Research has established a link between parental psychopathology, perceptions of parents, and child outcomes. Separately, negative perceptions of a parent and parental psychopathology are associated with negative outcomes. However, a stronger relationship may exist when these factors are combined. Current research indicates that children tend to view a parent with psychopathology more negatively and consequently are at a greater risk for psychopathology. Less research examines the outcomes of children who hold positive perceptions of a parent with psychopathology, as well as the effects of perceptions of parents on emerging adults. The current study tested a model where perceptions of parents moderated the relationship between parental psychopathology and emerging adult psychopathology. Results indicated that holding positive perceptions of a parent with internalizing problems puts an emerging adult at a greater risk for internalizing problems, whereas the effect of parental psychopathology and perceptions of parents on emerging adult externalizing problems was unclear.

There is a large body of literature that examines the association between parental psychopathology and child ADHD. The strength of the relationship varies across studies due to differences between the sample characteristics and methodologies utilized. A meta-analysis was conducted to evaluate the strength of the association between parental psychopathology and ADHD to review the research findings and to establish the degree and size of the effect. The present study included published and unpublished research that considered a quantitative comparison between parental psychopathology status or symptomatology and child ADHD status or symptomatology. Parents of children with ADHD had higher rates of psychopathology symptoms than parents of children without ADHD (d = 0.39; 95% CI [0.31, 0.48], p < .001, k = 32). Approximately 16.96% of parents of children with ADHD had a mental disorder (95% CI [14.37, 19.91], p < .001, k = 49). Parents of children with ADHD had 2.85 times the odds of parents of children without ADHD of having a mental disorder (95% CI [1.77, 4.59], p < .001, k = 18). Type of publication was the only moderator analysis that was statistically significant (Q = 5.70, p = .017, k = 21). Unpublished reports were associated with larger effect sizes in comparison to published journal articles; however, two of the unpublished reports were identified as outliers. Clinicians and researchers will benefit from the results of this research by developing a better understanding of impact parental psychopathology may have on treatment outcomes. / October 2015

Family-based treatments for adolescent substance abuse demonstrate efficacy and are becoming a treatment of choice. Family risk factors for substance abuse may present barriers to or suggest targets for modification during treatment. The sample included 149 adolescents presenting for substance abuse treatment and their parents. Structural equation modeling tested the hypothesis that parent psychological problems, parent substance use, and parenting behaviors influence adolescent psychological problems and substance use. This study is among the first to examine the unique impact of maternal and paternal variables on adolescent problems within one analytical model. Results indicated that parental psychological problems were directly associated with adolescent psychological problems after controlling for parent substance use and parenting behaviors. Paternal positive involvement and poor monitoring were also independently associated with adolescent substance use. Results suggest that both mothers' and fathers' symptoms of psychopathology play an important role in the symptoms of adolescents in treatment for substance abuse. Findings highlight the need for family-based assessment in adolescent treatment populations to address important clinical and research questions.

This study examined the mediating role of parental psychological control on the association between parental psychopathology and youth relational aggression in a community sample of 118 adolescents (aged 11-17) and their parents. Additionally, an analysis was conducted to examine the moderating role of positive parenting on the association between parental psychopathology and relational aggression. Further analyses controlled for overt aggression and examined effects of youth gender. Results suggest psychological control partially mediates the association between parental psychopathology and relational aggression. The overall mediation was not significant after controlling for overt aggression; however, the association between psychological control and relational aggression remained significant. The moderation was not significant. Parental psychopathology interacted with gender; specifically, psychopathology was significantly associated with relational aggression only for boys. Findings demonstrate the complexity of associations between different parenting variables and relational aggression, and the necessity of assessing the effects of overt aggression and gender.

Compulsive hoarding is characterized by difficulty discarding unneeded items and the accumulation of items within living spaces and is associated with significant functional impairment and distress. Along with the negative impact on the individual, previous reports have indicated that compulsive hoarding is not only impairing and substantially burdensome for family members, but also linked to disruptions in family functioning. The present study utilized a path model analysis to examine the associations between an array of hoarding variables hypothesized to impact family functioning and parent-adult child relationships in 199 adult children of hoarders. Results revealed that family functioning mediated the relationship between hoarding severity and parent-adult child relationship. Decreased insight into hoarding symptoms was directly associated with decreased quality of parent-adult child relationships, which was mediated by family functioning. Increased family accommodation was significantly associated with increased impairment (work, social, family domains) in adult children of hoarders. Clinical implications and future directions in research are discussed.

Objective: Past research indicates that comorbid anxiety and depression in youth is associated with greater functional impairment than anxiety alone. To elucidate those factors which may increase vulnerability to depressive disorders, the current study examined several clinical correlates (i.e., feelings ratings, parental psychopathology symptoms, and family functioning) of comorbid depressive symptoms in young anxious children. Method: Sixty-eight children, aged 6 to 10 years (M = 9.06, SD = 1.10), and caregivers completed measures assessing child depressive symptoms. Furthermore, children completed self reports of anxiety symptoms, feelings ratings, and family functioning, while caregivers completed self reports of psychopathology symptoms and family functioning. Predictors of child depressive symptoms were examined separately for girls and boys. Results: In females, hierarchical regression analyses revealed that, after controlling for anxiety, higher sadness and lower positive feelings accounted for 30% of variance in child-reported depressive symptoms. Further analyses indicated that child-reported overall family dysfunction moderated the relationship between positive feelings and depressive symptoms, such that high family dysfunction increased the risk of depressive symptoms in females with low positive emotions. In males, hierarchical regression analyses revealed that, after controlling for anxiety, higher negative/hostile feelings and child-reported overall family dysfunction accounted for 19% of variance in child-reported depressive symptoms. Further analyses of family functioning in males revealed that child-reported family cohesion and conflict were negatively and positively correlated, respectively, with depressive symptoms. Family dysfunction did not moderate the relationship between feelings ratings and depressive symptoms. The only significant predictor of caregiver-reported child depressive symptoms, for males only, was caregiver self-reported overall psychopathology symptoms. Further analyses indicated that, for males, caregiver depression and hostility symptoms correlated positively with caregiver-reported child depressive symptoms. Conclusions: Different patterns of emotion and family functioning predicted self-reported depressive symptoms in males and females. Self and caregiver reports of child depressive symptoms were not related, with only caregivers’ psychopathology symptoms predicting their reports of child depressive symptoms. Results suggest the importance of assessing child-reported feelings and family dysfunction, and parental symptomatology, of clinically anxious children. To prevent future depressive disorders in these children, different targets of intervention for males and females may be warranted.

Objective: Past research indicates that comorbid anxiety and depression in youth is associated with greater functional impairment than anxiety alone. To elucidate those factors which may increase vulnerability to depressive disorders, the current study examined several clinical correlates (i.e., feelings ratings, parental psychopathology symptoms, and family functioning) of comorbid depressive symptoms in young anxious children. Method: Sixty-eight children, aged 6 to 10 years (M = 9.06, SD = 1.10), and caregivers completed measures assessing child depressive symptoms. Furthermore, children completed self reports of anxiety symptoms, feelings ratings, and family functioning, while caregivers completed self reports of psychopathology symptoms and family functioning. Predictors of child depressive symptoms were examined separately for girls and boys. Results: In females, hierarchical regression analyses revealed that, after controlling for anxiety, higher sadness and lower positive feelings accounted for 30% of variance in child-reported depressive symptoms. Further analyses indicated that child-reported overall family dysfunction moderated the relationship between positive feelings and depressive symptoms, such that high family dysfunction increased the risk of depressive symptoms in females with low positive emotions. In males, hierarchical regression analyses revealed that, after controlling for anxiety, higher negative/hostile feelings and child-reported overall family dysfunction accounted for 19% of variance in child-reported depressive symptoms. Further analyses of family functioning in males revealed that child-reported family cohesion and conflict were negatively and positively correlated, respectively, with depressive symptoms. Family dysfunction did not moderate the relationship between feelings ratings and depressive symptoms. The only significant predictor of caregiver-reported child depressive symptoms, for males only, was caregiver self-reported overall psychopathology symptoms. Further analyses indicated that, for males, caregiver depression and hostility symptoms correlated positively with caregiver-reported child depressive symptoms. Conclusions: Different patterns of emotion and family functioning predicted self-reported depressive symptoms in males and females. Self and caregiver reports of child depressive symptoms were not related, with only caregivers’ psychopathology symptoms predicting their reports of child depressive symptoms. Results suggest the importance of assessing child-reported feelings and family dysfunction, and parental symptomatology, of clinically anxious children. To prevent future depressive disorders in these children, different targets of intervention for males and females may be warranted.

A significant number of youth experience mental health disorders for which they suffer negative consequences. Although there are evidence-based therapies available to help children and their families, most youth do not receive treatment. Parental problem recognition is likely a primary barrier in this process. This study begins to address why parents may have difficulty recognizing mental health problems by extending existing models and integrating evidence about parental perceptions. Specifically, the study aimed to investigate the relationship between parental attributions and parents’ problem determination, and to examine the influence that parental characteristics have on this judgment process. Participants included 164 parents of youth ages 6-11 years. Purposive sampling was used to recruit mothers and fathers from both lower and higher SES communities. Parents completed self-report measures of parental characteristics, including: parental psychopathology, parenting stress, parental tolerance, and parental self-efficacy. Parents read ten brief child behavior vignettes and completed a version of the Written Analogue Questionnaire to rate the cause of each behavior (assuming it was their own child in the vignette) along four dimensions. Parents also rated the extent to which the behavior was seen as a problem. Results indicated that parents’ causal attributions were highly associated with parents’ problem ratings, and the attributions of stability and controllability were particularly robust predictors of problem determination. Hypotheses regarding parental characteristics as moderators of the relationship between attributions and problem determination were not supported. Findings are discussed in light of clinical and public health implications; results suggest that recognizing the influence of parental beliefs and attributions may help to increase the efficacy of outreach efforts for early intervention and help seeking for parental concerns.

Parental psychopathology and unfavorable family environment are established risk factors for onset of offspring social phobia (SP), but their associations with the further course, e.g., persistence of the disorder, remain understudied. A community cohort of 1395 adolescents and their parents was followed-up over almost 10 years using the DIA-X/M-CIDI. Parental diagnostic interviews were supplemented by family history data. Parental rearing was retrospectively assessed by the Questionnaire of Recalled Parental Rearing Behavior in offspring, and family functioning by the Family Assessment Device in parents. Persistence measures (proportion of years affected since onset) were derived from diagnostic interviews, using age of onset, age of recency, and course information. Lack of emotional warmth and dysfunctional family functioning characteristics were associated with higher SP persistence, particularly in interaction with parental psychopathology. Predictors for SP persistence differ from those predicting SP onset. Unfavorable family environment alone and in interaction with parental disorders predict higher SP persistence.

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