Bakgrund: Stress och fysisk inaktivitet är bland de vanligaste riskfaktorerna till de folksjukdomar vi har idag. Vi lever idag ett mer stillasittande liv. Med hjälp av de digitala hjälpmedel vi har idag finns större möjlighet att få fler att röra på sig och därmed sänka folksjukdomarna och sänka upplevd känsla av stress. Syfte: Syftet med studien var att se huruvida effekterna på stressnivån skiljer sig genom att delta i digitala yogapass inomhus medan instruktören höll yogapasset i inomhus eller utomhusmiljö. Metod: 20 deltagare blev slumpmässigt indelade i två grupper (grupp A eller grupp B). Deltagarna i grupp A började med att delta i ett digitalt yogapass som yogainstruktören höll inomhus för att sedan göra det andra digitala yogapasset, där yogainstruktören höll yogapasset utomhus. Grupp B gjorde passen i omvänd ordning. Innan och efter varje pass fick deltagarna fylla i ett frågeformulär (STAI-S) där de fick skatta sin upplevda stressnivå för stunden. Resultat: Resultatet från Mann-Whitney U test visade inga signifikanta skillnader mellan effekten av stressnivån när en yogainstruktör höll klassen inomhus eller utomhus. Konklusion: Resultatet visade inga signifikanta skillnader. Mer forskning krävs, samt forskning med fler deltagare krävs då ämnet är sparsamt beforskat. / Background: Stress and physical inactivity are the most common risk factors to most of the welfare diseases today. We’re living in a more sedentary environment. Today we have more digital aids that can provide greater opportunity to get more people to move and reduce diseases and reduce perceived feelings of stress. Aim: The aim with this study was to find whether the effects on the stress level differ by participating in digital yoga sessions indoors while the instructor held the yoga session in an indoor or outdoor environment. Method: 20 participants were randomly divided into two groups (group A and group B). The participants in group A started with the study with a digital yogapass where the instructor held the class indoor and for the second class the instructor held the class outdoor. The other group (group B) did the workouts in reverse order. Before and after each class the participants had to fill in a questionnaire (STAI-S) where they had to estimate their perceived stress level for the moment. Results: The result from Mann-Whitney U test showed no significant differences between the effect of the stress level between indoor and outdoor. Conclusion: The results showed no significant differences. More research is required, as well as research with more participants is required as the subject is sparsely researched.

Anxiety disorders are very common and the primary feature is abnormal or inappropriate anxiety. Fear and anxiety is often mediated by the amygdala, a brain structure rich in substance P (SP) and neurokinin 1 (NK1) receptors. To learn more about how the human amygdala is modulated by fear and anxiety in event-triggered anxiety disorders and to investigate if the SP/NK1 receptor system is affected, regional cerebral blood flow (rCBF) ([15O]-water; Study I and II) and the SP/NK1 receptor system ([11C]GR205171; Study III and IV) were studied with positron emission tomography (PET). In Study I we investigated the neural correlates of affective startle modulation in persons with specific phobia by measuring rCBF during exposure to fearful and non-fearful pictures, paired and unpaired with acoustic startle stimuli. Fear-potentiated startle was associated with activation of the affective part of the anterior cingulate cortex and the left amygdaloid–hippocampal area. In Study II short-term drug treatment effects on rCBF in patients diagnosed with social phobia was evaluated, comparing the NK1 receptor antagonist GR205171 to the selective serotonin reuptake inhibitor citalopram and placebo. Social anxiety and neural activity in the medial temporal lobe including the amygdala was significantly reduced by both drugs but not placebo. In Study III we investigated if activity in the SP/NK1 receptor system in the amygdala would be affected by fear provocation in individuals with specific snake or spider phobia. Fear provocation was associated with a decreased uptake of the NK1 antagonist [11C]GR205171 in the amygdala, possibly explained by an increase in endogenous SP release occupying the NK1 receptors. Study IV was conducted to explore the resting state NK1 receptor availability in PTSD patients as compared to healthy controls. Increased resting state binding of the tracer [11C]GR205171 in the amygdala of patients with PTSD suggested an increased amount of available receptors. In summary, fear and fear-potentiated startle modulates the human amygdala, possibly through the SP/NK1 receptor system.