11 |
Molecular analysis of a female-sterile mutation in Drosophila melanogasterSaunders, Robert David Comrie January 1987 (has links)
No description available.
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12 |
Acquisition of meiotic competence in growing porcine oocytesChristmann, Leandro January 1994 (has links)
No description available.
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13 |
The lemming locus of Drosophilia melangasterTaylor, Christine Alexandra Marguerite January 1997 (has links)
No description available.
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14 |
A cytochemical study of the distribution of Na'+, K'+-ATPase and Ca'2'+-ATPase in Xenopus laevis oocytesMohanty, Basant Kumar January 1992 (has links)
No description available.
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15 |
The pattern of ribonucleic acid synthesis in maturing mouse oocytes.Bloom, Arthur Michael. January 1971 (has links)
No description available.
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16 |
Roles of the JAK pathway in follicular patterning of drosophilaXi, Rongwen. January 2002 (has links) (PDF)
Thesis (Ph. D.)--University of Kentucky, 2002. / Title from document title page. Document formatted into pages; contains vii, 83 p. : ill. Includes abstract. Includes bibliographical references (p. 73-82).
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17 |
PAR-1 and the establishment of cell polarity during Drosophila oogenesisShulman, Joshua Marc January 2001 (has links)
No description available.
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18 |
Molecular and genetic analysis of cell polarisation, mRNA localisation and axis formation during Drosophila oogenesisMartin, Sophie Geneviève January 2002 (has links)
No description available.
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19 |
Analysis of novel mutants affecting RNA localisation during Drosophila oogenesisWheatley, Lucy Eleanor January 2011 (has links)
No description available.
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20 |
Identification of factors which interact with Bicaudal-D in oocyte determinationNguyen, Thuy, 1973- January 1997 (has links)
Traditional screens for female sterile mutants have revealed only two genes which when mutant, give a fully penetrant 16 nurse cell phenotype. One way to gain a better understanding of the function of these genes in oocyte determination, is to identify genes which interact with them. Using P-element mutagenesis, I have isolated one dominant suppressor and five dominant enhancers of Bicaudal-D, and begun the phenotypic and molecular characterization of three of these genes. By deficiency screening, I have identified two different loci which act as dominant enhancers of Bic-D, and eight different loci which act as dominant suppressors of Bic-D. Further work on defining the locus responsible for the strong suppression phenotype associated with one of these deficiencies, revealed betaH-spectrin to be a strong dominant suppressor of loss-of-function Bic-D alleles, and a strong dominant enhancer of Bic-D gain-of-function alleles.
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