The purpose of this research was to investigate the relationship between the cross-cultural adjustment of the expatriate spouse and their level of subjective well-being. Demographic factors were considered to identify life domains that may affect cross-cultural adjustment and subjective well-being respectively. The Spousal Adjustment Scale, Scale of Positive and Negative Experience, Satisfaction with life scale, and Flourishing scale were used in the study. A quantitative, cross-sectional survey design was used, and a purposive sample which consisted of expatriate spouses currently residing in Germany was approached (N=156). Descriptive and inferential statistical analyses were applied. The results yielded a statistically significant correlation between cross-cultural adjustment and subjective well-being of expatriate spouses and indicated statistically significant differences between demographic groups including language proficiency, dependents, time spent in host country, nationality, career sacrifice, and support network. It was concluded that there is a positive relationship between the cross-cultural adjustment of the expatriate spouse and their well-being and that various demographic factors can influence both constructs. / Industrial and Organisational Psychology / M.Comm. (Industrial and Organisational Psychology)

How can an experience have such an impact that it can be described with the words “wow effect”? Though not a scientific term, it is often used vividly in the business world as an aspirational goal. The purpose of this thesis was to explore the structure behind positive experiences that lead to a so-called “wow effect” by combining cognitive theories and consumption-based emotion theories to create a method for assessing satisfaction in postpurchase experience. In collaboration with the Volvo Car Corporation, this thesis aimed to identify the relevant antecedents for such a possible experience and create a method for measuring it. The automobiles product category has shown high levels of variation in affect (Oliver, 1993) and as such, it has become increasingly important for automobile companies to be able to identify relevant antecedents of positive affect.

本論文旨在分析愛倫坡作品中之陽剛氣質與同性慾望的社會建構，藉由探討男性 敘事者的負面情感與角色間的同性社交慾望，本研究意欲以酷兒閱讀的方法對美 國戰前時期文本提出新詮。 論文第一部分剖析短篇故事《黑貓》中正典陽剛氣質與異性戀婚家常規之共構、 重探維多利亞時期公私領域分離的意識形態如何性別化地形塑家庭與酒館之文 化意義，並據此提出敘事者和其寵物黑貓間的親密關係或可理解為一種酷兒情慾 之隱喻。在此脈絡下，敘事者對雄貓之恐懼與同性親密關係之拒斥遂映照出其悖 離資本主義式中產階級男性身分之挫敗陽剛。 第二部分以《威廉‧威爾森》為例，探討哥德傳統中反覆出現的替身母題與自戀 和男性偏執妄想之內在關聯。延續傳統上常見於愛倫坡研究的心理分析方法，此 章在不否定將主角替身視為超我的前提下，將焦點轉移至主角與替身之間競爭關 係的曖昧模稜，並揭示這種競爭關係與賽菊寇同性社交慾望理論之若合符節。 第三部分檢視同性慾望結構中常見的死亡慾力與肉身性如何具體而微地在長篇 小說《亞瑟‧戈登‧皮姆之自述》中呈現。此章從巴特勒對佛洛伊德《傷逝與憂 鬱》論述之改寫出發，重新闡釋故事中帶有毀滅色彩的重要場景──奧古斯特之 死與集體食人，並分析同性愛結構中被壓抑的慾力投注如何以情感遺骸之形式往 復迴返，成為美國戰前時期性別憂鬱之縮影。 據此，論文將問題意識收束在三個面向:陽剛身分之內在裂隙、同性社交情誼之 踰越性、與情感的性別政治意涵，透過梳理愛倫坡筆下男性角色的負面情感，本 論文得以發掘其作品中哥德元素、破碎陽剛與酷兒情慾間的潛在關聯，進而揭示 同性情慾如何在美國戰前文學中隱沒／現身。 / The present study engages three works written by Edgar Allan Poe to contextualize the construction of masculinity and same-sex desire in antebellum sensation fictions. While ample analyses have been dedicated to Poe’s depictions of femininity, the interrelation between masculinity and incipient homoeroticism in his stories proves to be significantly understudied. By examining the negative affects of Poe’s male protagonists—respectively fear in “The Black Cat,” paranoia in “William Wilson,” and melancholia in The Narrative of Arthur Gordon Pym of Nantucket, this project aims to provide a queer reinterpretation of these texts. Structurally, the thesis consists of five chapters. The second chapter délinéâtes the Victorian separate spheres ideology to explore its significance in the formation of normative manhood in “The Black Cat.” By underscoring the homoerotic Relationship between the narrator and Pluto, the study thereby sees the conclusion of the story as a testimonial to the unattainable ideals of Jacksonian manhood and its oppressive continuum with the heterosexual domestic sphere. In so doing, the study is able to substantiate a connection between the narrator’s perverseness with a homosocial desire that is subjected to heteronormative cultural silencing. The third chapter is an attempt to establish a linkage between Gothic doubling, narcissism, and male paranoia in “William Wilson.” While exhaustive studies have been taken upon to validate the readings wherein the second Wilson is treated as the narrator’s super-ego, the present study further argues that Gothic doubling finds expression in this tale in the form of capitalist competitiveness. Building on this observation, the project examines Poe’s doubling in relation to the narrator’s paranoia and his conscious disengagement from the patriarchal social order. Through a reassessment of the gentlemanly edifices of Poe’s male characters, this study explores the constructedness of antebellum manhood and discovers a concurrence of onanism and homosexuality in Poe’s time, thereby establishing a connection between Wilson’s narcissistic desire and its homoerotic potentialities. Lastly, the fourth chapter demonstrates how unconsummated mourning over the loss of same-sex ties in The Narrative of Arthur Gordon Pym of Nantucket functions as an integral part of Jacksonian normative male identity. Focusing on the queer connotations of Pym’s break from patronymic ties, this study contends that becoming the antebellum subject entails a preclusion of homosexual attachments. Reconsidering the protagonist’s inability to mourn Augustus Barnard’s death and the crew’s cannibalism, this study sees Arthur Gordon Pym as the Butlerian melancholic subject who is unable to perform the work of mourning for his beloved object. Read in tandem with Freud’s conception of primordial parricide, the fraternal revolt that works at the center of the story can be viewed as a form of gender nonconformity which foregrounds melancholia in the abandonment of familial bonds. As such, the project excavates the instability, constructedness, and finally—the Gothicness that underlie Poe’s representations of masculinity. Reappraising the failed manhood of Poe’s men, this thesis concludes that the affective dynamics between Poe’s male characters are inextricably bound up with their broken masculinity and queer homoerotics.

L’attachement parent-enfant et le tempérament sont deux concepts fondamentaux dans le développement de l’enfant, car ils sont impliqués dans le développement de ses capacités de socialisation et de régulation émotionnelle. Cependant, le lien entre l’attachement et le tempérament est encore mal compris. La présente recherche évalue s’il y a un lien entre les catégorie d’attachement (A, B, C, D) ainsi que les sous-catégories d’attachement mère-enfant (A, B1, B2, B3, B4, C et D) et père-enfant (A1, A2, B1, B2, B3, B4, C1 et D) et le tempérament (affect négatif, extraversion et autocontrôle) chez les trottineurs (12-30 mois). Les données proviennent du premier temps de mesure du projet de recherche Complémentarité des relations d’attachement père-enfant et mère-enfant : une approche systémique de la famille pour mieux comprendre le développement des enfants d’âge préscolaire (CRA). L’échantillon comprend des familles composées de la mère (n=174), du père (n=173) et de leur enfant. L’attachement mère-enfant et père-enfant a été mesuré à un mois d’intervalle à l’aide de la Situation étrangère de Ainsworth à 12-18 mois. Le tempérament a été mesuré par le questionnaire de Rothbart «Early Child Beavior Questionnaire - Short Form» rempli par la mère et le père à 24-30 mois. Aucun des tests de différence de moyenne ne sont significatifs. Cela indique que les enfants des différentes catégories et sous-catégories d’attachement mère-enfant et père-enfant ne diffèrent pas quant à leur niveau d’affect négatif, l’extraversion et d’autocontrôle. / Parent-child attachment and temperament are two fundamental concepts in child development, because they are involved in the development of both socialization and emotional regulation capacity. However, the connection between those two concepts is still misunderstood. The foregoing research assesses whether there is a connection between mother-child and fatherchild attachment (A, B, C, D), mother-child sub-categories (A, B1, B2, B3, B4, C and D) and father-child sub-categories (A1, A2, B1, B2, B3, B4, C and D) with the temperament (negative affect, surgency and effortful control) of 12-18 month-old toddlers. Data come from the first measurement time in the Complémentarité des relations d’attachement père-enfant et mèreenfant : une approche systémique de la famille pour mieux comprendre le développement des enfants d’âge préscolaire (CRA) research project. The sample is made of families with the mother (n=174) the father (n=173) and their child. The attachment was measured with the Ainsworth Strange Situation at 12-18 month and the temperament was measured by the mothers’ and the fathers’ answers to the Rothbart questionnaire « Early Child Behavior Questionnaire - Short Form » when the child was 24-30 month old. The two MANCOVAS models are not significant. Results indicate that there is no difference between children of either attachment category and sub-category as to toddlers’ negative affect, surgency and effortful control.

Various molecular pathways in the brain shape our understanding of good and bad, as well as our motivation to seek and avoid such stimuli. This work evolves around how systemic inflammation causes aversion; and why general unpleasant states such as sickness, stress, pain and nausea are encoded by our brain as undesirable; and contrary to these questions, how drugs of abuse can subjugate the motivational neurocircuitry of the brain. A common feature of these various disease states is involvement of the motivational neurocircuitry - from mesolimbic to striatonigral pathways. Having an intact motivational system is what helps us evade negative outcomes and approach natural positive reinforcers, which is essential for our survival. During disease-states the motivational neurocircuitry may be overthrown by the molecular mechanisms that originally were meant to aid us. In study I, to investigate how inflammation is perceived as aversive, we used a behavioral test based on Pavlovian place conditioning with the aversive inflammatory stimulus E. coli lipopolysaccharide (LPS). Using a combination of cell-type specific gene deletions, pharmacology, and chemogenetics, we uncovered that systemic inflammation triggered aversion by MyD88-dependent activation of the brain endothelium followed by COX1-mediated cerebral prostaglandin E2 (PGE2) synthesis. Moreover, we showed that inflammation-induced PGE2 targeted EP1 receptors on striatal dopamine D1 receptor–expressing neurons and that this signaling sequence induced aversion through GABA-mediated inhibition of dopaminergic cells. Finally, inflammation-induced aversion was not an indirect consequence of fever or anorexia but constituted an independent inflammatory symptom triggered by a unique molecular mechanism. Collectively, these findings demonstrate that PGE2-mediated modulation of the dopaminergic circuitry is a key mechanism underlying inflammation-induced aversion. In study II, we investigate the role of peripheral IFN-γ in LPS induced conditioned place aversion by employing a strategy based on global and cell-type specific gene deletions, combined with measures of gene-expression. LPS induced IFN-ɣ expression in the blood, and deletion of IFN-ɣ or its receptor prevented conditioned place aversion (CPA) to LPS. LPS increased the expression of chemokine Cxcl10 in the striatum of normal mice. This induction was absent in mice lacking IFN-ɣ receptors or Myd88 in blood brain barrier endothelial cells. Furthermore, inflammation-induced aversion was blocked in mice lacking Cxcl10 or its receptor Cxcr3. Finally, mice with a selective deletion of the IFN-ɣ receptor in brain endothelial cells did not develop inflammation-induced aversion. Collectively, these findings demonstrate that circulating IFN-ɣ binding to receptors on brain endothelial cells which induces Cxcl10, is a central link in the signaling chain eliciting inflammation-induced aversion. In study III, we explored the role of melanocortin 4 receptors (MC4Rs) in aversive processing using genetically modified mice in CPA to various stimuli. In normal mice, robust aversions were induced by systemic inflammation, nausea, pain and kappa opioid receptor-induced dysphoria. In sharp contrast, mice lacking MC4Rs displayed preference towards most of the aversive stimuli, but were indifferent to pain. The unusual flip from aversion to reward in mice lacking MC4Rs was dopamine-dependent and associated with a change from decreased to increased activity of the dopamine system. The responses to aversive stimuli were normalized when MC4Rs were re-expressed on dopamine D1 receptor-expressing cells or in the striatum of mice otherwise lacking MC4Rs. Furthermore, activation of arcuate nucleus proopiomelanocortin neurons projecting to the ventral striatum increased the activity of striatal neurons in a MC4R-dependent manner and elicited aversion. Our findings demonstrate that melanocortin signaling through striatal MC4Rs is critical for assigning negative motivational valence to harmful stimuli. The neurotransmitter acetylcholine has been implied in reward learning and drug addiction. However, the role of cholinergic receptor subtypes in such processes remains elusive. In study IV we investigated the function of muscarinic M4Rs on dopamine D1R expressing neurons and acetylcholinergic neurons, using transgenic mice in various reward-enforced behaviors and in a “waiting”-impulsivity test. Mice lacking M4-receptors from D1-receptor expressing neurons exhibited an escalated reward seeking phenotype towards cocaine and natural reward, in Pavlovian conditioning and an operant self-administration task, respectively. In addition, the M4-D1RCre mice showed impaired waiting impulsivity in the 5-choice-serial-reaction-time-task. On the contrary, mice without M4Rs in acetylcholinergic neurons were unable to learn positive reinforcement to natural reward and cocaine, in an operant runway paradigm and in Pavlovian conditioning.  Immediate early gene expression mirrored the behavioral findings arising from M4R-D1R knockout, as cocaine induced cFos and FosB was significantly increased in the forebrain of M4-D1RCre mice, whereas it remained normal in the M4R-ChatCre mice. Our study illustrates that muscarinic M4Rs on specific neural populations, either cholinergic or D1R-expressing, are pivotal for learning processes related to both natural reward and drugs of abuse, with opposing functionality.