The problem of the study was to determine the better predictors of sprint performance for male and female sprinters from selected leg power and isokinetic strength tests. Ten male and five female sprinters volunteered to be measured for vertical jump performance, anaerobic power and capacity, peak isokinetic torque at the hip, knee, and ankle joint, and sprint performance. A forward stepwise multiple regression analysis was performed to allow selection from all strength and power variables regressed on the dependent variables of 30 meters, 60 meters, and flying 30 meter sprints. This procedure allowed one to examine the contribution of each predictor variable to the regression model. Only the independent variables that elicited a regression equation significant at the .05 level were used in final regression models. The regression models developed for the males were: 30 meters (crouch start) = 6.115 - .083(anaerobic power) - .055(vertical jump) - .044(plantarflex 120"/s) - .022(knee flex 60'/s); 60 meters (crouch start) = 11.111 - .145(vertical jump) - .086 (anaerobic power) - .172(hip flex 300'/s) - .098(knee flex 60'/s); and 30 meters (flying start) = 4.295- .055(anaerobic power) - .312(knee flex 180'/s) - .090(hip flex 300'/s). The regression models for the women were different than the males and were: 30 meters (crouch start) = 9.530 - .346(vertical jump); 60 meters (crouch start) = 18.083- .686(vertical jump); and 30 meters (flying start) = 8.733- .352(vertical jump) . By knowledge of the variance of the better strength and power measures, 83.2% to 98.0% of the variance of the respective sprint tests were explained. The regression models could allow for the identification of potential sprint performers and the development of optimal sprint training program.

Context: It has been suggested that muscle fatigue can lead to injury, however, research investigating this phenomenon in functional ankle instability (FAI) subjects is lacking. Aim: The purpose of this thesis was to research postural sway and muscular latency in FAI subjects and healthy controls, both before and immediately after localised and globalised fatigue protocols. Subjects: All subjects used in this project were males, between the ages of 18 and 25 years, and participated in regular (>2 x week) aerobic exercise. Subjects were categorised into healthy subjects, or subjects with a history of FAI using the FAI questionnaire. Methods: Neuromuscular control was analysed in FAI subjects and healthy controls through measures of muscular latency and postural sway. These measures were repeated both before and immediately after localised and globalised fatigue protocols. Results: The induction of localised and globalised fatigue had no effect on muscle latency in the FAI or healthy subjects. However, postural sway was significantly increased in the FAI subjects, following localised and globalised fatigue, with globalised fatigue also significantly increasing postural sway in the healthy subjects. The globalised football-specific fatigue protocol caused the greatest deficits in the FAI subjects, but also the healthy controls. Conclusions: In terms of muscle latency individuals that participate in sports, as well as sports clinicians and coaches, should not be concerned about the theorised relationship between the onset of fatigue and an increased injury risk at the ankle. However, in terms of postural sway the globalised football-specific fatigue protocol caused the greatest deficits. This highlights that the fatigued individual may be at greater risk of musculoskeletal injury during prolonged exercise that involves multiple joints, such as a football match.

Many devices are available on the market for the evaluation and rehabilitation of patients suffering from muscular disorders. Most of them are small, low-cost, passive devices based on the use of springs and resistive elements and exhibit very limited (or even not any) evaluation capabilities; extended muscular force evaluation is only possible on stationary, expensive, multi-purpose devices, available only in hospitals, which offer many exercise modes (e.g. isokinetic mode) that are not available on other devices.<p><p>The objective of this thesis is to make the functionalities currently only implemented on bulky multi-purpose devices available at a lower cost and in a portable fashion, enabling their use by a large number of independent practitioners and patients, even at home (tele-medecine applications).<p><p>In order to achieve this goal, a portable rehabilitation device, using a magneto-rheological fluid brake as actuator, has been designed. This particular technology was selected for its high level of compactness, simple mechanical design, high controllability, smooth and safe operation. The first part of this thesis is devoted to the design of MR-fluid brakes and their experimental validation. The second part is dedicated to the design of the rehabilitation device and the comparison of its performances with a commercial multi-purpose device (CYBEX). / Doctorat en Sciences de l'ingénieur / info:eu-repo/semantics/nonPublished

Musculoskeletal injuries are commonly associated with muscle atrophy as a function of immobilization or change of normal function. For example, injuries to the anterior cruciate ligament (ACL) which may involve ligament reconstruction, results in the "quadriceps avoidance" gait which leads to atrophy of the knee extensormuscles. In these situations it is not clear whether or not the atrophy is associated with loss of specific muscle fibre types with accompanying functional deficits. Such knowledge would be helpful in implementing exercise regimes designed to compensate for loss of particular fibre types. It is believed that isokinetic exercise performed at speeds below 180° per second strengthens type I muscle fibres, and type II fibres at fast speeds. However, there is no evidence to indicate the specific muscle fibre response to different rates of muscle contraction. Identification of muscle fibre type is most directly determined by biopsy technique but is too invasive for a routine measurement. Electromyography median frequency has been used as a non-invasive measure to infer muscle fibre composition in various studies. However, the reliability and accuracy of this technique has been questioned and improvement is necessary. This research was designed to provide a more accurate and reliable protocol for the determination of EMG median frequency which may be used, after validation against more direct biopsy techniques, as a routine method for inferring muscle fibre composition. The investigation also explored the muscular response as measured by EMG median frequency to varying speeds of muscle contraction, fatiguing exercise and atrophy following ACL reconstruction. The ultimate aim of this research was to improve the reliability of the determination of EMG median frequency to enhance its application as a predictor of muscle fibre composition. This provides information which may improve ACL rehabilitation programs designed to restore and prevent specific muscle fibre types loss that have not previously been targeted by current rehabilitation programs. This research was conducted in three studies. Study one determined the stability of the EMG median frequency bilaterally for the quadriceps and hamstrings muscles and identified the mode of contraction associated with the greatest reliability. The strength and EMG median frequency of the vastus lateralis, medial hamstrings and vastus medialis of 55 subjects was determined across 5 speeds from 0° to 240° per second using a Kin-Com isokinetic dynamometer and an EMG data acquisition system. Isometric contraction was found to have the least bilateral discrepancy (4.01% ±3.06) and between trials standard deviation (4.50) in the vastus lateralis, medial hamstrings and vastus medialis. Study two investigated the EMG median frequency changes in the vastus lateralis which occur immediately following different speeds of isokinetic exercise to the point of fatigue in normal subjects. Thirty-four subjects participated in the study, and performed a 90-second period of isokinetic exercise to activate the knee extensors at either 30° or 300° per second. EMG median frequency of the vastus lateralis was determined before, immediately after and 7 minutes after the fatiguing exercise. The percentage drop in EMG median frequency of the vastus medialis was gnificantly (p<0.05) greater after slow speed (27.9%) than fast speed (20.25%) exercise, while no significant difference was found for the percentage drop in extension torque. Full recovery was found 7 minutes after the fatiguing exercise. By reference to previous research showing a relationship between EMG median frequency and muscle fibre type, an increase in activation of type I muscle fibres with slow speed exercise and an increase in type II muscle fibres with fast speed exercise was observed. Study three identified the changes in EMG median frequency following ACL reconstruction and evaluated the bilateral differences in EMG median frequency of the knee muscles. The relationships between EMG median frequency and the measures of knee functional ability, knee muscle strength, age and time since surgery were also investigated. Twelve subjects who had undergone ACL reconstruction using a semitendinosus and gracilis graft 6 to 12 months earlier, participated in the study. EMG median frequency was determined from an 8-second isometric contraction and knee functional ability was assessed using the Cincinnati Rating Scale. Bilateral EMG median frequency shifts were inconsistent among subjects. On the basis of previous research which indicated a relationship between EMG median frequency and fibre type, no consistent pattern of muscular fibre type atrophy subsequent to ACL reconstruction occurred within 6 to 12 months (ranged from -43 to 57 Hz). Additionally, no significant correlations were found between the EMG median frequency and the knee functional score and knee extension and flexion torques, age, time since operation and the bilateral differences in EMG median frequency. The results of this investigation will serve to improve the reliability of EMG median frequency across a range of conditions in which it has been evaluated. Further research is needed to confirm the relationship between EMG median frequency and direct observations of muscle fibre composition to improve the predictive value of this measure. Following this validation it will be possible to evaluate the bilateral EMG median frequency shift to infer the type of muscle fibre atrophy, and use this measure in determining the efficacy of specific rehabilitation programs. In conclusion * An 8-second isometric contraction is recommended for determining EMG median frequency. * EMG median frequency of a muscle decreases significantly more after slow fatiguing exercise than after fast speed fatiguing exercise. * There was no generalised bilateral EMG median frequency shift found in a group of subjects 6 to 12 months following semitendinosus and gracilis graft ACL reconstruction. * The results of this study will serve to improve the reliability of procedures used to determine EMG median frequency under a range of different contractile conditions. The EMG median frequency changes in response to these conditions require further validations with muscle biopsy in future.

Made available in DSpace on 2016-06-02T20:18:10Z (GMT). No. of bitstreams: 1 2010.pdf: 2854762 bytes, checksum: b0421ce836a4726be750d7bf5a373fb9 (MD5) Previous issue date: 2008-08-18 / Universidade Federal de Sao Carlos / The ageing process is known to affect different tissues and systems. It is well-established that age-associated changes in cardiovascular structure and function are related to the risk of cardiovascular diseases. Because of the vast amount of cardiovascular modifications observed with ageing, the present study focused on three important topics: heart rate variability (HRV), blood pressure variability (BPV) and endothelial dysfunction. Furthermore, we also investigated the effects of physical activity (endurance and strength) on the autonomic control of heart rate (HR), which might be used as non-pharmacological therapy. Thirty five young subject between 18 and 30 years old (14 sedentary men, 5 sedentary women and 16 active men) and thirty eight middleaged/older subjects between 55 and 70 years old (16 sedentary men, 14 sedentary women and 8 active men) were studied. In addition, the subjects are distributed among 3 different studies. In the first one, the effects of the ageing process and active life-style on the autonomic control of HR were investigated in young and middleaged/older subjects. Electrocardiogram was recorded during 15 minutes of rest and 4 minutes of controlled breathing (5 to 6 cycles/min) in the supine position. HR and RR intervals were analyzed by time and frequency domain methods. The active groups presented lower HR and higher HRV (time domain) than the sedentary groups, whereas both middle-aged/older groups showed lower HRV (frequency domain). Additionally, interaction between ageing and life-style effects was observed for respiratory sinus arrhythmia (ASR) indexes (calculated during the controlled breathing test). The sedentary middle-aged/older group presented lower ASR magnitude than the other groups studied. The results suggest that ageing reduces HRV, however, regular physical activity improves vagal modulation on the heart and, consequently, attenuates the effects of ageing on the autonomic control of HR. In the second study, we aimed to investigate if strength training is able to improve cardiac autonomic control in healthy middle-aged/older men. HRV was evaluated before and after 12 weeks of isokinetic eccentric strength training (2days/week, 2-4 sets of 8-12 repetitions at 75-80% peak torque), involving knee flexion and extension. Strength training decreased the systolic blood pressure and increased the torque. However, an autonomic imbalance towards sympathetic modulation predominance was induced by an unknown mechanism. In the third study, we evaluated the effect of ageing on the BPV and endothelial function. We also sought for correlations between increased BPV and impaired endothelium dependent-dilation (EDD) in the middle-aged/older group. Intra-brachial artery BPV and conduit vessel EDD (brachial artery flow-mediated dilation, FMD) were determined in healthy young and middle-aged/older subjects. Moreover, endothelial function of resistance vessels was evaluated by venous occlusion plethysmoghaphy in the middle-aged/older group. The young group presented lower systemic oxidative stress, lower systolic BPV and higher FMD compared with the middle-aged/older group. After split this group according to the BPV, lower FMD was observed in the middleaged/older group with higher BPV. In addition, FMD was inversely correlated to BPV. The lower BPV group showed a great reduction (55%) in the forearm blood flow responses when NG-monometyl-L-arginine (nitric oxide inhibitor) was co-infused with acetylcholine (vs 14% in the higher BPV group). The results suggest that ageing process increases BPV and reduces endothelial function. Additionally, middle-aged/older subjects with higher BPV also have impaired EDD compared with their peers with lower BPV. General Conclusions: the results from the studies described above suggest that ageing process causes decrease of HRV, increase of BPV and decrease of endothelial function. Moreover, aerobic exercise has a cardioprotector effect, since it was able to attenuate the ageing effects on the cardiac vagal modulation. This same benefit, however, was not observed after 12 weeks of eccentric strength training. On the other hand, the strength training program performed by healthy older subjects modified the sympato-vagal balance toward the sympathetic modulation. Finally, systolic BPV oscillations seem to have a narrow relationship with vasodilation mediated by nitric oxide. Then, more studies are needed to clarify the cause-effect relation between those important variables. / O envelhecimento é um processo complexo que causa alterações em vários sistemas do organismo. Em relação ao sistema cardiovascular, modificações na sua estrutura e função estão diretamente relacionadas com o risco aumentado de desenvolvimento de doenças cardiovasculares em idosos. Dentre as diversas alterações cardiovasculares observadas com o envelhecimento, apenas as relacionadas à variabilidade da freqüência cardíaca (VFC), variabilidade da pressão arterial (VPA) e disfunção endotelial foram abordadas no presente estudo. Além disso, foram também investigados os efeitos de dois tipos distintos de exercício físico, ou seja, de resistência aeróbia e de força muscular, sobre o controle autonômico da freqüência cardíaca (FC) de sujeitos saudáveis, como uma forma alternativa de terapia não-farmacológica. Participaram do presente estudo: 35 sujeitos jovens na faixa etária de 18 a 30 anos (14 homens sedentários, 5 mulheres sedentárias e 16 homens ativos) e 38 sujeitos meia-idade/idosos na faixa etária de 55 a 70 anos (16 homens sedentários, 14 mulheres sedentárias e 8 idosos ativos), os quais estão distribuídos em 3 estudos distintos. No primeiro estudo, os efeitos do envelhecimento e do estilo de vida sobre o controle autonômico da FC foram investigados em jovens e meiaidade/idosos com padrão de vida sedentário ou ativo. O sinal eletrocardiográfico foi obtido durante 15 minutos de repouso e 4 minutos de respiração controlada (5-6 ciclos/min), ambos na posição supina. A FC e os intervalos RR foram analisados no domínio do tempo e da freqüência. Adicionalmente, os índices da arritmia sinusal respiratória (ASR) também foram calculados. Os grupos ativos apresentaram menor FC e maior VFC (domínio do tempo) em relação aos grupos sedentários, enquanto que ambos os grupos idosos apresentaram menor VFC (domínio da freqüência). Além disso, foi observado interação entre idade e estilo de vida, já que a magnitude da ASR foi menor no grupo meia-idade/idoso sedentário comparativamente aos demais grupos. Os resultados indicam que a VFC reduz com o aumento da idade. Entretanto, a atividade física regular produz efeitos positivos sobre a modulação vagal cardíaca e, conseqüentemente, atenua os efeitos do envelhecimento sobre o controle autonômico da FC. No segundo estudo, foi avaliado se o treinamento de força excêntrica é capaz de modificar o controle autonômico da FC de idosos saudáveis. A VFC foi avaliada, durante o repouso supino e sentado, após 12 semanas de treinamento de força isocinética excêntrica (extensão e flexão do joelho, 2 dias/semana, 2-4 séries de 8-12 repetições, 75-80% do pico de torque). O treinamento de força foi capaz de aumentar o torque muscular e reduzir a pressão arterial (PA) sistólica de idosos saudáveis. Entretanto, o mesmo causou um desbalanço simpato-vagal, em direção a predominância simpática, o qual foi produzido por mecanismos desconhecidos. No terceiro estudo, foi avaliado se a VPA está aumentada com o avançar da idade e, ainda, se a mesma tem alguma relação com reduções na vasodilatação endotélio-dependente (VED) em sujeitos meiaidade/idosos saudáveis. A VPA intra-arterial e a VED (dilatação mediada por fluxo, DMF) da artéria braquial (i.e., vaso de condutância) foram avaliadas em sujeitos jovens e meia-idade/idosos de ambos os sexos. Adicionalmente, o grupo meia-idade/idoso também foi submetido à pletismografia de oclusão venosa para avaliar a função endotelial dos vasos de resistência. Os jovens apresentaram menor estresse oxidativo sistêmico, menor VPA sistólica e maior DMF, comparativamente ao grupo meia-idade/idoso. Quando esse grupo foi dividido de acordo com a VPA, observou-se DMF reduzida no grupo com alta VPA. Adicionalmente, a DMF mostrou correlação inversa com a VPA. Em relação aos vasos de resistência, o grupo com baixa VPA mostrou redução de 55% na resposta do fluxo sangüíneo quando NG-monometil-L-arginina (inibidor da produção de óxido nítrico) foi co-infudido com acetilcolina (vs 14% no grupo com alta VPA). Os resultados indicam que o envelhecimento causa redução da função endotelial e aumento da VPA. Além disso, sujeitos meia-idade/idosos com alta VPA apresentam DMF reduzida quando comparados aos seus pares com baixa VPA. Conclusão geral: os resultados obtidos nos três estudos sugerem que o envelhecimento causa redução na VFC, aumento da VPA e redução da função endotelial. Além disso, a atividade física aeróbia possui um efeito cardioprotetor, já que essa foi capaz de atenuar os efeitos do envelhecimento sobre a modulação vagal cardíaca. Entretanto, esses efeitos benéficos não foram observados com o treinamento de força excêntrica, pois 12 semanas de treinamento alteraram o balanço simpato-vagal em direção a modulação simpática. Por fim, o aumento nas oscilações da PA sistólica mostrou uma estreita relação com a vasodilatação mediada pelo óxido nítrico, a qual necessita de maiores investigações no sentido de determinar a relação de causa e efeito entre essas duas importantes variáveis.