos ( 98,1% ) apresentaram resultados nonnatS para a alfa-l microglobulina, encontramos microalbuminúria em 12 dos 104 doadores ( 11,5% ). Observamos associação (p < 0,01) entre a presença de microalbuminúria e o achado de crioglobulinas e fatores reumatóides no soro, detectados respectivamente em 26,4% e 17,5% dos pacientes do grupo estudado. A presença de fator reumatóide e crioglobulinas estiveram, por sua vez, associadas entre si, e com os níveis elevados de gama-glutamil-transferase e de gamaglobulinas séricas (p < 0,01); com níveis elevados de anticorpos contra o vírus da hepatite C, (relação DO/C maior ou igual a 3), e com a etnia (raça negra) (p < 0,05). Não observamos nos 104 doadores soro-positivos para hepatite C associação entre a presença de crioglobulinas e fatores reumatóides circulantes com elevação da alanina-amino-transferase, ou decréscimo das frações C3 e C4 do complemento sérico. Concluímos que em indivíduos portadores de hepatite C, mesmo com funções hepática e renal normais, a presença de microalbuminúria deveria ser avaliada quando houver crioglobulinas e/ou fatores reumatóides circulantes / Abstract: One hundred and four individuals found to be serurn-positive for hepatitis C during blood donation, were evaluated for microalbuminuria (for evaluation of glomerular proteinuria) and urinary alpha-1micro- globulin excretion (for evaluation of tubular proteinuria). All were asymptomatic and presented normal renal and hepatic function, normal urine sedimentation and absence of macroproteinuria. While practica1ly a1l (98.1 %) presented normallevels of alpha-1 microglobulin, we found microalbuminuria in 12 out of 104 patients (11.5%). We observed correlation between microalbuminuria and the presence of cryoglobulinemia and circulating rheumatoid factor (p<0.01). The presence of cryoglobulinemia and rheumatoid factor in these individuals were related.to elevated levels ofhepatitis C vírus antibodies (p<0.05), ethnic background (non-white) (p<0.05), elevated levels of gamma-globulin (p<0.01) and of gamma-glutamyl transferase (p<0.01). We found no relation between the presence of microalbuminuria and serurn levels of alanineaminotransferase, either with decreased in C3 and C4 fractions of the serurn complemento Therefore we concluded thàt even in asymptomatic iIÍdividuals with normal renal and hepatic functions, the presence of microalbuminuria should be evaluated when rheumatoid factor and/or cryoglobulinemia are present in serum, with the purpose of identify initial renal disease / Mestrado / Clinica Medica / Mestre em Clinica Medica
2

Estudo dos efeitos da puromicina em ratos uninefrectomizados

Prates, Liliane Cury 31 August 2001 (has links)
Orientador: Vera Maria Santoro Belangero / Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas / Made available in DSpace on 2018-08-02T13:40:11Z (GMT). No. of bitstreams: 1 Prates_LilianeCury_M.pdf: 18017213 bytes, checksum: 39eb4c1143cd7ba63a0d4d601b7de1c1 (MD5) Previous issue date: 2001 / Resumo: Síndrome nefrótica (SN) é caracterizada por um conjunto de manifestações clínico-laboratoriais incluindo edema, proteinúria intensa, hipoproteinemia, hiperlipidemia e lipidúria. As anormalidades metabólicas que acompanham a SN são responsáveis por muito da morbidade e mortalidade desta condição. Considerando-se as dificuldades em estudos clínicos, vários modelos de SN experimental têm sido desenvolvidos, a maioria utilizando a Puromicina e a Adriamicina. A finalidade do presente estudo foi obter um modelo de SN de instalação aguda, utilizando-se infusão única endovenosa de Puromicina em ratos com unine&ectomia. Foram utilizados 64 ratos machos, Wistar, com 6 a 8 semanas de idade, peso médio de 230,8 gramas, divididos em 4 grupos: ... Observação: O resumo, na íntegra, poderá ser visualizado no texto completo da tese digital / Abstract: Nephrotic syndrome is characterized by edema, increased proteinuria, hypoproteinemia, hyperlipidemia and lipidic factors. Metabolic changes following Nephrotic Syndrome (NS) cause a great ratio of deaths in this condition. Several models of experimental NS have been developed to clear many aspects that are difficult to understand inclinical studies. Among the ways to develop NS in laboratory animaIs, Puromycin and Adriamycin are the most used drugs. This present study shows a NS model using an unique perfusion of Puromycin intravenously in rats with unilateral nephrectomy. 64 male Wistar rats, 6 to 8 weeks old, with median weight of 220 grams were studied. They were divided into 4 groups: ... Note: The complete abstract is available with the full electronic digital thesis or dissertations / Mestrado / Pediatria / Mestre em Saude da Criança e do Adolescente
3

Renal abnormalities in patients with sickle cell disease / AlteraÃÃes renais em pacientes com doenÃa falciforme

Geraldo Bezerra da Silva JÃnior 26 July 2013 (has links)
nÃo hà / ABSTRACT Background - Kidney abnormalities are one of the main chronic complications of sickle cell disease (SCD). The aim of this study is to investigate the occurrence of renal abnormalities among patients with SCD. Methods - This is a cohort study with 26 SCD patients followed in a medical center in Fortaleza, CearÃ, Brazil. Urinary acidification and concentration tests were performed using calcium chloride (CaCl2), and after a 12h period of water and food deprivation. Fractional excretion of sodium (FENa), transtubular potassium gradient (TTKG) and solute free water reabsorption (TcH2O) were calculated. The SCD group was compared to a group of 15 healthy volunteers (control group). Aquaporin-2 (AQP2) and renal outer medullary K+ channels (ROMK) were quantified through exosomes search in urine. Results - Patient`s average age and gender were similar to controls. Urinary acidification deficit was found in 5 SCD patients (19.2%), who presented urinary pH > 5.5 after CaCl2 test. Urinary osmolality was significantly lower in SCD patients (355Â60 vs. 818Â202mOsm/kg, p=0.0001, after 12h period water deprivation). Urinary concentration deficit was found in all SCD patients (100%). FENa was higher among SCD patients (0.75Â0.3 vs. 0.55Â0.2%, p=0.02). The TTKG was higher in SCD patients (5.5Â2.5 vs. 3.0Â1.5, p=0.001), and TcH2O was lower (0.22Â0.3 vs. 1.1Â0.3L/day, p=0.0001). The search for AQP2 did not show significant difference between SCD patients and control group (102Â6.0 vs. 100Â7.2%, p=0.874), as well as for ROMK (172Â38 vs. 100Â25%, p=0.207). Conclusions - SCD is associated with important kidney dysfunction. The main abnormalities found were urinary concentrating and incomplete distal acidification defect. There was also an increase in the potassium transport and decrease in water transport, evidencing the occurrence of distal tubular dysfunction. / RESUMO IntroduÃÃo - AlteraÃÃes renais representam uma das complicaÃÃes crÃnicas principais da doenÃa falciforme (DF). O objetivo deste estudo à investigar a ocorrÃncia de alteraÃÃes renais em pacientes com DF. MÃtodos - Foi realizado estudo de coorte com 26 pacientes com DF acompanhados em um ambulatÃrio de Fortaleza, CearÃ, Brasil. Testes de acidificaÃÃo e concentraÃÃo urinÃrias foram realizados usando cloreto de cÃlcio (CaCl2) e apÃs perÃodo de 12h de jejum e privaÃÃo hÃdrica. Foram calculados fraÃÃo de excreÃÃo de sÃdio (FENa), transporte transtubular de potÃssio (TTKG) e transporte de Ãgua livre de solutos (TcH2O). O grupo de pacientes com DF foi comparado com um grupo de 15 voluntÃrios sadios (grupo controle). Os transportadores aquaporina-2 (AQP2) e canal de K+ apical (ROMK) foram quantificados pela pesquisa de exossomas na urina. Resultados - A mÃdia de idade e a distribuiÃÃo de gÃnero foi similar entre os dois grupos. DÃficit de acidificaÃÃo urinÃria foi encontrada em 5 pacientes com DF (19,2%), que apresentaram pH urinÃrio > 5,5 apÃs o teste com CaCl2. A osmolaridade urinÃria foi significativamente menor entre os pacientes com DF (355Â60 vs. 818Â202mOsm/kg, p=0,0001, apÃs perÃodo de 12h de jejum e privaÃÃo hÃdrica). DÃficit de concentraÃÃo urinÃria foi encontrado em todos os casos de DF (100%). A FENa foi maior entre os pacientes com DF (0,75Â0,3 vs. 0,55Â0,2%, p=0,02). O TTKG tambÃm foi maior nos pacientes com DF (5,5Â2,5 vs. 3,0Â1,5, p=0,001), e o TcH2O foi menor (0,22Â0,3 vs. 1,1Â0,3L/dia, p=0,0001). A pesquisa de AQP2 nÃo mostrou diferenÃa significativa em relaÃÃo ao grupo controle (102Â6,0 vs. 100Â7,2%, p=0,874), bem como a do canal ROMK (172Â38 vs. 100Â25%, p=0,207). ConclusÃo - A DF à associada a importantes alteraÃÃes renais. As principais alteraÃÃes encontradas foram dÃficit de concentraÃÃo e acidificaÃÃo urinÃria. Foi ainda observado aumento no transporte
4

Estenose da ArtÃria do Enxerto Renal: PrevalÃncia e Fatores Associados em uma Unidade de Transplante Renal / Stenosis of the Renal Artery Graft: Prevalence and Associated Factors in a Renal Transplant Unit

Jarinne Camilo Landim Nasserala 22 February 2016 (has links)
nÃo hà / IntroduÃÃo: A estenose da artÃria do enxerto renal (EAER), complicaÃÃo vascular mais comum pÃs-transplante (Tx) renal, pode levar à hipertensÃo resistente, piora da funÃÃo renal e atà perda do enxerto. Objetivos: Investigar a prevalÃncia e fatores associados à EAER. MÃtodos: Estudo caso-controle retrospectivo, em populaÃÃo de receptores de Tx realizados de janeiro de 2008 a marÃo de 2014, em um centro de referÃncia em Tx renal no nordeste do Brasil. Foram avaliados como fatores associados à EAER caracterÃsticas demogrÃficas e clÃnicas do receptor e doador, dados relacionados a cirurgia, dados laboratoriais e nÃmero de anti-hipertensivos. A anÃlise estatÃstica foi realizada atravÃs do programa SPSS 17.0, valores descritivos abaixo de 5% (p < 0,05) foram considerados estatisticamente significativos. Resultados: Foram avaliados 494 de 529 receptores, sendo 24 pacientes com EAER, prevalÃncia de 4,8%. MÃdia do tempo do diagnÃstico 89,9 dias pÃs-Tx. Fatores associados a EAER foram nÃmero de anti-hipertensivos &#8805; 2 e enxerto com duas ou mais artÃrias (p < 0,05). Houve reduÃÃo significativa na mÃdia da PAS (147,1  23,7 para 127,8  15,2mmHg, p=0,001) e da PAD (86,6  13,0 para 77,6  9,4mmHg, p=0,001) apÃs a correÃÃo da EAER, bem como na creatinina sÃrica (de 2,8  2,4 para 1,9  1,8mg/dL, p=0,04). ConclusÃo: A prevalÃncia de EAER mantem o padrÃo descrito na literatura e enxertos com duas ou mais artÃrias estÃo associados com EAER, bem como pacientes que utilizam maior nÃmero de anti-hipertensivos. A correÃÃo da EAER associou-se à melhora do controle pressÃrico e da funÃÃo renal. / Introduction: The transplant renal artery stenosis (TRAS) is the most common vascular complication post-kidney transplant (Tx) that can lead to resistant hypertension, impaired renal function and even loss of the graft. Objectives: To investigate the prevalence and factors associated with TRAS. Methods: A retrospective case-control study was carried out in a population of Tx recipients from January 2008 to March 2014 in a renal Tx reference center in northeastern Brazil. Demographic and clinical characteristics of the recipient and donor, data related to the surgery, laboratory data and number of antihypertensive drugs were assessed as factors associated with TRAS. Statistical analysis was performed using SPSS 17.0. Results: A total of 494 of 529 recipients were assessed, of which 24 had TRAS. The prevalence of TRAS was 4.8% (24 patients), of which 23 had a deceased donor. Twelve patients (50%) were males, mean age of 46.7  13.5 years (range 17-78 years). Mean time of diagnosis was 89.9 days post-Tx. The risk factors associated with TRAS were number of antihypertensive drugs &#8805; 2 (OR: 17.0; CI: 4.1 to 70.4, p = 0.001) and grafting with two or more arteries (OR 8.9, CI: 1.4 -56.6, p = 0.021). There was a significant reduction in mean SBP (147.1  23.7 to 127.8  15.2 mmHg, p = 0.001) and DBP (86.6  13.0 to 77.6  9.4 mmHg, p = 0.001) after TRAS repair, as well as in serum creatinine (2.8  2.4 to 1.9  1.8 mg / dL, p = 0.04). Conclusion: The prevalence of TRAS maintained the pattern described in the literature and grafts with two or more arteries are associated with TRAS, as well as patients that use a higher number of antihypertensive drugs. TRAS repair was associated with improved blood pressure control and renal function.
5

Papel do sistema renina-angiotensina no mecanismo da formação do edema na sindrome nefrotica em crianças : avaliação atraves do efeito do captopril sobre a retenção de sodio

Belangero, Vera Maria Santoro, 1952- 19 April 1989 (has links)
Orientador : Edgard Ferro Collares / Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas / Made available in DSpace on 2018-07-14T03:41:09Z (GMT). No. of bitstreams: 1 Belangero_VeraMariaSantoro_M.pdf: 1241447 bytes, checksum: 86e6d9c27d7dd96207fe3c26daaafd46 (MD5) Previous issue date: 1989 / Resumo: o papel do SRAA na formaçio do edema na Síndrome Nefrótica descompensada foi analisada atraves do emprego do captopril. Foram estudados 8 episódios de descompensaçao, em crIanças de 4 a 11 anos de idade, que estavam em franco desenvolvimento do edema, com dieta sem restrição de sódio ou água e sem diureticos ou corticoterapia, por pelo menos uma semana previa ao inicio do estudo. Nestes episódios foram analisados, entre outros parâmetros, a excreçio urinária de sódio em urina de 24 horas, o incremento diário de peso e o volume plasmático...Observação: O resumo, na integra, podera ser visualizado no texto completo da tese digital. / Abstract: The role of the Renin Angiotensin System (RAS) in the development of oedema in descompensated nephrotic syndrome was studied through the use of captopril. Eight episodes of descompensation were studied in children aged 4 te 12, who were develeping oedema, were on an unrestricted diet, and were taking no diuretics or cort:icoesteroids for at least a week before admission...Note: The complete abstract is available with the full electronic digital thesis or dissertations / Mestrado / Mestre em Medicina
6

Nofrolitotomia anatrofica simplificada

Castilho, Lísias Nogueira 10 February 1988 (has links)
Orientador: Nelson Rodrigues Netto Jr / Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas / Made available in DSpace on 2018-07-15T11:01:46Z (GMT). No. of bitstreams: 1 Castilho_LisiasNogueira_M.pdf: 738620 bytes, checksum: 6220d91e82f2efbe770073c15a82c3b9 (MD5) Previous issue date: 1988 / Resumo: Descrevemos uma sistematização simplificada da técnica de nefrolitotomia anatrófica no tratamento de 9 pacientes potadores de 12 unidades renais com calculo coraliforme, com idade média de 40 anos e proporção mulher/homem de 3,5:1. As principais modificações introduzidas em relação à técnica original de Smith e Royce (1968) são: 1- A não utilização de injeção vascular de corantes para definir os limites entre os lobos anterior e posterior do rim. 2- Clampeamentode todo o pediculo renal. 3- Abertura de todos os infundíbulos caliciais, estenóticos ou não. 4- A não realização de calicoplastias e calicorrafias na tentativa de melhorar a drenagem intra-renal. 5- Revisão da hemostasia após o desclampeamento vascular com parenquima ainda aberto. 6- A não utilização de derivação urinária. O tempo médio de seguimento foi de 16 meses e os resultados obtidos quanto a tempo de cirurgia (média de 204min.), tempo de isquemia ( média de 61 min.), transfusão de sangue (2 unidades em 9 pacientes), complicações (22%), litíase residual (11 %), litiase recorrente (zero), mortalidade (zero) e internação pós-operatória (média de 7 dias) são semelhantes aos de outros autores que empregam a técnica original. / Abstract: We describe a simplified sistematization of the anatrophic neprolithotomy technique in the treatment of 9 patients with 12 renal units with staghorn calculus, with an average age of 40 years and woman/man ratio of 3,5:1. The main changes introduced in relation to the original Smith and Boyce technique (1968) are: 1- Non-utilization of vascular injection of dyes in order to determine the limits between anterior and posterior renal segments. 2- Clamping of the whole renal pedicle. 3- Opening of all the calyceal infundibulum, stenotic or not. 4- Non-realization of calycoplasties or calyorrhaphies as an attempt to improve the intra-renal drainage. 5- Review of hemostasis after the release of the vascular clamp, prior to renal closure. 6- Non-utilization of urinary derivation. The average time of follow-up was 16 months and the final results referring to the time of surgery (average 204 min.), ischemic period (average 61 min.), blood transfusion (2 units for 9 patients), complications (22%), residual lithiasis (11%), recurrent lithiasis (zero), mortality (zero) and post-operatory stay (average 7 days) are similar to the results of other authors who practice the original technique. / Mestrado / Mestre em Medicina
7

O valor da radiologia uretero pielorrenal

Carvalho, Joaquim Roberto de January 1919 (has links)
No description available.
8

Identificazione di profili di rischio cardiovascolare nel trapianto di rene: polimorfismi di geni coinvolti nei processi di infiammazione e di apoptosi

Cappuccilli, Maria <1969> 30 May 2007 (has links)
Introduction. Cardiovascular disease (CVD) represents the main cause of morbidity and mortality in kidney recipients. This study was undertaken to assess the impact of functional polymorphisms located in cytokine and apoptosis genes on CVD after kidney transplantation. Cytokine polymorphisms, generally located in gene regulatory regions, are associated with high and low cytokine production and are likely to modulate the magnitude of inflammatory responses following transplantation, depending on the balance between the levels of pro-inflammatory and antiinflammatory cytokines. The role of apoptosis in atherosclerosis has not been completely elucidated, and here we explored the hypothesis that the heterogeneity in cardiovascular risk in kidney recipients may also be linked to functional polymorphisms involved in apoptosis induction. Purpose. In the search for relevant genetic markers of predisposition to CVD after renal transplant, the present investigation was undertaken to identify the clinical impact of polymorphisms of cytokines TNF-α, TGF-β, IL-10, IL-6, IFN-γ and IL-8 and of apoptosis genes Fas and Caspase 9 in a population of kidney transplant recipients. Materials and methods. The study involved 167 patients who received cadaveric kidney transplantation at our centre between 1997 and 2005 (minimum follow-up of 12 months); 35 of them had experienced cardiovascular events (CVD group) and 132 had no cardiovascular complications (non-CVD group). Genotyping was performed using RFLP (Restriction Fragment Length Polymorphism) for RFLP per IL-8/T-251A, Fas/G-670A e Casp9/R221Q polymorphism and SSP (Sequence Specific Primer) for TNF-α/G-308A, TGF-β/L10P, TGF-β/R25P, IL-10/G-1082A, IL- 10/C-819T, IL-10/C-592A, IL-6/G-174C, IFN-γ/T+874A polymorphisms.Results. We found a significant difference in TNF-α and IL-10 genotype frequencies between the patients who had suffered cardiovascular events and those with no CVD history. The high producer genotype for proflogistic cytokine TNF-α appeared to have a significantly superior prevalence in the CVD group compared to the non-CVD group (40.0% vs 21.2%) and it resulted in a 2.4-fold increased cardiovascular risk (OR=2.361; p=0.0289). On the other hand, the high producer genotype for the antiinflammatory cytokine IL-10 was found in 2.8% of the CVD group and in 16.7% of non-CVD group; logistic regression showed a 0.3-fold reduced risk of CVD associated with genetically determined high IL-10 production (OR=0.278; p<0.0001). The other polymorphisms did not prove to have any impact on CVD. Conclusions. TNF-α and IL-10 gene polymorphisms might represent cardiovascular risk markers in renal transplant recipients.
9

Effetti dell'acido urico sulle cellule glomerulari mesangiali: meccanismi intracellulari di trasduzione del segnale e possibili implicazioni nella progressione del danno renale e nella sindrome infiammatoria in corso di nefropatie croniche

Festuccia, Francescaromana <1973> 30 May 2007 (has links)
Uric acid is a major inducer of inflammation in renal interstitium and may play a role in the progression of renal damage in hyperuricemic subjects with primary nephropathies, renal vascular disease, and essential hypertension. At the same time, UA also acts as a water-soluble scavenger of reactive oxygen species. We evaluated the cellular effects of UA on cultured HMC as a potential interstitial target for abnormally elevated levels in acute and chronic renal disease. Intracellular free Ca2+ ([Ca2+]i) was monitored by microfluorometry of fura 2-loaded cells, while oxidation of intracellularly trapped non-fluorescent 2’,7’-dichlorofluorescein diacetate (DCFHDA, 20 uM) was employed to assess the generation of reactive oxygen species during 12-hr incubations with various concentrations of UA or monosodium urate. Fluorescent metabolites of DCFH-DA in the culture media of HMC were detected at 485/530 nm excitation/emission wavelengths, respectively. UA dose-dependently lowered resting [Ca2+]i (from 102±9 nM to 95±3, 57±2, 48±6 nM at 1-100 uM UA, respectively, p <0.05), leaving responses to vasoconstrictors such as angiotensin II unaffected. The effect was not due to Ca2+/H+ exchange upon acidification of the bathing media, as acetate, glutamate, lactate and other organic acids rather increased [Ca2+]i (to max. levels of 497±42 nM with 0.1 mM acetate). The decrease of [Ca2+]i was abolished by raising extracellular Ca2+ and not due to effects on Ca2+ channels or activation of Ca2+-ATPases, since unaffected by thapsigargin. The process rather appeared sensitive to removal of extracellular Na+ in combination with blockers of Na+/Ca2+ exchange, such as 2’,4’-dichlorobenzamil, pointing to a countertransport mechanism. UA dose-dependently prompted the extracellular release of oxidised DCFH (control 37±2 relative fluorescence units (RFU)/ml, 0.1uM 47±2, 1 uM 48±2, 10 uM 51±4, 0.1 mM 53±4; positive control, 10 uM sodium nitroprusside 92±5 RFU/ml, p<0.01). In summary, UA interferes with Ca2+ transport in cultured HMC, triggering oxidative stress which may initiate a sequence of events leading to interstitial injury and possibly amplifying renal vascular damage and/or the progression of chronic disease.
10

Utilità della biopsia epatica nella valutazione di idoneità al trapianto di rene del paziente con infezione da virus C

Maiorca, Paolo <1971> 30 May 2007 (has links)
No description available.

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