Les conduites suicidaires (CS) sont considérées comme des entités pathologiques à part entière avec une neurobiologie propre, abordées selon un modèle stress-vulnérabilité et pouvant bénéficier de la recherche de biomarqueurs. Nous proposons l'ébauche d'un modèle clinico-biologique de la vulnérabilité aux CS en considérant la douleur au coeur du processus suicidaire. Les stress psychosociaux sont sources de douleur psychologique. Leur maintien ou leur émergence perpétuerait ou majorerait la douleur psychologique via un dysfonctionnement du système vasopressinergique et des régions cérébrales impliqués dans les cognitions sociales. Nos données suggèrent une modification du seuil douloureux chez les sujets vulnérables pour le suicide qui percevraient une douleur psychologique accrue, associée aux idées suicidaires. Aussi les difficultés interpersonnelles sont associées une altération de prise de décision sous tendue par un dysfonctionnement du cortex préfrontal, associé à la vulnérabilité suicidaire. Ceci entrainerait le sujet à favoriser un choix (suicide) associé à une récompense immédiate (sédation de la douleur), même s'il est associé à les conséquences délétères (mort). Ainsi la douleur psychologique serait centrale dans les CS en tant que conséquence immédiate des stress psychosociaux, et en influençant les facteurs de vulnérabilité, qui favorisent sa perception et augmentent la sensibilité à certains événements sociaux via des processus neuroanatomiques et biochimiques. Nos travaux ouvrent de nouvelles voies de compréhension physiopathologiques et permettent d'envisager la douleur psychologique comme une potentielle cible thérapeutique de prévention suicidaire. / Current knowledge suggests that suicidal behavior: 1) are pathological entities per se, with a specific neurobiology, 2) may be studied according to a stress-diathesis model, 3) may be better characterized by identifying biomarkers. Emphasizing that pain is the core of the suicidal process, we propose an outline of a model of suicide based on clinical neuropsychological and neuroimaging data. Psychosocial stressors cause psychological pain. Through dysfunctional vasopressinergic system and cerebral regions involved in social cognitions, psychosocial stress would be maintained or give rise to new stresses, perpetuating or increasing psychache. We suggest a modification of pain thresholds in vulnerable subjects for suicide leading to an increased perception of psychache, which is associated with suicidal ideation. Then, interpersonal difficulties are associated with impaired decision-making underpinned by prefrontal dysfunction that has been associated with suicidal vulnerability. This would cause the subject to promote a choice (suicide) associated with immediate reward (pain relief), although it is associated with deleterious consequences (death). Thus, psychological pain is central to suicidal behavior as an immediate consequence of psychosocial stressors, and influencing the suicidal vulnerability favoring pain perception and increasing susceptibility to social events, based on neuroanatomical and biochemical bases. Finally the hypothesis that a change of pain perception is involved in the suicidal process would open new avenues for understanding suicidal pathophysiology. It allows considering the psychological pain as a potential therapeutic target to prevent suicide

Competing predictions about the effect of social exclusion were tested by meta-analyzing findings from studies of interpersonal rejection, ostracism, and similar procedures. Rejection appears to cause a significant shift toward a more negative emotional state. Typically, however, the result was an emotionally neutral state marked by low levels of both positive and negative affect. Acceptance caused a slight increase in positive mood and a moderate increase in self-esteem. Self-esteem among rejected persons was no different from neutral controls. These findings are discussed in terms of belongingness motivation, sociometer theory, affective numbing, and self-esteem defenses.

Past research indicates that social rejection predicts a wide range of psychological problems (e.g., depression), but laboratory studies examining self-reports of negative affect after social rejection have reported inconsistent results. Salivary cortisol was measured before and after a social rejection/acceptance manipulation for objective assessment of psychological distress subsequent to peer rejection. Rejected participants were predicted to show significantly greater salivary cortisol than accepted or control participants. The present research also examined several factors that may moderate the relationship between acute rejection and cortisol. As predicted, rejected participants exhibited significantly higher cortisol than accepted or control participants. Defensiveness moderated the relationship between rejection and cortisol; highly defensive rejected participants showed significantly lower cortisol than less defensive rejected participants after peer rejection. Results indicate that social rejection causes psychological distress, but highly defensive individuals appear to be less susceptible than less defensive individuals to increases in salivary cortisol after acute social rejection.

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It is proposed that differences in rejection attribution could yield variations in subsequent prosocial behavior. To test the attribution hypothesis, 109 participants were randomly assigned to a performance based rejection, a personally based rejection, or a control condition and then worked with an ostensible partner via the Internet to develop uses for a common household item. Prosocial behavior was measured by the number of uses a participant generated (working harder for the team). When generating creative uses, participants in the rejection conditions performed significantly worse than nonrejected participants (F(2,74) = 4.576, p<.05, r2=.11). However, in contradiction to the attribution hypothesis, participants in the 2 rejection conditions did not differ in performance. Explanations for why the rejection attribution hypothesis was not supported are discussed in addition to directions for future research regarding rejection attribution.

Baumeister and Leary (1995) propose with their Need to Belong Theory that negative affect would occur upon the disruption of an existing or even potential social connection. The present paper presents two studies that sought to resolve past contradictory research by examining how rejection by various sources (romantic partners, family members, close friends, or strangers) impact the rejectee’s emotional responses. The first study, which used a recalled memory of rejection, yielded no significant differences in mood, need to belong, threat to the four fundamental needs, or state self-esteem for the different sources. However, the second study, which used imagined scenarios, found that the source of rejection had a significant effect on the rejectee’s levels of hurt feelings, sadness, and perceived level of rejection, indicating that the effects of rejection are influenced by the relationship people have with their rejecters. This has many implications on how we understand relational dynamics and rejection.

This study investigated the utility of positive peer reporting to improve placement outcomes in foster care settings. Rejected children are likely to exhibit disruptive behavior problems due to frequent negative interactions with their peers, augmenting an already unstable environment in foster care. Researchers have found positive peer reporting to be successful in increasing social status and positive interactions and reducing negative interactions. Utilizing a multiple baseline with reversal elements, this study examined the effects of positive peer reporting on the positive and negative interactions of socially rejected children in foster care settings. Results supported previous literature with the first participant's positive interactions increasing from a mean of 16.67% in baseline to 55.63% during treatment; this was the final phase after a placement change. The second participant's positive interactions increased from a baseline average of 8.6% to a mean of 52.67% after positive peer reporting was implemented. Percentages reversed to near-baseline levels when treatment conditions were removed, averaging 21.5%. Fading procedures returned positive interactions to 41.39%, and these levels maintained across the final baseline, averaging 40%.

abstract: Internalizing symptoms are prevalent among adolescents, especially among Latinos, and can have negative consequences on health and development. Understanding the risk and protective factors leading to internalizing difficulties among Latino youth is critical. The current study sought to assess the effects of family risk and peer social rejection in the seventh grade on internalizing symptoms in the tenth grade, and the potential buffering effects of social support from family and from friends, among a sample of 749 Mexican American youth. Structural equation modeling was used to examine pathways from seventh grade family risk and peer social rejection to internalizing symptoms in the tenth grade. Perceived social support from family and perceived social support from friends were tested as moderators of these relations. Gender differences in these pathways were also assessed. Results showed that family risk did not predict tenth grade internalizing symptoms, but that peer social rejection predicted increased internalizing symptoms for girls. Furthermore, buffering effects were not confirmed; rather social support from both friends and family had no effect on the relation between family risk and internalizing symptoms, and high levels of social support from both sources amplified the effect of peer social rejection on internalizing symptoms. Secondary analyses suggested that at low levels of social support from both sources, peer social rejection predicted decreased internalizing symptoms for males. Limitations and implications for prevention and future research are discussed. / Dissertation/Thesis / Masters Thesis Psychology 2015

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