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Pseudomonas syringae pv tagetis host range and toxin production ability in vivo and in vitro /Rhodehamel, Nicholas H. January 1986 (has links)
Thesis (M.S.)--University of Wisconsin--Madison, 1986. / Typescript. eContent provider-neutral record in process. Description based on print version record. Bibliography: leaves 42-45.
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Resistance of Phaseolus WBR 133 to Pseudomonas syringaeDaub, Margaret E. January 1979 (has links)
Thesis--University of Wisconsin--Madison. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 139-150).
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Characterization of CorS, a histidine protein kinase involved in temperature-dependent synthesis of the phytotoxin coronatine in Pseudomonas syringaeSmirnova, Angela Vladimirovna. January 2001 (has links) (PDF)
Marburg, University, Diss., 2001.
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Thermoresponsive secretion of the extracellular enzyme levansucrase from Pseudomonas syringaeLi, Hongqiao. January 2001 (has links) (PDF)
Marburg, University, Diss., 2001.
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Molecular genetic characterization of a pathogenicity-attenuated mutant of Pseudomonas syringae pathovar syringaeZhao, Yuqi 03 May 1991 (has links)
A pathogenicity locus of Pseudomonas syringae pv.
syringae identified by Tn5 mutagenesis was investigated.
The mutant strain PS9024 is attenuated for disease
expression in its host, Phaseolus vulgaris, but produces
the hypersensitive reaction (HR) in the nonhost, tobacco
(Nicotina tabacum). A cosmid clone carrying 16 kilobases
(kb) of contiguous genomic DNA partially complements this
mutant. Altered growth of the mutant in planta was also
partially restored. Marker exchange mutagenesis with Tn3-
HoHo1 at two other sites within this locus results in
mutants with attenuated and severely reduced
pathogenicity. The locus is complex and contains
repetitive DNA sequences. Northern analysis reveals that
this locus is expressed in planta, but is not expressed in
a rich growth medium, and the transcript is larger than 10
kb, suggesting that the locus is transcribed as a
polycistronic mRNA. Comparison of total cellular protein
profiles of R32 and PS9024 using SDS-PAGE analysis further
reveals that at least nine protein bands ranging from
approximately 100 kD or above in size are present in the
wild type strain R32, but absent from the mutant.
Additionally, a protein of approximately 45 kD is absent
from the mutant. The site of Tn5 insertion has been
partially sequenced. The initial search of the data banks
suggested a gene or genes related to the ornithine
biosynthetic pathway map to this locus. Further study
strongly suggest a gene that encodes a membranceassociated
protein and under the control of a promoter
identical to appA gene promoter maps at this site and it
is involved in the process of pathogenesis. / Graduation date: 1991
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Untersuchungen zur Rolle des Kohlenhydratmetabolismus während Pflanze-Pathogen-Interaktionen und der KeimlingsentwicklungBonfig, Katharina Barbara January 2008 (has links)
Würzburg, Univ., Diss., 2008. / Zsfassung in engl. Sprache.
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Bacterial blight of lima beanMya Thaung, Maung, January 1956 (has links)
Thesis (Ph. D.)--University of Wisconsin--Madison, 1956. / Typescript. Abstracted in Dissertation abstracts, v. 16 (1956) no. 11, p. 1991. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 34-37).
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Pseudomonas syringae type III secretion system and effectorsFu, Zhengqing. January 2008 (has links)
Thesis (Ph.D.)--University of Nebraska-Lincoln, 2008. / Title from title screen (site viewed Jan. 13, 2009). PDF text: 227 p. : ill. (some col.) ; 2 Mb. UMI publication number: AAT 3323493. Includes bibliographical references. Also available in microfilm and microfiche formats.
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The molecular battle between virulence weapons of Pseudomonas syringae and integrated defense responses of Arabidopsis thalianaKim, Min Gab 13 September 2006 (has links)
No description available.
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Nutrient assimilation and amino acid utilisation in Pseudomonas syringaeJones, Rachel January 2010 (has links)
The aim of this study was to increase understanding of amino acid utilisation and amino acid permease (aa_permease) genes in the plant pathogen Pseudomonas syringae, which inhabits the nutrient-limited environment of the plant surface and intracellular apoplast. aa_permease genes are significantly reduced in P. syringae genomes compared to non plant-pathogenic Pseudomonas. Accordingly, this work demonstrates that P. syringae can utilise a restricted number of amino acids for growth compared to non plant-pathogenic Pseudomonas. The remaining aa_permease genes in Pseudomonas syringae pv. tomato are annotated as transporting GABA, ethanolamine, proline and aromatic amino acids. Sequence analyses, phylogenetic analyses, chemotaxis assays and gene expression analyses supported the functional annotations of the GABA, ethanolamine and aromatic aa_permeases, but showed that expression of the proline permease was specifically induced by histidine, which suggests that this encodes a histidine transporter. Expression of the GABA and histidine permeases was also induced in the presence of tomato apoplast, indicating that these amino acids may be assimilated during apoplast colonisation. P. syringae pv. tomato exhibited chemotaxis towards tomato apoplast and several constituent amino acids, which may be important for invasion of the apoplast from the plant surface. Uptake of several amino acids including GABA and histidine appeared to be negatively affected in alkaline media. The aa_permeases use electrochemical potential energy to transport substrates and this mechanism may be affected by pH. The plant apoplast becomes increasingly alkaline during infection and this may select against the retention of particular transporters in P. syringae. As GABA is an abundant amino acid in the apoplast and can be used by P. syringae strains for growth, the growth of P. syringae pv. tabaci was monitored in transgenic tobacco plants with increased GABA. Although growth was similar in all plants, significantly fewer symptoms and more callose deposition was observed upon infection.
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