Made available in DSpace on 2015-04-14T13:33:07Z (GMT). No. of bitstreams: 1 451398.pdf: 671181 bytes, checksum: 9b2ce376bc2dfdff2a7a720c916e7210 (MD5) Previous issue date: 2012-04-27 / Introduction: In children, hearing loss can be a complication of Adenotonsillar Hypertrophy (ATH) due to upper airway obstruction with a corresponding middle ear effusion. Nonetheles, it is not clear if ATH can also lead to impairment of the inner ear. Objective: to detect and characterize hearing impairment in children with Adenotonsillar Hypertrophy.Methods: A case-control study in children with and without Adenotonsillar Hypertrophy. All subjects underwent tonal audiometry, impedance, transient otoacoustic emissions and distortion product otoacoustic emissions. Results: The study involved 32 children with ATH and 37 controls, from 5 to 13 years of age. There were no significant differences between groups regarding gender (p=0.763), age (p=0.349), weight (p=0,653) and race (0.285). Tonal audiometry was normal for both groups in both ears, although the ATH showed slight falls at high frequencies. Transient otoacoustic emissions (TOE) and distortion product otoacoustic emissions (DPOE) were present in both ears of the control group. In the ATH group, TOE were present in the right ear of all children but only in 30% in the left ear; DPOE were present in only 12%, but in 68% of the right ear. Conclusion: There is an association between Adeno-tonsillar Hypertrophy and hearing neurosensory impairment, identified in otoacoustic emissions but not in tonal audiometry / Introdu??o: Em crian?as, a perda auditiva pode ser uma das complica??es da Hipertrofia Adenotonsilar (HAT) devido ? obstru??o de vias a?reas superiores com consequente ac?mulo de secre??o na orelha m?dia (efus?o). Contudo, desconhecemos se a HAT tamb?m compromete a orelha interna. Objetivos: Identificar e caracterizar poss?veis altera??es auditivas em crian?as portadoras de Hipertrofia Adenotonsilar. M?todos: Trata-se de um estudo caso-controle no qual foram recrutadas crian?as com hipertrofia adenotonsilar e seus respectivos controles. Todos os sujeitos realizaram exames de audiometria tonal, impedanciometria, emiss?es otoac?sticas transientes e emiss?es otoac?sticas por produto de distor??o. Resultados: Participaram do estudo 32 crian?as com HAT e 37 controles com idades entre cinco e 13 anos, de ambos os sexos. N?o houveram diferen?as significativas entre os grupos em rela??o a g?nero (p=0,763), idade (p=0,349), peso (p=0,653) e ra?a (p=0,285). Em rela??o ? audiometria tonal, o grupo HAT n?o apresentou diferen?a entre o grupo controle em ambas as orelhas, por?m, o grupo com HAT apresentava discretas quedas nas altas frequ?ncias. As emiss?es otoac?sticas transientes (EOAT) e por produto de distor??o (EOAPD) estavam presentes no grupo controle e em estudo em ambas orelhas. No grupo HAT, as EOAT estavam presentes na orelha direita 100% das crian?as, mas apenas em 30% na orelha esquerda; as EOAPD estavam presentes na orelha direita de apenas 12% das crian?as, mas em 68% delas na orelha esquerda. Conclus?es: H? uma associa??o entre hipertrofia adenotonsilar e altera??es sens?rio-neurais da audi??o, identificadas nas emiss?es otoac?sticas, mas n?o na audiometria tonal