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Studies on the structure, subcellular localization and biochemical effects of cytokinins in transfer ribonucleic acidsSwaminathan, Santhanam, January 1900 (has links)
Thesis (Ph. D.)--University of Wisconsin--Madison, 1975. / Typescript. Vita. Description based on print version record. Includes bibliographical references (leaves 207-218).
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Function and regulation of citron kinase during cytokinesis in animal cellsBassi, Irma Zuni January 2014 (has links)
No description available.
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Genetic analysis of the role of pebble during cytokinesis in Drosophila /O'Keefe, Louise. January 2001 (has links) (PDF)
Thesis (Ph.D.)--University of Adelaide, Dept. of Molecular Biosciences, 2002? / Errata pasted onto back page. Bibliography: p. 133-149.
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The regulation of division of higher plant cellsO'Sullivan, A. M. January 1988 (has links)
No description available.
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A study on the role of polarity, Rho family GTPases, and cell fate in cytokinesisZhuravlev, Yelena January 2017 (has links)
Cytokinesis is the physical partition of one cell into two. In Chapter 1, I provide a brief introduction to cytokinesis and some of the proteins whose functions I parse out throughout my studies. In Chapter 2, I present work I’ve contributed to elucidate the role of polarity proteins in cytokinesis, as well as a look at the differential requirement for canonically essential cytokinetic proteins in the 4-cell embryo. In Chapter 3, I address a long-standing controversy in the field regarding the relationship between the Rac GAP protein Cyk-4 and the small GTPase Rac, and in particular the inhibitory role of Rac during cell division. My major body of work highlights the necessity not to close the books on the GAP activity of Cyk-4 and its inhibition of Rac. I show that Rac is unable to rescue cytokinesis failure in downstream Rho effectors whose loss weakens the contractile ring, suggesting it is not a promiscuous suppressor of cytokinesis. Additionally, I found that levels of non-muscle myosin-II and the actin binding domain of Utrophin were unchanged with loss of Cyk-4. From this, I infer that Cyk-4 is unlikely to be an activator of the RhoGEF Ect-2. These results emphasize the need to probe further into the cross-talk between these GTPases. In chapter 4, I show inconclusive data addressing the role of cell fate signaling in protection against cytokinesis failure. Overall, this thesis represents my contributions to the field, revealing the complexity involved in assuring successful completion of cytokinesis.
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Screening new cytokinesis genes and investigation of regulation of Hof1 in cytokinesisPark, Jung Eun, January 2007 (has links) (PDF)
Thesis (M.S.)--University of Missouri--Rolla, 2007. / Vita. The entire thesis text is included in file. Title from title screen of thesis/dissertation PDF file (viewed December 7, 2007) Includes bibliographical references (p. 28-35).
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A FINE STRUCTURE STUDY OF MICROTUBULES AND KINETOCHORES AND THEIR IMPLICATIONS FOR MOVEMENTS OF CELL COMPONENTS IN MITOTIC AND MEIOTIC CELLSWilson, Harold Jenkins January 1969 (has links)
No description available.
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Formation of the midzone microtubule bundles during cytokinesisLee, Kian-Yong January 2011 (has links)
No description available.
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Chlamydia Trachomatis Disrupts Cytokinesis to Accelerate Nutrient Acquisition from Host CellsSun, He Song 01 September 2014 (has links)
Chlamydia trachomatis is the leading cause of bacterial sexually transmitted infections
worldwide and it has been linked to increased risks of cervical cancer. Our study aims to understand the mechanisms by which chlamydia infection contributes to higher risks of tumorigenesis. We discovered in over 90% of infected human epithelial cells that chlamydia inclusions localized to the host cell centre during mitosis and prevented the completion of cleavage furrow ingression. Infected cells that failed to divide into two daughter cells re-entered
interphase as tetraploid multinuclear cells, which are known initiators of tumorigenesis. In order to address whether chlamydia actively sought the host cell centre during mitosis, we created vacuoles using internalized latex beads and our findings indicated that chlamydia inclusions
localized to the host cell centre much more frequently than similarly sized vacuoles. In addition, we demonstrated that metabolically inactive chlamydia inclusions localized to the cell centre less frequently than inclusions under normal conditions. Together, these results suggested that
chlamydia actively localized to the host cell centre to efficiently block host cell mitosis. We took advantage of the microtubule-displacing capability of the C. trachomatis inclusions and demonstrated that astral microtubules could promote furrow initiation independent of the central spindle microtubules. Microdomains on chlamydia inclusions play important roles in inclusion localization and chlamydia replication. We discovered that these structures were surprisingly resistant to
bacterial protein synthesis and host Src family kinase inhibitors. Finally, we demonstrated that multinuclear cells that resulted from unsuccessful mitosis contained significantly higher Golgi content, an important nutrient source for chlamydia. Our results indicated that C. trachomatis in
multinuclear cells intercepted Golgi-derived lipids faster than in mononuclear cells. Together, my results reveal that C. trachomatis inclusions robustly localize to the cell centre to block host cell mitosis in order to acquire host Golgi nutrients more quickly in multinucleated cells.
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Role of microtubules in budding yeast cytokinesisPark, Su Young, January 2008 (has links) (PDF)
Thesis (M.S.)--Missouri University of Science and Technology, 2008. / Vita. The entire thesis text is included in file. Title from title screen of thesis/dissertation PDF file (viewed January 22, 2009) Includes bibliographical references (p. 34-40).
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