Thesis (Ph. D.)--University of Wisconsin--Madison, 1936. / Typescript. Includes abstract and vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves [317]-318).

Thesis (Ph. D.)--University of Wisconsin--Madison, 1958. / Typescript. Abstracted in Dissertation abstracts, v. 19 (1958) no. 6, p. 1157-1158. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 126-134).

Thesis (Ph. D.)--University of Wisconsin--Madison, 1961. / Typescript. Vita. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references.

No description available.

Thesis (B. Sci. (Hons.))--University of Adelaide, Faculty Agricultural Science,1955. / Typewritten copy. Includes bibliographical references.

Typescript, etc. / Digitized by Kansas State University Libraries

Two varieties of sideoats grama (Vaughn and NM-28) were planted in late summer of 1980. The seedlings were treated with a variety of broadleaf weed control treatments at 1-, 3-, 5-leaf, and tillering stages of grass seedling growth to determine the effectiveness of these treatments for controlling weeds and the appropriate stages of grass seedling growth to apply them and consequently on grass establishment, density, forage production and plant height. Four months after seeding, NM-28 stands were more dense, produced more forage and the plants were taller than Vaughn. Treatments of dicamba at 0.28 and 0.56 kg/ha were the most effective herbicidal treatments in controlling weeds and resulted in highest stand densities and yields when applied at the 1- and 3-leaf seedling growth. Both treatments gave over 90% control of weeds at the earlier stages but control decreased at later stages of treatment. Lower control resulted when 2,4-D at 0.56 and 1.12 kg/ha were applied. Even though hand weeding treatment resulted weed-free plots during the first four months of seedling development, it resulted in less stand densities and yields when compared to herbicidal treatments. One year after treatment, Vaughn established into taller stands and produced higher yields than NM-28. Dicamba 0.28 kg/ha and 2,4-D 0.56 kg/ha effectively aided in better stand density and yield. Treatments applied at earlier stages of growth resulted in better establishment of both varieties. Dicamba at 0.28 kg/ha was the most effective herbicidal treatment for both varieties. However, hand weeding evaluated one year after seeding, effectively increased both accessions' yield and was the most effective treatment. Greenhouse and laboratory experiments were conducted to determine the uptake and distribution of ('14)C-2,4-D in both sideoats grama accessions and palmer amaranth seedling plant parts at 1, 4, 8 and 24h after treatment. Over all harvest times, 25.3, 2.8 and 3.1% activity were recovered in Vaughn treated leaf, tillers and crown, respectively. That was compared to 33.9, .68 and 6.8% in the same NM-28 plant parts, respectively. Higher percentages of activity were recovered in palmer amaranth plant parts; 50.5% in treated leaf, 8.5% above treated leaf and 5.4% below.

No description available.

Thesis (Ph. D.)--Oregon State University, 2007. / Printout. Includes bibliographical references (leaves 101-112). Also available on the World Wide Web.

Celiac disease (CD) is an immune-mediated enteropathy caused by permanent intolerance to dietary gliadin in wheat gluten and related prolamines in barley and rye. The pathogenesis of CD is still unknown and several different environmental factors have been associated with CD, such as dysbiosis of the microflora. In this translational study we investigated the immune status and the interplay of T-cells and Tregs in the mucosa of children with CD and controls, as well as the immune status in treated CD patients, provoked by either dietary oats, CD associated bacteria or gluten. The major findings in the studies were: First, indicators of extrathymic T-cells maturation (ETCM), i.e., the RAG1 enzyme required for recombination of the T cell receptor (TCR) genes and the preTα-surrogate chain in the immature TCR, were both expressed at lower levels in CD patients compared to controls. In addition, IELs expressing RAG1 were less abundant in CD patients compared to controls. The levels of these two indicators stayed low in treated CD patients as well, suggesting that impaired capacity of ETCM is an inherent feature of CD patients. Second, IL-17A, a cytokine involved in both inflammation and anti-bacterial responses was increased in active CD. The major cellular source was CD8+IELs. Furthermore, ex vivo challenge of biopsies from treated CD patients with gluten and with CD-associated bacteria induced an IL-17A response. The CD-associated bacteria also influenced the magnitude of the IL-17A response to gluten. Third, we investigated the effect of dietary oats on local immune status in the intestinal mucosa by comparing CD patients receiving GFD with and without oats. 22 different mRNAs for immunity effector molecules and tight junction proteins were analyzed. We found that expression of two down-regulatory cytokines, two activating NK-receptors and the tight-junction protein claudin-4 normalized in patients on a standard GFD while they did not normalize in patients on a GFD with oats. Fourth, we analyzed the expression level of mRNAs for chemokines, cytotoxic effector molecules, NK-receptors and their ligands in IELs and epithelial cells. Expression levels of several of these genes follow disease activity, suggesting massive recruitment of immune cells by both cell types accompanied by increased IEL-mediated cytotoxicity in the epithelium of inflamed mucosa. In this thesis we have identified three potential risk factors for development of CD: 1) an inherent lower level of ETCM in the small intestinal mucosa than in controls. This could lead to decreased generation of regulatory T cells and less capacity to tolerate gluten and adapt to the local milieu in the mucosa. 2) Dysbiosis of the resident microbiota with increased IL-17A production that could promote local inflammation and immune cell infiltration as well as antibacterial reactions. 3) Dietary oats may provoke a local immune response in a sub-population of CD patients. These patients should probably avoid oats in their GFD but larger studies are needed.