No description available.

No description available.

A unilingual and a bilingual primed lexical decision task were used to investigate priming effects produced by attended and ignored visual stimuli. In the Chinese language unilingual experiment, accelerated responses to the traditional Chinese character probe targets were observed when the traditional character probe target was the same as the preceding prime target (i.e., attended repetition, AR). However, when a traditional character “matched” a preceding simplified Chinese character prime distractor (i.e., ignored repetition, IR), the expected impaired responses (negative priming) were not observed. In the bilingual experiment (Chinese – English), prime stimuli were in Chinese and probe stimuli were in English. Both AR positive priming and IR negative priming between Chinese – English translation equivalents were produced by bilingual subjects in experiment 2. Further analyses were carried out by dividing subjects into two groups, one less proficient and the other more proficient in English. The contrasting patterns of performance produced by the more and less proficient bilinguals indicate that inhibitory mechanisms can simultaneously operate at two levels of abstraction – global language and local word; and these two types of inhibition can work in a quite independent manner. The contrasting response patterns by the more versus less proficient bilingual subjects also convincingly suggest shared storage for the conceptual representations of a Chinese-English bilingual’s two languages. Moreover, obtaining negative priming in Experiment 2, which uses a large set of 795 words as stimuli, provides strong evidence against the notion that negative priming is contingent on stimulus repetition. Rather, it confirms that processing demand or selection difficulty is critical for producing negative priming.

No description available.

In this thesis, I characterized the association of Arf4GDP with ER-Golgi intermediate compartment membranes. We confirmed that GDP-arrested Arf4 mutants associated with membranes irrespective of nature of tag or mutation. Recruitment appeared specific since loss of N-terminal myristoylation abolished binding. Surprisingly, mutations of residues unique to class II Arfs did not prevent recruitment of Arf4 to peripheral puncta. We then examined the failure of the GDP-arrested Arf4 mutant to disrupt Golgi structure. We identified residues R79 and E113 (likely involved in salt bridge interaction) only present in Arf1 and Arf5 as critical to the ability of their GDP-arrested mutants to disrupt Golgi structure. As predicted, introduction of these residues transformed Arf4GDP into a dominant negative mutant. Interestingly, overexpression of the putative ArfGDP receptor membrin prevented the effects of dominant negative Arf1 but not dominant negative Arf4. These results will facilitate identification of a novel Arf target critical to protein trafficking.

Thesis (Ph. D.)--Virginia Polytechnic Institute, 1963. / Vita. Abstract. Includes bibliographical references (leaves 124-126). Also available via the Internet.

Thesis (doctoral)--Rheinische Friedrich-Wilhelms-Universität, Bonn, 2005. / Description based on print version record. Includes bibliographical references (p. [225]-242) and indexes.

Thesis (M.S.)--California Polytechnic State University, 2008. / "June 2008." "In partial fulfillment of the requirements for the degree [of] Master of Science in Electrical Engineering." "Presented to the faculty of California Polytechnic State University, San Luis Obispo." Major professor: Dean Arakaki, Ph.D. Includes bibliographical references (leaves 146-148). Also available online and on microfiche (2 sheets).

Thesis (doctoral) - Rheinische Friedrich-Wilhelms-Universität, Bonn, 2005. / Includes bibliographical references (p. [225]-242) and indexes.

University, Diss., 2006--Würzburg.