The central proposition of this thesis is that intergroup attributions and explanations, like any other intergroup behaviour, are affected by the perceived relations between the groups. Social Identity Theory (SIT) was adopted as the theoretical framework within which to investigate intergroup relations. According to this theory, intergroup behaviour is affected by the relative status the groups bear to each other, together with the perceived legitimacy and stability of this status hierarchy. The thesis proposes two amendments to the theory. It is argued that perceived legitimacy and perceived stability have different effects on intergroup behaviour and hence can not be subsumed under the general category of "insecure comparisons". It is also suggested that intragroup variability exists in these perceptions. A study was conducted which confirmed these propositions, and predictions emerging from SIT concerning the effect of these perceptions on intergroup behaviour and attitudes received considerable support. In examining the effect of the intergroup perception factors on bias in intergroup attributions, the thesis focuses on two types of groups: race and gender. Three types of attribution were considered in different studies: explanations of intergroup inequality, attributions of blame for rape or robbery and attributions for individual group members' actions and outcomes. The results indicate that the relative status of the groups, together with the perceptions of the legitimacy of this status hierarchy influence the first two - the explanations for inequality and the attributions of blame made to the victim and perpetrator of crimes. In general, it appears that the 'prise de conscience' of the illegitimacy of the intergroup situation is the motivating force in rejecting the domination of the high status group over the low status group, by members of both the high and low status groups. However, the third type of attributions, the intergroup explanations made for individual group members' actions and outcomes were not in general affected by these factors. It was suggested that either the intergroup aspect of these vignettes lacked the salience to elicit an intergroup response, or that the actors in the vignettes were treated as atypical of the groups.

Campylobacter is currently the most common cause of acute bacterial diarrhoea in Australia among all the notified enteric pathogens with more than 15,000 cases each year. The incidence of notified campylobacteriosis has steadily increased during the past 15 years from 67.0/100,000 population in 1991 to 121.4/100,000 in 2005, though the factors contributing to this increase had not been studied. Adjusting for under-reporting there are, at this point in time, an estimated 225,000 infections occurring each year in Australia, most of which are sporadic in nature. Much of our knowledge in Australia about risk factors for sporadic disease has been based on overseas literature. Prior to the studies undertaken in this thesis, the epidemiology of Campylobacter infection had not been thoroughly studied in Australia, nor had there been any national studies examining risk factors for locally-acquired infection. The broad aim of this thesis was to examine in depth the descriptive epidemiology of Campylobacter infection in Australia, explore the reasons for the sustained increase in incidence of infection and to identify the major risk factors for locally acquired infection using a multi-centre case-control study design. The descriptive study of the epidemiology of campylobacteriosis in Australia was based on Australian notifiable disease surveillance data collected over a 15-year period between 1991 and 2005. This study described the key epidemiological characteristics of this disease in Australia and identified some significant differences in incidence trends across states and territories and among different age groups which had not been previously reported. The study identified gaps in our knowledge of this disease in Australia and made recommendations for future research including the investigation of factors associated with the decline in incidence of infection among children aged  4 years and further studies to identify age and sex-specific risk factors for infection. The issue of seasonality, transmission routes and infection was addressed and areas for further research were specified including longitudinal studies at a regional level that incorporate a comparison of human, animal and environmental genotypes. This study also provided strong compelling evidence to support the hypothesis that the increase in notification rates in Australia during this period represented a real increase in the incidence of infection and that the main driving force behind this rise has been the ongoing increase in chicken consumption among the Australian public. The multi-centre case-control study, involving 1,714 participants  5 years of age, identified the major foodborne and non-foodborne risk factors for Campylobacter infection among the general population in Australia. This study confirmed that chicken meat is a major source of sporadic infection in this country and is responsible for almost one-third of all cases that occur in the Australian community each year. Other independent risk factors for sporadic infection in Australia included consumption of offal and ownership of domestic dogs or chickens aged  6 months. The Nagelkerke R² value of 16% for the final multivariable model indicated a considerable proportion of our case-patients had unexplained risk factors. The combined population attributable risk (PAR) estimate for the independent foodborne risk factors in this study was 31%, which is considerably less than the 75% to 80% of cases in the general population which are thought to be caused through foodborne transmission. Possible explanations for these results include the likelihood that a proportion of foodborne transmission in Australia occurs through food vehicles other than chicken due to cross-contamination from raw products, and the likelihood that much of the population attributable risk that is unaccountered for, may in fact be due to inherent limitations of study design resulting in systematic errors (information bias) and possibly reduced estimates of effect. The burden of illness among the general population in Australia attributable to different independent risk factors was estimated using a novel method developed specifically for this study. Briefly, community incidence data was coupled with PAR data from the case-control study and simulation techniques were used to: (i) estimate the number of infections attributable to specific risk factors, and (ii) derive credible intervals for these estimates by modeling the uncertainty in each variable component. This model of using case-control data in conjunction with pre-existing surveillance data provides researchers with a simple but robust tool for conducting source attribution studies on enteric pathogens. In conclusion, the studies undertaken in this thesis have made important contributions to our understanding of the epidemiology of sporadic Campylobacter infection in Australia.

Borderline personality disorder (BPD) is a complex psychiatric condition whose severity is compounded by the heterogeneous psychological functioning of those who suffer from the disorder. This heterogeneity has made the identification of a unified treatment strategy difficult and attempts to resolve this variation within the disorder by investigating subtypes of BPD have been made. However, the clinical utility of this approach has not been examined. The major object of this research project was to investigate the presence of subtypes of BPD and to examine whether treatment effectiveness varied as a function of subtype. Data from 61, predominantly female, participants with BPD were entered into a cluster analysis. Using variables that are central to cognitive behavioural models of BPD and have been shown to be heterogeneously distributed in previous BPD samples, two subgroups were identified and defined on the basis of whether participants attributed the causes o f negative events as being themselves or other people. Consistent with hypotheses, the subgroup with a tendency to blame others for negative events showed far lower levels of change between admission and discharge than the subgroup who blamed themselves for negative events, on both measures of statistical and clinical significance. Alternate means of identifying participants who optimally responded to the intervention were explored and a cluster analysis identified two groups of participants that were separated on the basis of whether they had shown clinically significant change on a range of variables. It was found that data from admission to the program could successfully predict which participants would belong in the optimal or mixed response groups upon discharge. Taken together, the findings of this research project suggest that not only can theoretically valid subgroups of BPD be identified, but that they have clinical utility in understanding participants' response to intervention. Further, the findi ngs suggest that profiles of clinical change can be identified and predicted. The findings of this research project are discussed with respect to their methodological limitations, suggestions for future research, and their implications for both theory and practice.

Theory of mind, the ability to attribute mental states to oneself and others, has traditionally been investigated in humans and nonhuman primates. However, non-primate species, such as domestic dogs, may also be potential candidates for such a faculty. Domestic dogs (Canis familiaris) evolved from a social-living, wolf-like ancestor, and were the first species to be domesticated, with likely selection for sensitivity to human cues and human-like cognitive abilities. Dogs typically spend their lives in the rich social environment of human families, and thus dogs are naturally enculturated. The combination of these factors make dogs an excellent candidate for having a functional theory of mind. Yet perhaps surprisingly, prior research on theory of mind in dogs is limited, with inconclusive and contradictory results. The research described in this thesis is a systematic investigation of dogs' potential to demonstrate a functional theory of mind in their interactions with humans. Four experiments are presented, based on the Knower-Guesser paradigm (Povinelli et al., 1990), in which a knowledgeable and an ignorant human informant indicated the location of hidden food to the dog. In Experiment 1, one informant was absent (Guesser) and one present (Knower) during the food-hiding, and the dogs chose the Knower. However, when both informants were present, the dogs chose the informant that did the baiting, but this preference was less than when the Guesser was absent. In Experiments 2 and 3, a third experimenter hid the food while the informants covered their cheeks (Knower) or eyes (Guesser) with their hands, or were attentive (Knower) or inattentive (Guesser) to the food-hiding. In both cases, the dogs showed a significant preference for the Knower. In Experiment 4, the dogs showed no preference between the informants when they had equal perceptual access to the baiting, and were unsuccessful at selecting any container when the informants did not provide communicative cues. Overall, the present research provides the most definitive evidence yet that domestic dogs may be able to attribute differential states of knowledge to human observers, and thus may possess a functional theory of mind.

Thesis (Ph.D.)--Kent State University, 2007. / Title from PDF t.p. (viewed Nov. 14, 2007). Advisor: Michael Y. Hu. Keywords: marketing, service. Includes questionnaire. Includes bibliographical references (p. 134-145).

Thesis (M.A.)--University of Notre Dame, 2009. / Thesis directed by David A. Smith for the Department of Psychology. "March 2009." Includes bibliographical references (leaves 53-57).

Thesis (Ph. D.)--Michigan State University. Dept. of Counseling, Educational Psychology, and Special Education, 2006. / Title from PDF t.p. (viewed on Nov. 16, 2008) Includes bibliographical references (p. 59-64). Also issued in print.

Thesis (Ph. D.)--University of Hong Kong, 2008. / Includes bibliographical references (leaf 183-218) Also available in print.

Thesis (Ph. D.)--University of Nevada, Reno, 2008. / "May, 2008." Includes bibliographical references (leaves 181-186). Online version available on the World Wide Web.

Thesis (M.S.)--University of North Texas, May, 2009. / Title from title page display. Includes bibliographical references.