Die vorliegende Arbeit beschäftigt sich mit der Fragestellung, ob die Frühgeburtlichkeit eine Auswirkung auf den Spracherwerb im ersten Lebensjahr hat. Insbesondere wurde der Frage nachgegangen, ob sich die Verarbeitung der rhythmisch-prosodischen Eigenschaften von Sprache im ersten Lebensjahr und deren weitere Ausnutzung für die Entwicklung des Lexikons bei sehr untergewichtigen Deutsch lernenden Frühgeborenen im Vergleich zu Reifgeborenen unterscheidet. Die besondere Spracherwerbssituation Frühgeborener liefert weitere Erkenntnisse bezüglich der Frage, inwieweit der frühe Spracherwerb durch prädeterminierte reifungsbedingte Mechanismen und Abläufe bestimmt wird und inwieweit dessen Verlauf und die relevanten Erwerbsmechanismen durch individuelle erfahrungsabhängige Faktoren beeinflusst werden. Damit liefern die Ergebnisse auch einen weiteren Beitrag zur Nature-Nurture-Diskussion. / This research addresses the question if and how premature birth effects language acquisition during the first year of life. In particular the study focus on whether prosodic processing of language and the utilization of this knowledge for the acquisition of the lexicon during the fist year of life differs between German learning very low birth weight infants and term born infants. The specific exposure and postnatal situation of premature infants provide insights on the determination of early language acquisition and processes, and on the determination of language acquisition and its relevant mechanisms by individual experience. The results contribute to the nature-nurture discussion.

This study examined people’s attitudes towards killing bugs and their bug-killing behaviour in the context of nature vs. nurture explanations of bug killing. Previous research shows that exposure to genetic (i.e., nature) explanations could have undesirable effects on people’s attitudes and behaviour, compared to the exposure to experiential(i.e., nurture) explanations. Genetic explanations for killing may affect attitudes towards killing and killing behaviour, because they suggest that killing behaviour is predetermined or programmed by nature. Such explanations may also be used by individuals to overcome guilt and dissonance from prior killing or killing in which they are about to participate. This study tested the idea that exposure to genetic explanations for bug killing would lead people to view killing bugs as more morally acceptable, as well as lead them to kill more bugs. A sample of university students was randomly assigned into three conditions, in which they read either genetic or experiential explanations for why people kill bugs or read a neutral passage. The study utilised a procedure in which participants were led to believe that they were killing bugs (although in actuality no bugs were killed), to observe their killing behaviour in a self-paced killing task. Half of the participants were also asked to kill a bug prior to the self-paced killing task. Results showed that participants who read genetic explanations viewed bug killing as more morally acceptable, compared to participants who read experiential explanations, and this occurred particularly among those who engaged in the prior killing task. However, no similar effects emerged for the number of bugs killed, though there was a positive correlation between the moral acceptance of bug killing and the number of bugs killed. Implications of genetic explanations with respect to aggression and killing are discussed.

This study examined people’s attitudes towards killing bugs and their bug-killing behaviour in the context of nature vs. nurture explanations of bug killing. Previous research shows that exposure to genetic (i.e., nature) explanations could have undesirable effects on people’s attitudes and behaviour, compared to the exposure to experiential(i.e., nurture) explanations. Genetic explanations for killing may affect attitudes towards killing and killing behaviour, because they suggest that killing behaviour is predetermined or programmed by nature. Such explanations may also be used by individuals to overcome guilt and dissonance from prior killing or killing in which they are about to participate. This study tested the idea that exposure to genetic explanations for bug killing would lead people to view killing bugs as more morally acceptable, as well as lead them to kill more bugs. A sample of university students was randomly assigned into three conditions, in which they read either genetic or experiential explanations for why people kill bugs or read a neutral passage. The study utilised a procedure in which participants were led to believe that they were killing bugs (although in actuality no bugs were killed), to observe their killing behaviour in a self-paced killing task. Half of the participants were also asked to kill a bug prior to the self-paced killing task. Results showed that participants who read genetic explanations viewed bug killing as more morally acceptable, compared to participants who read experiential explanations, and this occurred particularly among those who engaged in the prior killing task. However, no similar effects emerged for the number of bugs killed, though there was a positive correlation between the moral acceptance of bug killing and the number of bugs killed. Implications of genetic explanations with respect to aggression and killing are discussed.

Die Dissertation thematisiert sprecherspezifische Variabilität bei ein- und zweieiigen Zwillingen hinsichtlich Artikulation, Akustik und Perzeption. Die zentrale Fragestellung ist, ob sprecherspezifische Charakteristika auf physiologisch-biologischen Differenzen der Sprecher beruhen (BIOLOGIE), oder sich auf gelernte, umweltabhängige Unterschiede zurückführen lassen (UMWELT). Artikulatorische und akustische Daten wurden von 4 eineiigen Zwillingspaaren (EZ, 100% genetische Übereinstimmung) und 3 zweieiigen Zwillingspaaren (ZZ, 50% genetische Übereinstimmung) analysiert. Zusätzlich wurde ein Perzeptionstest zur auditiven Ähnlichkeit der Zwillinge durchgeführt. Auf einen großen Einfluss des Faktors BIOLOGIE lässt sich schließen, wenn sich EZ ähnlicher sind als ZZ. Sind sich aber ZZ genauso ähnlich wie EZ, zeigt sich die Wichtigkeit der gleichen Lernumgebung (UMWELT). Die Ergebnisse weisen auf einen großen Einfluss des Faktors UMWELT und stützen die Hypothese, dass sprachliche Ziele gelernt sind und sich am auditiven Feedback orientieren. Darüber hinaus wurden drei Faktoren gefunden, die den Einfluss der BIOLOGIE intensivieren: a) Lautklasse, b) Wortakzent und c) Koartikulation. Plosive und Sibilanten sind aufgrund des stärker ausgeprägten linguo-palatalen Kontaktes mehr durch die individuelle Physiologie beeinflusst als Vokale. Außerdem wurde ein größerer Effekt des Faktors BIOLOGIE in unbetonten als in betonten Silben gefunden. Zusätzlich stellten sich koartikulatorische Prozesse als wichtig heraus: dynamische Parameter – artikulatorische Gesten und akustische Transitionen – sind stärker durch die Physiologie beeinflusst als statische Parameter – artikulatorische Ziele und stabile akustische Regionen. Sowohl der Faktor BIOLOGIE als auch der Faktor UMWELT sind einflussreiche Größen hinsichtlich sprecherspezifischer Variabilität. Welcher der beiden Faktoren die übergeordnete Rolle übernimmt, hängt von den spezifischen Charakteristika des untersuchten Parameters ab. / This dissertation examines inter-speaker variability in monozygotic (MZ) and dizygotic (DZ) twin pairs in regard to articulation, acoustics and perception. The aim of the study is to evaluate whether speaker-specific variability reflects physiological differences between speakers (NATURE) or bases on learned variation due to social environmental influences (NURTURE). Articulatory and acoustic data was analyzed from 4 MZ twin pairs (100% identical genes) and 3 DZ twin pairs (50 % identical genes). Additionally, a perception experiment was carried out to explore the perceived auditory similarity. The effect of NATURE should have a larger impact than the effect of NURTURE, if a parameter differs more in DZ than in MZ twin pairs. If MZ and DZ twins show the same amount of inter-speaker variability, NURTURE seems to be crucial. Results point to the importance of NURTURE and shared social environment. Nevertheless, three factors were found that intensify the effect of NATURE: a) phoneme class, b) lexical stress, and c) degree of coarticulation. Somatosensory feedback plays a larger role for consonants than for vowels, and thus individual physiology was found to shape articulation more in sibilants and stops than in vowels. Additionally, a stronger impact of NATURE was found in parameters that are auditorily less salient: unstressed syllables were more similar in MZ than in DZ twins, while for stressed syllables this was not the case. Moreover, coarticulation turned out to be essential: dynamic parameters – articulatory gestures and acoustic transitions – were more influenced by physiological constraints (NATURE) than static parameters – articulatory targets and stable acoustic regions. Thus, both NATURE and NURTURE are crucial influencing factors in speaker-specific variability. However, the relative importance of the two factors is highly dependent on the specific characteristics of the investigated parameter.

Behavioural scientists argue that ‘social learning' provides the link between biological phenomena and cultural phenomena because of its role in the ‘cultural transmission' of knowledge among individuals within and across generations. However, leading authors within the social sciences have proposed alternative ways of thinking about social life not founded on the Modern oppositions including nature-culture, biology-culture, body-mind, and individual-society. Similarly, the distinction between a domain of nature and a domain of nurture has also been extensively criticized within biology. Finally, advocates of ‘radical embodied cognitive science' offer an alternative to the representational-computational view of the mind which supports the conventional notion of culture and cultural information. This thesis attempts to integrate developmental systems theory, radical embodied cognitive science, and relational thinking, with the goal to bring the field of social learning closer to these critical theoretical developments. In Chapter 2, I find no justification for the claim that the genome carries information in the sense of specification of biological form. Chapter 3 presents a view of ontogeny as a historical, relational, constructive and contingent process. Chapter 4 uses the notions of environmental information, abilities, affordances, and intentions to make sense of behaviour and learning. In Chapter 5, I argue that the notion of social learning can be understood in terms of relational histories of development rather than in terms of transmission of information. I then report empirical studies investigating behavioural coordination and social learning consistent with this theoretical framework. Chapter 6 presents evidence that dyads in a joint making activity synchronize their attention constrained by their changing situation and that coordination of attention is predictive of implicit and explicit learning. Chapter 7 presents evidence that joint attention does not require gaze following and that attentional coordination is predictive of learning a manual task. Together, these theoretical and empirical studies suggest a new way of thinking about how humans and other animals live and learn socially, one that is consistent with critical theoretical and philosophical developments that are currently neglected in the literature on social learning.

La présente étude porte sur les aspects Nature-Culture relatifs à l’émergence de variations interindividuelles quant à la capacité universelle de régulation d’une émotion primaire, la tristesse. Cette problématique représente un exemple du lien entre la conception évolutionniste d’une nature humaine universelle, innée et génétiquement prescrite, mais susceptible de variation dans son expression en fonction d’expériences individuelles liées aux processus de socialisation et d’enculturation. À l’aide du devis génétiquement informatif des jumeaux, nous nous sommes d’abord penchés sur l’étiologie gènes-environnement de la dépression à l’enfance, une dysfonction du système de régulation émotionnelle de la tristesse. Puis, nous nous sommes interrogés quant à l’influence du traitement et de l’état psychique maternels sur cet aspect du développement émotionnel de l’enfant. Nos analyses de la symptomatologie dépressive indiquent une absence d’influence génétique dans le développement de ce trouble de l’humeur. Les variations individuelles de la régulation de la tristesse reposent ainsi uniquement sur les effets de l’environnement. Nos résultats révèlent également l’existence d’une relation importante entre l’état psychique de la mère, évalué lorsque les jumeaux avaient cinq mois, et la présence de symptômes dépressifs chez ces derniers mesurés huit ans plus tard. L’état psychique de la mère est considéré comme l’un des meilleurs indicateurs de la qualité du traitement maternel en bas âge. Nos mesures directes des comportements maternels envers le nourrisson et le développement ultérieur du trouble de dépression indiquent également l’existence de tendances statistiques allant dans le sens de notre hypothèse d’un traitement maternel sous-optimal contribuant au développement de dysfonctions émotionnelles ultérieures. / The present study explores the Nature-Nurture aspects pertaining to the emergence of interindividual variation in the emotional regulation of a primary emotion, sadness. This question is an example of the more encompassing issue of a universal human nature, biologically evolved and genetically underwritten, being nevertheless variable in its expression depending on individual experiences linked to socialization and enculturation. Using a genetically informative twin design, we explored the genetic-environmental aetiology of child depression, which is understood to be a dysfunction in the emotional regulation system of sadness. We then assessed the influences of maternal behavior and mental state on the development of children’s depressive symptoms. Our analysis of child depression shows an absence of genetic influence in the emergence of this mood disorder. Thus, individual variation in sadness regulation appears to be solely generated by environmental influences. Our results also show a strong and significant relation between maternal psychological state, assessed when the twins were 5 months of age, and children’s depressive symptoms measured 8 years later. Maternal psychological state is considered to be one of the most reliable indicators of the quality of maternal treatment in childhood. Our direct measures of maternal behavior toward the infant and the subsequent development of depressive disorder also point to statistical trends supporting our hypothesis of suboptimal maternal treatment contributing to the development of later emotional dysfunction.

La présente étude porte sur les aspects Nature-Culture relatifs à l’émergence de variations interindividuelles quant à la capacité universelle de régulation d’une émotion primaire, la tristesse. Cette problématique représente un exemple du lien entre la conception évolutionniste d’une nature humaine universelle, innée et génétiquement prescrite, mais susceptible de variation dans son expression en fonction d’expériences individuelles liées aux processus de socialisation et d’enculturation. À l’aide du devis génétiquement informatif des jumeaux, nous nous sommes d’abord penchés sur l’étiologie gènes-environnement de la dépression à l’enfance, une dysfonction du système de régulation émotionnelle de la tristesse. Puis, nous nous sommes interrogés quant à l’influence du traitement et de l’état psychique maternels sur cet aspect du développement émotionnel de l’enfant. Nos analyses de la symptomatologie dépressive indiquent une absence d’influence génétique dans le développement de ce trouble de l’humeur. Les variations individuelles de la régulation de la tristesse reposent ainsi uniquement sur les effets de l’environnement. Nos résultats révèlent également l’existence d’une relation importante entre l’état psychique de la mère, évalué lorsque les jumeaux avaient cinq mois, et la présence de symptômes dépressifs chez ces derniers mesurés huit ans plus tard. L’état psychique de la mère est considéré comme l’un des meilleurs indicateurs de la qualité du traitement maternel en bas âge. Nos mesures directes des comportements maternels envers le nourrisson et le développement ultérieur du trouble de dépression indiquent également l’existence de tendances statistiques allant dans le sens de notre hypothèse d’un traitement maternel sous-optimal contribuant au développement de dysfonctions émotionnelles ultérieures. / The present study explores the Nature-Nurture aspects pertaining to the emergence of interindividual variation in the emotional regulation of a primary emotion, sadness. This question is an example of the more encompassing issue of a universal human nature, biologically evolved and genetically underwritten, being nevertheless variable in its expression depending on individual experiences linked to socialization and enculturation. Using a genetically informative twin design, we explored the genetic-environmental aetiology of child depression, which is understood to be a dysfunction in the emotional regulation system of sadness. We then assessed the influences of maternal behavior and mental state on the development of children’s depressive symptoms. Our analysis of child depression shows an absence of genetic influence in the emergence of this mood disorder. Thus, individual variation in sadness regulation appears to be solely generated by environmental influences. Our results also show a strong and significant relation between maternal psychological state, assessed when the twins were 5 months of age, and children’s depressive symptoms measured 8 years later. Maternal psychological state is considered to be one of the most reliable indicators of the quality of maternal treatment in childhood. Our direct measures of maternal behavior toward the infant and the subsequent development of depressive disorder also point to statistical trends supporting our hypothesis of suboptimal maternal treatment contributing to the development of later emotional dysfunction.