Spelling suggestions: "subject:"kyrosine kinase"" "subject:"lyrosine kinase""
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RET receptor tyrosine kinase in developing, adult and polycystic kidneys李震威, Lee, Chun-wai, Davy. January 2000 (has links)
published_or_final_version / Paediatrics / Doctoral / Doctor of Philosophy
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The regulation of cardiac potassium channels by protein tyrosine kinasesZhang, Deyong, 張德勇 January 2008 (has links)
published_or_final_version / Medicine / Doctoral / Doctor of Philosophy
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The binding of 14-3-3 proteins to the Ron receptor is required for its biological activitySantoro, Massimo M. January 2000 (has links)
No description available.
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Expression, function and conservation of the c-Ret proto-oncogeneMarcos-Gutierrez, Camelia Victoria January 1997 (has links)
No description available.
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The Eph growth factor receptorTuzi, Nadia Lucia January 1995 (has links)
No description available.
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Regulatory domains of GABAâ†A receptorsDunne, Emma Louise January 2000 (has links)
No description available.
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Chemical diversity from the oxidation of bioactive phenolsWells, Geoffrey January 1998 (has links)
No description available.
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Anti-tumour activity of novel phenolic compoundsSeaton, Angela January 1998 (has links)
No description available.
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Anti-cancer peptides containing modified tyrosine residuesCooper, Margaret S. January 1995 (has links)
No description available.
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Production and function of a soluble c-Kit moleculeRead, Stuart Hamilton. January 2001 (has links) (PDF)
"Research conducted at the Department of Haematology, Hanson Centre for Cancer Research, Institute of Medical and Veterinary Science."--T.p. Includes bibliographical references (leaves 170-214). Elevated levels of receptor tyrosine kinases have been implicated in carcinogenesis. It is possible that high expression of c-Kit by the leukaemic cell provides them with a growth advantage over their normal counterparts in the bone marrow microenvironment. Thus, a means of inhibiting the interaction of c-Kit on these cells with ligand Steel Factor may remove proliferation and survival signals. Main aim of the study was to produce a biological inhibitor of this interaction and evaluate its ability to prevent ligand Steel Factor from binding to c-Kit on live cells.
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