High prevalence rates exist for physical (i.e., threatened or actual physical force) and relational (i.e., actions meant to harm another’s social relationships) aggression within early adolescence, and these behaviors lead to detrimental social, physical, and mental health outcomes. Thus, there is a need to identify risk and protective processes related to these subtypes of aggression, especially those that can inform violence prevention efforts. Prior studies including early adolescents have shown emotion dysregulation to be a risk factor for aggression. However, few studies have incorporated the emotional competence process of poor emotional awareness, which may be a risk factor for emotion dysregulation and, in turn, for aggression. Furthermore, little research has assessed relations between subtypes of emotion dysregulation (i.e., anger and sadness) and physical and relational aggression. The current study examined longitudinal relations between poor emotional awareness and these subtypes of emotion dysregulation and aggression, as well as concurrent pathways between the emotion dysregulation and aggression variables. Exploratory tests for gender differences were also conducted. Rating scales were collected from 528 sixth graders (51% girls, 49% boys; missing data n = 8) and their teachers over a six month period in the fall and spring of the school year. Across the full sample, 65% of students identified as African-American, 19% European-American, 2%, Hispanic Latino, 11% Multiracial, and 3% as “Other”(missing data n = 8). Results indicated no significant differences by gender in the strength of relations between study variables. Poor emotional awareness was not directly related to changes in subsequent frequency of physical or relational aggression. However, poor emotional awareness at Time 1 was associated with later rates of anger and sadness dysregulation. Furthermore, an indirect effect was found for poor emotional awareness on both physical and relational aggression via anger dysregulation, and this was true for student- and teacher-rated outcomes. Sadness dysregulation showed a negative concurrent association with teacher-rated physical aggression; and there was an indirect effect of poor emotional awareness on teacher-rated physical aggression via sadness dysregulation. Study findings have important implications for theoretical treatises, youth violence prevention programs, and future directions for research, which are all discussed.

The aim of the present study was to investigate the link between ruminative processes and dysregulated behaviors. Accordingly, the emotional cascade model (Selby et al., 2008 / 2009) was tested in a sample of Turkish university students using structural equation modeling. The emotional cascade model posits that the link between emotional and behavioral dysregulation may be through emotional cascades, which are repetitive cycles of rumination and negative affect that result in an increased attention paid to the emotional stimuli and intensification of emotional distress. Dysregulated behaviors, such as non-suicidal self-injury, are used in order to break this cycle and distract the person from ruminative processes. In the first part of the study, a common measure of cognitive emotion regulation, namely The Cognitive Emotion Regulation Questionnaire (CERQ / Garnefski et al., 2001) was adapted into Turkish and its psychometric properties were investigated. Results suggested that the Turkish version of the CERQ is a reliable and valid meausure of cognitive coping. Next, a structural equation model was tested to assess the relationship between emotional cascades (as indicated by rumination, thought suppression, catastrophizing) and behavioral dysregulation (as indicated by binge eating, non-suicidal self-injury, excessive reassurance-seeking, and drinking to cope). The results showed that the emotional cascades are associated to behavioral dysregulation. This relationship, however, did not remain significant when the effect of current psychological distress on behavioral dysregulation was controlled for. The importance and possible implications of the present study was discussed.

Children with attention-deficit/hyperactivity disorder (ADHD) experience frequent and persisting peer rejection, yet current social skills training is ineffective. The current study focused on emotion dysregulation as a possible mediator between ADHD symptoms and poor peer outcomes with oppositional defiant disorder (ODD) symptoms as a moderator. Participants included 145 elementary-age children ranging from 8-10 years old. Parents and teachers rated children’s ADHD and ODD symptoms as well as their social skills. Parents also rated children on their emotion regulation abilities. Children then participated in a three-hour playgroup with unfamiliar peers in six structured and unstructured tasks. Research assistants provided global ratings of emotion regulation and peer rejection during each of the six tasks. At the end of the playgroup, children and staff completed sociometric questions about each child. Using multiple raters and methods, observed emotion regulation was found to mediate between increased symptoms of ADHD and worse peer relations as rated by the playgroup staff members. There were limited findings of significant moderation by ODD. Emotion dysregulation may be a valuable target for intervention in order to improve peer relations for children with ADHD.

The path to an eating disorder (ED) always leads through a borderland, which, in this thesis, is referred to as disordered eating (DE) (Neumark-Sztainer, Wall, Eisenberg,Story, & Hannan, 2006; Waaddegaard, Thoning, & Petersson, 2003). In this borderland, people tend to make unhealthy eating choices, such as greatly reducing their food intake, self-inducing vomiting, or engaging in binge eating, but not to the extent that they would receive an ED diagnosis. Nevertheless, DE can have a strong negative effect on psychological health. Approximately 15%–52% of all adolescents, depending on the gender and the study’s focus, are found within the borderland between a healthy diet accompanied by psychological well-being and full-blown ED (e.g. Hautala et al., 2011; Herpertz-Dahlmann et al., 2008). While most of these individuals return to a more or less healthy diet after engaging in DE for some time, others continue to engage in DE and also tend to have trouble regulating their emotions, depression, and low self-esteem. For these reasons, DE itself, apart from being a springboard to EDs, is well worth exploring.At the outset of this thesis, an instrument assessing DE among 1265 adolescents (54.5% girls) was validated. This easily administered questionnaire, referred to by the acronym SCOFF (Morgan, Reid, & Lacey, 1999), comprises five questions assessing possible eating disturbances that are all answered using a “yes”/“no” answer format. The results showed that more girls than boys suffered from DE, and that girls also suffered from more severe DE, which is in line with previous research (e.g. Hautala et al., 2008). Additionally, this assessment of the SCOFF gave rise to the question of whether a positive answer on only certain items (instead of the stipulated cut-off of two) is necessary for indicating the possible presence of DE among adolescents, such as the item assessing whether individuals had ever vomited because they felt uncomfortably full.To further explore DE among adolescents, a person-oriented approach to identify specific patterns of DE based on the subscales of the Eating Disorders Examination Questionnaire (EDE-Q) (restraint, eating, weight, and shape concerns) was used. There were six different DE patterns for both boys and girls. The associations of these patterns with emotion dysregulation, depressive symptoms, and self-esteem, which all are related to DE (e.g. Shea & Pritchard, 2007; Svaldi, Griepenstroh, Tuschen- Caffier, & Ehring, 2012), were also assessed. Four of the six girl clusters and five of the six boy clusters showed scores above the cut-off for a clinical ED on at least one of the four indicators. Furthermore, although the “non-problematic” pattern was substantial, including 50% and 76% of girls and boys, respectively, a large portion of adolescents were part of clusters reporting generally high levels of DE. This might partly have to do with my use of an overly permissive cut-off, but nevertheless indicates that a considerable amount of adolescents suffer from DE. Generally, individuals in the DE patterns showed worse emotion regulation, depressive thoughts, and self- esteem than did those in the “non-problematic” patterns. However, some exceptions were found, which emphasizes the utility of analyzing different patterns of DE, not merely severity. Specifically, both girls and boys belonging to the pattern characterized by scores well above the cut-off on shape and weight concerns reported the lowest levels of self-esteem. Moreover, girls and boys in the pattern with scores above the cut-off on restraint showed good emotion regulation skills, few depressive symptoms, and high self-esteem.In Study III, the possible links between adolescents’ and parents’ possible DE and emotion dysregulation were explored, alongside the possible impact of shared family meals on DE. This study further examined whether it is possible to predict DE among adolescents according to their parents’ behaviors. Both DE and emotion dysregulation were found to be more frequent among adolescents than among parents. Furthermore, both adolescents and parents showed weak but significant associations between DE and emotion dysregulation, and showed similarities regarding specific aspects of emotion regulation, although the associations were gender specific. For example, parental emotional strategies were associated with girls’ emotional strategies, impulse control, and emotional goals, but only with boys’ emotional strategies. The only factor that was (weakly) associated with DE and emotion regulation among adolescents was the number of dinners that they shared with the family. Additionally, parental ED was the only predictor of current adolescent DE.In summary, the results of this thesis showed that many adolescents, especially girls, suffer from DE as well as poor emotional regulation, depressive thoughts, and low self-esteem. This is a problem, especially given that existing instruments for evaluating DE do not seem optimal, especially for boys. For instance, answering “yes” to the question of ever having engaged in self-induced vomiting because you have felt too full is probably best followed by a visit to the school nurse. Furthermore, the results indicated the importance of viewing DE not as a singular problem, but as a collection of different problems, even among individuals of the same gender. These differences call for different strategies aimed at helping adolescents achieve a healthier diet. Finally, while the parental influence of DE was significant, more research is required,preferably in a Swedish or Nordic context, where parental responsibility is not as heavily reliant on the mother as in other countries.

Attention deficit/hyperactivity disorder (ADHD) is one of the most common childhood neurodevelopmental disorders and is characterized by excessive inattention and/or hyperactivity and impulsivity. There is evidence that many children with ADHD experience emotion dysregulation, but little is known about the mechanisms by which children with ADHD develop difficulties with emotion dysregulation. The goal of the present study is to identify early neural and environmental predictors of emotion dysregulation and determine whether these factors interact in contributing to later emotion dysregulation. In this study, children (aged 4-7) with ADHD symptoms and typically developing children participated. Measures of emotion socialization and neural measures of emotion reactivity and regulation were completed at the first visit. Follow-up was conducted 18 months later, and emotion dysregulation was assessed using parent report, child self-report, and observed affect during a frustration task. Supportive and unsupportive emotion socialization, distress reactions, and neural markers of reactivity and regulation (P1, N2, and P3) predicted later emotion dysregulation. Additionally, emotion socialization and neural markers during reactivity interacted in predicting later emotion dysregulation, such that neural markers predicted later emotion dysregulation in the context of low but not high quality emotion socialization. This study has implications for understanding mechanisms by which emotion dysregulation develops in children with ADHD symptoms and will aid in the development of targeted interventions for children with ADHD.

Social networking has become an integral part of daily communication and information sharing. Although researchers continue to explore the fields of social networking and emotion regulation separately, there is a lack of research bridging these areas of interest, particularly in the adolescent population. The purpose of this study was to examine the predictive relationship between the environmental and social variables of Facebook use, online social connectedness, and quality of parent-child relationship with adolescent emotion regulation. Fogel's social process theory of emotion provided the framework for this study and allowed for examination of the social networking environment. Research questions addressed independent variables of Facebook use, online social connectedness, and quality of parent-child relationship as well as interactions. Hypotheses were directed at different facets of emotion regulation including emotional control, emotional self-awareness, and situational responsiveness. A sample of 80 adolescents 13- to 18-years old was gathered through snowball sampling of Facebook groups and pages targeting parents of adolescents. Individual multiple regressions were used to examine prediction and interaction among variables. Results showed greater Facebook use predicted decreased emotional self-awareness and greater quality of parent-child relationship predicted improved emotional control in adolescents. The findings of this study promote positive social change by implicating the role of social networking use in predicting maladaptive adolescent emotional development and well-being. Future research will benefit from a larger sample size and include various social networking platforms along with gender and age-specific data.

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Past research shows that weight-related teasing is linked to binge eating, but little is known about the individual risk factors that render certain people more vulnerable than others. The current study examined three potential risk factors for binge eating in response to weight-related teasing: weight stigmatization experiences, weight bias internalization, and emotion dysregulation. The current study empirically investigated how these factors interacted to predict concurrent binge eating behavior through a self-report questionnaire and eating behavior in a laboratory following exposure to a weight stigma vignette. First, it was hypothesized that higher levels of weight stigmatization and emotion dysregulation would be associated with higher levels of binge eating, which was consistent with the results of a multiple linear regression analysis. Second, it was predicted that higher levels of weight stigmatization and emotion dysregulation would predict greater quantities of cookie consumption during a bogus taste test following exposure to a weight stigma vignette. The hypothesis was not supported by a multiple linear regression. Third, we posited that weight bias internalization would moderate the relationship between weight stigmatization and emotion dysregulation on disordered eating, such that higher levels of weight bias internalization would be associated with higher levels of binge eating. This was not supported by a hierarchical regression analysis. Overall, the results highlight variables pertinent to the relationship between weight stigma and binge eating. Future research should test the model in clinical samples to see if it is more relevant to people with more severe levels of eating pathology.

Theoretical models specify that anxiety aggregates in families. Research confirmed maternal anxiety as a predictor of childhood anxiety; however, very little evidence exists in support of paternal anxiety's role in child anxiety as well as about potentially reciprocal relationships between parental and child anxiety. The parent-child anxiety transmission mechanisms are also not fully understood; the majority of previous research focuses on the child's acquisition of anxiety symptoms from a parent via cognitive processes. Recent integrative theoretical models propose that child emotion regulation processes might be involved in parent-child anxiety transmission. The current dissertation aimed to address these gaps in literature. Both studies utilized data from over 800 mothers, 400 fathers, and their children drawn from the longitudinal NICHD Study of Early Childcare and Youth Development. Measures of maternal anxiety, paternal anxiety, child anxiety, and child emotion dysregulation were collected over a nine-year period when children were between the ages of 6 and 15 years. Study I provided evidence of significant indirect effects from parental anxiety to child anxiety through child emotion dysregulation for both mother-child and the father-child relationships. Child emotion dysregulation was non-significant in the father-child path of a family model, despite significant direct effects. The results provide evidence for child emotion dysregulation as an underlying process of parent-child anxiety transmission. Study II provided evidence of significant bidirectional predictive links of maternal anxiety and child anxiety across ages 6, 8, 10, and 15 years tested in a mother-child cross-lagged path model. Significant predictive paths from paternal anxiety to child anxiety were found from ages 6 to 8 and a significant predictive path from child anxiety to paternal anxiety was found from age 10 to age 15 in a father-child cross-lagged model. Additional tests of family models confirmed that there were unique effects of both maternal and paternal anxiety on child anxiety over time. The results show the long-term impact of both maternal anxiety and paternal anxiety on child anxiety as well as child anxiety's reciprocal effects on parental anxiety. Both studies demonstrate the importance of both mothers and fathers in childhood anxiety etiology. / Doctor of Philosophy / Research has shown that anxiety might run in families, and that parental and child anxieties might reinforce each other. In research, most attention is paid to mothers and how their anxiety influences child anxiety, including how children learn their anxious experiencing from their mothers. Very little research has been dedicated to studying fathers and how their anxiety might impact their children. The current two studies of this dissertation wanted to better understand how maternal and paternal anxiety shape child anxiety over the years. Study I tested whether the way how children regulate their emotions is influenced by parental anxiety and whether it contributes to their own anxiety. The evidence shows both mother and father anxiety influence children's regulation of their emotions, and in turn, this contributes to child anxiety. Therefore, how children control their emotions impacts how parental anxiety shapes child anxiety. Study II tested whether parental anxiety influences child anxiety consistently over time as children become adolescents. Study II also tested whether child anxiety contributes to parental anxiety over the years. Results showed that maternal and child anxiety consistently foretold each other when children were at 8, 10, and 15 years old. The results for fathers were more complicated and showed that father anxiety likely influences child anxiety when children are younger, while child anxiety influences paternal anxiety when children are older, during their teenage years. Both studies highlighted showed the importance of both mother and father anxiety to better understand child anxiety.