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The effect of superior cervical ganglion sympathectomy on repair of the intermaxillary suture following rapid palatal expansionGluck, Arthur Joel. January 1983 (has links)
Thesis (M.S.)--University of Michigan, Ann Arbor, 1983. / Typescript (photocopy). Includes bibliographical references (leaves 93-104). Also issued in print.
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The effect of superior cervical ganglion sympathectomy on repair of the intermaxillary suture following rapid palatal expansionGluck, Arthur Joel. January 1983 (has links)
Thesis (M.S.)--University of Michigan, Ann Arbor, 1983. / Typescript (photocopy). eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 93-104).
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Developmental Effects of Estrogen on the Superior Cervical Ganglion and HypertensionChiappini-Williamson, Christine 17 April 2009 (has links)
No description available.
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Changes in adult rat superior cervical ganglion following axotomyMorris, Teresa Ann 13 August 2010 (has links)
No description available.
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THE LOCALIZATION OF BASIC FIBROBLAST GROWTH FACTOR (FGF-2) IN RAT SUBMANDIBULAR GLANDSSAKANAKA, MASAHIRO, KOBAYASHI, SHIGERU, UEDA, MINORU, SHIGETOMI, TOSHIO, KOSAKI, KENICHI, KAGAMI, HIDEAKI, HIRAMATSU, YOSHIYUKI 26 December 1994 (has links)
No description available.
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Cocaine- and Amphetamine-Regulated Transcript-Immunoreactivity in the Rat Sympatho-Adrenal AxisDun, N. J., Dun, S. L., Kwok, E. H., Yang, J., Chang, J. K. 07 April 2000 (has links)
Distribution of cocaine- and amphetamine-regulated transcript-like immunoreactivity (CART-LI) was studied in the rat spinal cord, sympathetic ganglia and adrenal glands by immunohistochemical methods, utilizing a polyclonal antiserum raised against the CART peptide fragment 55-102. CART-LI was detected in nerve fibers and in basket-like terminals surrounding many postganglionic neurons of the superior cervical ganglion (SCG), stellate, paravertebral and prevertebral ganglia. Postganglionic neurons exhibited low or non-detectable levels of CART-LI. Surgical sectioning of the cervical sympathetic trunk for 6-7 days resulted in a nearly complete loss of CART-LI fibers and terminals in the SCG. In the adrenal gland, CART-LI nerve fibers formed a plexus underneath the capsule, some of which bifurcated and made a sharp turn toward the adrenal medulla, where clusters of chromaffin cells were intensely labeled. The detection of CART-LI in sympathetic ganglia and adrenal glands extends the previous observation of the presence of CART-LI in sympathetic preganglionic neurons and further supports the notion that CART peptide(s) may function as a signaling molecule in the sympatho-adrenal axis.
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THE ROLE OF NERVE GROWTH FACTOR AND PRE-GANGLIONIC INPUT IN THE REGULATION OF TYROSINE HYDROXYLASE EXPRESSION IN SYMPATHETIC NEURONSMaynard, Lance M. 17 July 2003 (has links)
No description available.
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Plasticity of adult sympathetic neurons following injuryWalker, Ryan G. 14 August 2009 (has links)
No description available.
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Characterizing the regeneration of peripheral neurons: Re-innervation of the superior cervical ganglionWalsh, Brian F. 07 May 2010 (has links)
No description available.
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Žmogaus simpatinio kamieno viršutinio kaklinio mazgo neurohistologinių ypatumų sąsajos su amžiumi ir galvos smegenų išeminiu infarktu / Age-related and ischemic cerebral infarction-related neurohistological peculiarities of the human superior cervical ganglionLiukienė, Gineta 06 March 2008 (has links)
Su amžiumi susiję struktūriniai simpatinės nervų sistemos pokyčiai sukelia įvairių organų funkcijos sutrikimus. Simpatinio kamieno viršutiniai kakliniai mazgai, pagrindiniai galvos smegenų kraujagyslių simpatinės inervacijos šaltiniai, yra svarbūs galvos smegenų kraujotakos autonominės reguliacijos centrai. Tačiau žmogaus simpatinių mazgų morfofunkciniai tyrimai yra pavieniai. Todėl šio darbo tikslas buvo ištirti trijų amžiaus grupių žmonių ir mirusiųjų dėl išeminio galvos smegenų infarkto simpatinio kamieno viršutinio kaklinio mazgo morfologinius bei neurocheminius ypatumus, ir nustatyti jų sąsajas su amžiumi ir išeminiu galvos smegenų infarktu. Tyrimo rezultatai rodo, kad amžiaus eigoje vystosi žmogaus simpatinio kamieno viršutinio kaklinio mazgo neuronų morfologinių parametrų, neurofilamentų baltymo, nervų augimo faktoriaus receptorių p75 imunoreaktyvumo pokyčiai. Skirtingose amžiaus grupėse aptinkami neuronų ir satelitinių ląstelių apoptoz����s požymiai. Pagyvenusio amžiaus žmonių, mirusių dėl išeminio galvos smegenų infarkto, simpatiniuose mazguose vystosi neuropatologinės alteracijos požymiai: padidėja neuronų afinitetas baziniams dažams, atsiranda neuronų su degeneraciniais ir hipertrofijos požymiais židiniai, satelitinių ląstelių proliferacija, limfocitų židininė infiltracija, sumažėja neuronų kūno, branduolio, citoplazmos plotai, aptinkamas didesnis neurofilamentams ir nervų augimo faktoriaus receptoriams p75 imunoreaktyvių neuronų skaičius, didesnis apoptozinis... [toliau žr. visą tekstą] / Age-related structural changes of the sympathetic nervous system cause the disturbance of the involuntary functions of various organs. The superior cervical ganglia are the main source of sympathetic innervation of the cerebral arteries and important center of autonomic regulation of the cerebral circulation. However, only single morphofunctional studies of the human sympathetic ganglia are performed. Therefore, the aim of the study was to investigate morphological and neurochemical peculiarities of the human superior cervical ganglion in three age groups and in the subjects dead of ischemic cerebral infarction and establish their relation to age and ischemic cerebral infarction. The results of the present study show that changes of neurons morphologic parameters and immunoreactivity to neurofilament and nerve growth factor receptor p75 in the human superior cervical ganglion are developed in the course of aging. Morphological features of apoptosis in the sympathetic neurons and satellite cells in different age groups are detected. The signs of neuropathological alteration of the sympathetic ganglion of subjects with ischemic cerebral infarction were the following: increased tinctorial affinity of neurons, foci of neurons with features of neuron death and hypertrophy, proliferation of satellite cells, focal lymphocyte infiltration, decrease of neuron body, nucleus, cytoplasm area, increase of neurofilament and nerve growth factor receptor p75 immunoreactive neurons... [to full text]
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