Spelling suggestions: "subject:"état brain"" "subject:"stat brain""
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Brain uptake of L-Tryptophan and diazepam in the anaesthetised rat : The role of protein bindingFenerty, C. A. January 1986 (has links)
No description available.
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Uptake and release of monamines from rat nucleus raphe dorsalis synaptosomesLewis, D. J. January 1987 (has links)
No description available.
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Studies on pro-opiomelanocortin peptide-containing neuronal pathways in the rat brainHaws, C. January 1986 (has links)
No description available.
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Functional aspects of the interactions between neurotensin and dopamine in the brainFord, A. P. D. W. January 1988 (has links)
No description available.
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A functional role for histamine in brain : Interactions with thyrotrophin releasing hormone (TRH)Bristow, L. J. January 1988 (has links)
No description available.
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Role of monoamine oxidase in the regulation of extracellular 5-HT and 5-HT1̲ receptor functionSleight, A. J. January 1988 (has links)
No description available.
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An investigation of long-term pro-active non-associative mechanisms by which theta-driving sepatal stimulation alters behaviour in ratsWilliams, J. H. January 1987 (has links)
No description available.
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The role of peptides and other transmitters in the regulation of dopamine synthesis in the rat brainChowdhury, Muhammad Rezwan January 1988 (has links)
No description available.
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Binary Mixtures of Pyrethroids Interact with Voltage-Sensitive Calcium and Chloride Channels in Isolated Presynaptic Nerve Terminals from Rat BrainHodgdon, Hilliary E. 01 January 2008 (has links) (PDF)
Select pyrethroid binary mixtures (deltamethrin plus S-bioallethrin, β-cyfluthrin, cypermethrin, and fenpropathrin) elicit a more-than-additive response on L-glutamate release from rat brain synaptosomes that is independent of calcium influx. Using a variety of chloride channel antagonists, anthracene-9-carboxylic acid (9-AC), rChlorotoxin (ClTx), 4,4’-dintitrostilbene-2,2’-disulfonic acid (DNDS), 5-nitro-2-(3-phenylpropylamino) benzoic acid (NPPB), and picrotoxinin (PTX), we have identified two mechanisms by which pyrethroids may enhance L-glutamate release. The results from this study indicate that only ClTx and NPPB, at their EC50s (0.1 μM and 70 μM, respectively), significantly increase L-glutamate release when in the presence of our most potent pyrethroid, deltamethrin, at its EC50 (2 x 10-12 M). When these two antagonists were used in the presence of deltamethrin plus cypermethrin and deltamethrin plus fenpropathrin, a more-than-additive response was elicited at lower concentrations of the binary mixtures. Likewise, NPPB in the presence of the additive binary mixture, deltamethrin plus tefluthrin, first elicited a more-than-additive response at the 1:10 mixture. Since both ClTx and NPPB are inhibitors of voltage-gated chloride channels (ClC-2) and calcium-activated chloride channels, our findings suggest that these channels are potential target sites for certain pyrethroids and likely are important in pyrethroid neurotoxicity.
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Molecular Investigation Of The Effects Of Antioxidants On Rat Brain TissuesAkkas, Sara Banu 01 January 2003 (has links) (PDF)
MOLECULAR INVESTIGATION OF THE EFFECTS OF ANTIOXIDANTS
ON RAT BRAIN TISSUES
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