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A preliminary investigation of periodontal disease and rheumatoid arthritis /Dong, Xiaoxiao. January 2005 (has links)
Thesis (M. D. S.)--University of Hong Kong, 2005.
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The phenomenological lived experience of rheumatoid arthritisIaquinta, Monica L. January 2001 (has links)
Thesis (M.S.)--West Virginia University, 2001. / Title from document title page. Document formatted into pages; contains v, 76 p. Includes abstract. Includes bibliographical references (p. 38-42).
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Explanatory models of rheumatoid arthritisSamford Fair, Betty Susan, 1953- 17 May 2011 (has links)
Not available / text
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The diagnostic and prognostic value of anti-CCP assay in the juvenile idiopathic arthritis (JIA)Hui, Kwun-ho., 許冠浩. January 2004 (has links)
published_or_final_version / Medical Sciences / Master / Master of Medical Sciences
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The role of self-efficacy in rheumatoid arthritis patientsChu Sok-iam, Peony January 1994 (has links)
published_or_final_version / abstract / toc / Clinical Psychology / Master / Master of Social Sciences
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Electrophoretic studies on hemoglobin in rheumatoid arthritisHenderson, J. Frank (Joseph Frank), 1933- January 1956 (has links)
No description available.
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In vitro effects of nitrogen mustard on rheumatoid and non-rheumatoid cellsRatajczak, Helen Vosskuhler, 1938- January 1970 (has links)
No description available.
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Persons with rheumatoid arthritis and social isolationJohnson, Shirley Ann, 1935- January 1976 (has links)
No description available.
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Selective antagonism of potential proinflammatory PG receptor subtypes by AGN-211377Wang, J. Weizhen January 2010 (has links)
PG receptor and COX gene expression patterns and LPS induced PG production have been investigated in human macrophages and osteoclasts as cell models to study rheumatoid arthritis. The results suggest that PG receptors DP<sub>2</sub>, EP<sub>1</sub> and EP<sub>3</sub> may not be directly involved in macrophage-mediated inflammatory responses and osteoclast-mediated bone erosion due to the absence of receptor expression. Because of the undetectable levels of natural ligand, IP receptor effects are dependent on the availability of PGI<sub>2</sub> secreted from other types of cells in the close vicinity. Meanwhile, DP<sub>1</sub>, EP<sub>2</sub>, EP<sub>4</sub>, FP and TP receptors, and their natural agonists, may play important and diverse roles in these cells. TXA<sub>2</sub> is the most abundant prostanoid found in these cells with or without LPS stimulation. The TP receptor pathway in inflammation and bone resorption may have been previously overlooked. AGN-211377 was discovered with a unique pharmacological profile, which provided antagonism at specific proinflammatory PG receptor subtypes with the rank order of TP > DP<sub>1</sub> ≥FP >EP<sub>1</sub> > DP<sub>2</sub> and no apparent antagonism on EP<sub>2</sub>, EP<sub>3</sub>, and IP receptors. AGN-211377 significantly and dose-dependently suppresses LPS- or TNFα-induced release of 12 proinflammatory cytokines/ chemokines in macrophages, and osteolysis activity in osteoclasts. Collectively, these results suggest the existence of prostanoid receptors DP<sub>1</sub> FP, and TP in human macrophages and osteoclasts, which are likely to play a proinflammatory role. AGN-211377 is at least as effective as diclofenac (a NSAID) and rofecoxib (a COX1B) in suppression of proinflammatory cytokine/chemokine production in macrophages, and in suppression of osteoclast function. Moreover, AGN-211377 may represent a safer replacement for COX1Bs. The therapeutic advantages of AGN-211377 can be extended beyond RA treatment.
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Cortisol metabolism in vitro : biological and structural determinantsAbel, Stuart Michael January 1993 (has links)
No description available.
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