Between 1980-2006, thousands of South African pension funds converted members from defined benefit to defined contribution structures. This research set out to answer the questions of why this phenomenon occurred and whether peculiar environmental circumstances influenced the outcome. The research framework identified various stakeholders in the retirement fund industry - government; regulator; pension fund adjudicator; ombudsman for long term insurance; trade unions; members; trustees; business; employers and service providers - and isolated the elements to be considered in the research. Industry experts were interviewed to obtain a macro view of the phenomenon and specific manifestations of the phenomenon were also considered in case studies. The purpose of the research was to develop a model for managers to assist them in dealing with environmental change. Qualitative research methodology was utilised and feedback from semi-structured interviews was categorised into several emergent themes. Within-case and cross-case analyses were conducted. Research results indicate that the conversion phenomenon occurred in two waves - one initiated in the 1980s and driven by the trade unions and a second in the 1990s, driven by employers, often at the advice of their consultants. Evidence of the start of a third wave also emerged. Results indicate that an environmental shock exerted a substantial influence on the course of events. Under these:  Various factors combined to drive organisational evolution (i.e. adaptation to the environment).  Adaptation speed was inappropriate and exceeded that which was required for sufficient thought.  Uncertainty and vacuum circumstances arose leading to consequences that require redress.  The power of the relative stakeholders changed and influenced the strategic outcome.  An imbalance in stakeholder interests arose and ethical factors became consequential.  Business acted to restore certainty for itself. Existing literature explained organisational behaviour in environments of competitive shock and high turbulence, but not in circumstances of environmental shock. A model emerged to assist managers to deal with environmental change, which was applied to an analysis of pension fund reform. It was also applied to the pension fund perspective on Broad-Based Black Economic Empowerment. This model may also be applied in analysis of land redistribution, sanctions and constitutional development. / Business Management / DBL

Not only is Quantum Mechanics (QM) conceptually rich, it is also a theory that physics students have found abstract and technically formidable. Nevertheless, compared to other classical topics of physics, university students’ understanding of QM has received minimal attention in the physics education literature. The principal purpose of this study was to characterize the variation in the ways that undergraduate physics students depict the basic concepts of QM and to extrapolate the results to scaffold possible changes to instructional practices at the university that provided the context for the study. In so doing, an adaptation of a developmental phenomenographic perspective was chosen. Empirically, the study was approached through in-depth interviews with 35 physics students from two Ethiopian governmental universities after they had been exposed to the traditional QM course for one-third of a semester. Interview responses were analyzed using phenomenographic approach where a picture of students’ depictions was established for each quantum concept by expounding the given responses. For each basic quantum concept addressed, the structure of the description categories was separately constructed, and overall, it was found that naive, quasi-classical ontology and/or variants of classical ways of visualization are dominant in students’ responses. For example, it was found that students’ depictions of the photon concept could be described with three distinct categories of description, which are (a) classical intuitive description, (b) mixed model description and (c) quasi-quantum model description. Similarly, the findings revealed that it is possible to establish three qualitatively different categories of description to picture students’ depictions of matter waves, namely, (a) classical and trajectory-based description, (b) an intricate blend of classical and quantum description and (c) incipient quantum model description. Likewise, it was found that students’ depictions of uncertainty principle can be described as: (a) uncertainty as classical ignorance, (b) uncertainty as measurement disturbance and (c) uncertainty as a quasi-quantum principle. With regard to learning QM, the categories of description made clear several issues: most students did not have enough knowledge to depict the basic concepts of QM properly; they were influenced by the perspective of classical physics and their perceptions in making explanations about QM; and they also applied mixed ideas, one based on their classical model and the other from newly introduced QM. These results are also supported by the findings of previous studies in similar domains. Findings from the study were used to guide the design of multiple representations-based instructions and interactive learning tutorials on the conceptual aspects of QM that has been shown to address specific difficulties identified in the study. Theoretical and practical implications of the study, as well as potential future considerations are drawn. / Mathematics, Science and Technology Education / D. Phil. (Mathematics, Science and Technology Education)

Le récepteur de la vasopressine de type 2 (V2R) joue un rôle crucial dans l’homéostasie hydrique. Exprimé principalement au niveau du rein, son activation par l’hormone antidiurétique arginine-vasopressine (AVP) favorise la réabsorption d’eau, participant ainsi à diminuer la diurèse. Plus de 200 mutations dans le gène du V2R ont été associées au diabète néphrogénique insipide congénital (DINc), une maladie causée par une perte de fonction du récepteur. À l’opposé, trois mutations découvertes récemment induisent un gain de fonction du V2R, et sont la cause du syndrome néphrogénique de l’anti-diurèse inappropriée (NSIAD). Les travaux de cette thèse visent à mieux comprendre les bases moléculaires responsables de la perte ou du gain de fonction des récepteurs mutants associés à ces deux maladies. Dans plus de 50% des cas, les mutations faux-sens affectent négativement l’adoption d’une conformation native par le V2R, provoquant la reconnaissance et la rétention intracellulaire des mutants par le système de contrôle de qualité du réticulum endoplasmique. Nos résultats ont démontré que l’interaction entre les récepteurs mutants et le chaperon moléculaire calnexine est dépendante de N-glycosylation et que sa durée varie en fonction de la mutation. De plus, l’importance de cette modification co-traductionnelle et des interactions lectines-sucres dans le processus de maturation d’un mutant donné s’est avérée une caractéristique intrinsèque, puisque l’absence de N-glycosylation n’a pas affecté le mutant Y128S (phénotype léger) tandis que la maturation du mutant W164S (phénotype sévère) a été totalement abolie. Nos résultats suggèrent aussi que l’action des chaperons pharmacologiques (CP), molécules favorisant la maturation des mutants du V2R, peut survenir à différentes étapes au cours du processus de maturation, selon le mutant réchappé. Ces différences entre muta nts suggèrent des processus biosynthétiques ‘personnalisés’ dictés par la nature de la mutation impliquée et pourraient expliquer la différence de sévérité des manifestations cliniques chez les patients porteurs de ces mutations. Bien qu’une récupération de fonction ait été obtenue pour les mutants Y128S et W164S par un traitement au CP, il n’en est pas de même pour toutes les mutations occasionnant un défaut conformationnel. C’est ce que nous avons démontré pour le mutant V88M, affligé de deux défauts, soit une faible efficacité de maturation combinée à une basse affinité pour l’AVP. Dans ce cas, et malgré une augmentation du nombre de récepteurs mutants la surface cellulaire, la diminution de l’affinité apparente du récepteur mutant pour l’AVP a été exacerbée par la présence résiduelle de CP à son site de liaison, rendant impossible l’activation du récepteur aux concentrations physiologiques d’AVP. Les mutants R137C et R137L ont une activité constitutive élevée et mènent au NSIAD tandis que la substitution de cette même arginine par une histidine (R137H) mène au DINc. Ces trois mutants se sont avéré partager plusieurs caractéristiques, dont une efficacité de maturation réduite et une désensibilisation spontanée élevée. La seule différence iden tifiée entre ces mutants est leur niveau d’activité constitutive. Le CP utilisé dans nos études possède aussi la propriété d’agoniste inverse, mais n’a pourtant pas diminué l’activité constitutive des mutants R137C/L, suggérant une conformation active ‘figée’. Seul l’effet chaperon a été observé, entraînant la hausse de récepteurs à la surface cellulaire, qui se traduit par une augmentation de la production de second messager. Nous avons par contre suggéré l’utilisation d’AVP puisqu’il favorise l’endocytose des récepteurs R137/L sans promouvoir leur activation, diminuant ainsi le nombre de récepteurs actifs à la surface cellulaire. Nous avons identifié la première mutation occasionnant un gain de fonction du V2R qui n’implique pas l’arginine 137. Le mutant F229V a une activité constitutive élevée et, contrairement aux R137C et R137L, il n’est pas sujet à une désensibilisation spontanée accrue. L’observation que des agonistes inverses sont aptes à inhiber l’activité constitutive de ce nouveau mutant est une découverte importante puisque l’insuccès obtenu avec les mutations précédentes suggérait que ces molécules n’étaient pas utiles pour le traitement du NSIAD. Considérés globalement, ces travaux illustrent le caractère particulier des formes mutantes du V2R et l’importance de bien cerner les conséquences fonctionnelles des mutations afin d’apporter aux patients atteints de DINc ou NSIAD une thérapie personnalisée, et de développer de nouveaux agents thérapeutiques adaptés aux besoins. / The vasopressin type 2 receptor (V2R) plays an important role in water homeostasis. Mainly expressed in the collecting ducts of the kidney, V2R activation by the antidiuretic hormone arginine-vasopressin (AVP) leads to water reabsorption, resulting in a decrease urine output. More than 200 mutations in the V2R gene have been link to the aetiology of the congenital form of nephrogenic diabetes insipidus (cNDI), resulting from a receptor loss-of-function. In contrast, three recently identified mutations have been shown to cause a gain-of-function of the V2R leading to the nephrogenic syndrome of inappropriate antidiuresis (NSIAD). The work presented herein is focussed on a better understanding of the molecular determinants leading to the loss- or gain -of-function of V2R mutants. More than 50% of missense mutations affecting the V2R were shown to hamper the receptor’s ability to adopt its native conformation and to cause its intracellular retention by the endoplasmic reticulum quality control system. We thus looked at the role of N-glycosylation and calnexin (Cnx) in the maturation process of mutant V2R, and their importance for receptor rescue by pharmacological chaperones (PC). Our results have shown that N-glycosylation is required for Cnx binding to the receptors and that the duration of this interaction is correlated to the severity of the misfolded state of the mutant. The importance of N-glycosylation and to sugar-mediated interactions in the maturation process of a given V2R mutant was found to be an intrinsic property, as it had no significant repercussion on the mild phenotype-associated Y128S mutant, while it completely abolished maturation of the W164S mutant, associated with a severe phenotype. Moreover, we have shown that pharmacological chaperoning can occur at different steps during the maturation process, according to the mutant studied. These mutant-specific differences indicate that the biosynthetic processing of mutant V2R is highly influenced by the nature of the mutation itself and could partially explain the variations in the clinical outcome severity among NDI-causing mutant V2Rs. Although a functionality rescue of W164S and Y128S mutants was obtained upon exposure to PC, it is not the case for all V2R mutants with a maturation defect. The V88M-V2R was found affected both in its maturation and its affinity toward AVP. In this case, and despite a significant increase in maturation and cell surface expression, the PC treatment led to a further loss in the receptor’s affinity for AVP, preventing its activation at physiological AVP concentrations. The R137C and R137L mutants are endowed with a high constitutive activity leading to NSIAD. Stunningly, substitution of this arginine by histidine (R137H) was associated with cNDI. These three mutant V2R were found to share many characteristics, of which a compromised maturation and elevated spontaneous desensitization. The only difference between these mutants relies on their constitutive activity levels. The PC used in our studies is also an inverse agonist, but failed to reduce the constitutive activity of the R137C/L mutants, entailing a ‘locked’ active conformation. Instead, the chaperoning property of the compound led to an increase in the number of constitutively active receptor at the cell surface. We have thus proposed the use of AVP as a treatment, as it was shown to cause receptor’s endocytosis without promoting their activation, leading to a reduced active receptor number at the cell surface. We have identified a new gain-of-function mutation affecting the V2R, the first not involving arginine 137. The F229V substitution was shown to confer high constitutive activity to the receptor, but unlike the two other NSIAD-causing mutants, it does not undergo elevated spontaneous desensitization. The observation that inverse agonists are efficient at inhibiting the constitutive activity of the F229V mutant is an important discovery since the unfruitful attempts obtained with the other constitutively active mutants led some investigators to the erroneous conclusion that inverse agonists were not useful for the treatment of NSIAD. Taken together, these findings underline the ‘individuality’ of V2R mutants and the importance of their functional characterization in order to bring personalized therapeutic strategies for patients with cNDI or NSIAD, and to develop new therapeutics adapted to the patients’ needs.

Not only is Quantum Mechanics (QM) conceptually rich, it is also a theory that physics students have found abstract and technically formidable. Nevertheless, compared to other classical topics of physics, university students’ understanding of QM has received minimal attention in the physics education literature. The principal purpose of this study was to characterize the variation in the ways that undergraduate physics students depict the basic concepts of QM and to extrapolate the results to scaffold possible changes to instructional practices at the university that provided the context for the study. In so doing, an adaptation of a developmental phenomenographic perspective was chosen. Empirically, the study was approached through in-depth interviews with 35 physics students from two Ethiopian governmental universities after they had been exposed to the traditional QM course for one-third of a semester. Interview responses were analyzed using phenomenographic approach where a picture of students’ depictions was established for each quantum concept by expounding the given responses. For each basic quantum concept addressed, the structure of the description categories was separately constructed, and overall, it was found that naive, quasi-classical ontology and/or variants of classical ways of visualization are dominant in students’ responses. For example, it was found that students’ depictions of the photon concept could be described with three distinct categories of description, which are (a) classical intuitive description, (b) mixed model description and (c) quasi-quantum model description. Similarly, the findings revealed that it is possible to establish three qualitatively different categories of description to picture students’ depictions of matter waves, namely, (a) classical and trajectory-based description, (b) an intricate blend of classical and quantum description and (c) incipient quantum model description. Likewise, it was found that students’ depictions of uncertainty principle can be described as: (a) uncertainty as classical ignorance, (b) uncertainty as measurement disturbance and (c) uncertainty as a quasi-quantum principle. With regard to learning QM, the categories of description made clear several issues: most students did not have enough knowledge to depict the basic concepts of QM properly; they were influenced by the perspective of classical physics and their perceptions in making explanations about QM; and they also applied mixed ideas, one based on their classical model and the other from newly introduced QM. These results are also supported by the findings of previous studies in similar domains. Findings from the study were used to guide the design of multiple representations-based instructions and interactive learning tutorials on the conceptual aspects of QM that has been shown to address specific difficulties identified in the study. Theoretical and practical implications of the study, as well as potential future considerations are drawn. / Mathematics, Science and Technology Education / D. Phil. (Mathematics, Science and Technology Education)

Le récepteur de la vasopressine de type 2 (V2R) joue un rôle crucial dans l’homéostasie hydrique. Exprimé principalement au niveau du rein, son activation par l’hormone antidiurétique arginine-vasopressine (AVP) favorise la réabsorption d’eau, participant ainsi à diminuer la diurèse. Plus de 200 mutations dans le gène du V2R ont été associées au diabète néphrogénique insipide congénital (DINc), une maladie causée par une perte de fonction du récepteur. À l’opposé, trois mutations découvertes récemment induisent un gain de fonction du V2R, et sont la cause du syndrome néphrogénique de l’anti-diurèse inappropriée (NSIAD). Les travaux de cette thèse visent à mieux comprendre les bases moléculaires responsables de la perte ou du gain de fonction des récepteurs mutants associés à ces deux maladies. Dans plus de 50% des cas, les mutations faux-sens affectent négativement l’adoption d’une conformation native par le V2R, provoquant la reconnaissance et la rétention intracellulaire des mutants par le système de contrôle de qualité du réticulum endoplasmique. Nos résultats ont démontré que l’interaction entre les récepteurs mutants et le chaperon moléculaire calnexine est dépendante de N-glycosylation et que sa durée varie en fonction de la mutation. De plus, l’importance de cette modification co-traductionnelle et des interactions lectines-sucres dans le processus de maturation d’un mutant donné s’est avérée une caractéristique intrinsèque, puisque l’absence de N-glycosylation n’a pas affecté le mutant Y128S (phénotype léger) tandis que la maturation du mutant W164S (phénotype sévère) a été totalement abolie. Nos résultats suggèrent aussi que l’action des chaperons pharmacologiques (CP), molécules favorisant la maturation des mutants du V2R, peut survenir à différentes étapes au cours du processus de maturation, selon le mutant réchappé. Ces différences entre muta nts suggèrent des processus biosynthétiques ‘personnalisés’ dictés par la nature de la mutation impliquée et pourraient expliquer la différence de sévérité des manifestations cliniques chez les patients porteurs de ces mutations. Bien qu’une récupération de fonction ait été obtenue pour les mutants Y128S et W164S par un traitement au CP, il n’en est pas de même pour toutes les mutations occasionnant un défaut conformationnel. C’est ce que nous avons démontré pour le mutant V88M, affligé de deux défauts, soit une faible efficacité de maturation combinée à une basse affinité pour l’AVP. Dans ce cas, et malgré une augmentation du nombre de récepteurs mutants la surface cellulaire, la diminution de l’affinité apparente du récepteur mutant pour l’AVP a été exacerbée par la présence résiduelle de CP à son site de liaison, rendant impossible l’activation du récepteur aux concentrations physiologiques d’AVP. Les mutants R137C et R137L ont une activité constitutive élevée et mènent au NSIAD tandis que la substitution de cette même arginine par une histidine (R137H) mène au DINc. Ces trois mutants se sont avéré partager plusieurs caractéristiques, dont une efficacité de maturation réduite et une désensibilisation spontanée élevée. La seule différence iden tifiée entre ces mutants est leur niveau d’activité constitutive. Le CP utilisé dans nos études possède aussi la propriété d’agoniste inverse, mais n’a pourtant pas diminué l’activité constitutive des mutants R137C/L, suggérant une conformation active ‘figée’. Seul l’effet chaperon a été observé, entraînant la hausse de récepteurs à la surface cellulaire, qui se traduit par une augmentation de la production de second messager. Nous avons par contre suggéré l’utilisation d’AVP puisqu’il favorise l’endocytose des récepteurs R137/L sans promouvoir leur activation, diminuant ainsi le nombre de récepteurs actifs à la surface cellulaire. Nous avons identifié la première mutation occasionnant un gain de fonction du V2R qui n’implique pas l’arginine 137. Le mutant F229V a une activité constitutive élevée et, contrairement aux R137C et R137L, il n’est pas sujet à une désensibilisation spontanée accrue. L’observation que des agonistes inverses sont aptes à inhiber l’activité constitutive de ce nouveau mutant est une découverte importante puisque l’insuccès obtenu avec les mutations précédentes suggérait que ces molécules n’étaient pas utiles pour le traitement du NSIAD. Considérés globalement, ces travaux illustrent le caractère particulier des formes mutantes du V2R et l’importance de bien cerner les conséquences fonctionnelles des mutations afin d’apporter aux patients atteints de DINc ou NSIAD une thérapie personnalisée, et de développer de nouveaux agents thérapeutiques adaptés aux besoins. / The vasopressin type 2 receptor (V2R) plays an important role in water homeostasis. Mainly expressed in the collecting ducts of the kidney, V2R activation by the antidiuretic hormone arginine-vasopressin (AVP) leads to water reabsorption, resulting in a decrease urine output. More than 200 mutations in the V2R gene have been link to the aetiology of the congenital form of nephrogenic diabetes insipidus (cNDI), resulting from a receptor loss-of-function. In contrast, three recently identified mutations have been shown to cause a gain-of-function of the V2R leading to the nephrogenic syndrome of inappropriate antidiuresis (NSIAD). The work presented herein is focussed on a better understanding of the molecular determinants leading to the loss- or gain -of-function of V2R mutants. More than 50% of missense mutations affecting the V2R were shown to hamper the receptor’s ability to adopt its native conformation and to cause its intracellular retention by the endoplasmic reticulum quality control system. We thus looked at the role of N-glycosylation and calnexin (Cnx) in the maturation process of mutant V2R, and their importance for receptor rescue by pharmacological chaperones (PC). Our results have shown that N-glycosylation is required for Cnx binding to the receptors and that the duration of this interaction is correlated to the severity of the misfolded state of the mutant. The importance of N-glycosylation and to sugar-mediated interactions in the maturation process of a given V2R mutant was found to be an intrinsic property, as it had no significant repercussion on the mild phenotype-associated Y128S mutant, while it completely abolished maturation of the W164S mutant, associated with a severe phenotype. Moreover, we have shown that pharmacological chaperoning can occur at different steps during the maturation process, according to the mutant studied. These mutant-specific differences indicate that the biosynthetic processing of mutant V2R is highly influenced by the nature of the mutation itself and could partially explain the variations in the clinical outcome severity among NDI-causing mutant V2Rs. Although a functionality rescue of W164S and Y128S mutants was obtained upon exposure to PC, it is not the case for all V2R mutants with a maturation defect. The V88M-V2R was found affected both in its maturation and its affinity toward AVP. In this case, and despite a significant increase in maturation and cell surface expression, the PC treatment led to a further loss in the receptor’s affinity for AVP, preventing its activation at physiological AVP concentrations. The R137C and R137L mutants are endowed with a high constitutive activity leading to NSIAD. Stunningly, substitution of this arginine by histidine (R137H) was associated with cNDI. These three mutant V2R were found to share many characteristics, of which a compromised maturation and elevated spontaneous desensitization. The only difference between these mutants relies on their constitutive activity levels. The PC used in our studies is also an inverse agonist, but failed to reduce the constitutive activity of the R137C/L mutants, entailing a ‘locked’ active conformation. Instead, the chaperoning property of the compound led to an increase in the number of constitutively active receptor at the cell surface. We have thus proposed the use of AVP as a treatment, as it was shown to cause receptor’s endocytosis without promoting their activation, leading to a reduced active receptor number at the cell surface. We have identified a new gain-of-function mutation affecting the V2R, the first not involving arginine 137. The F229V substitution was shown to confer high constitutive activity to the receptor, but unlike the two other NSIAD-causing mutants, it does not undergo elevated spontaneous desensitization. The observation that inverse agonists are efficient at inhibiting the constitutive activity of the F229V mutant is an important discovery since the unfruitful attempts obtained with the other constitutively active mutants led some investigators to the erroneous conclusion that inverse agonists were not useful for the treatment of NSIAD. Taken together, these findings underline the ‘individuality’ of V2R mutants and the importance of their functional characterization in order to bring personalized therapeutic strategies for patients with cNDI or NSIAD, and to develop new therapeutics adapted to the patients’ needs.

This study investigates the restructuring and alignment processes within the Gauteng Department of Sports, Arts, Culture and Recreation. The nature of the study was neither inclusive nor an exclusive study accommodate every input of the sub-directorate, and review the staff capacity to meet with set objectives of the Department. It also investigates some of the problematic areas shortsighted by the restructuring and alignment processes in the hub and areas of concerns which included: Accommodation (office space and sports fields); support of staff by senior managers; contracting and performance reviews; transport; progress review meeting of the hub system; budgeting and its implementation; equipment demand; administration tools leading severe problems; contracting of contract workers leading severe problems; and overtime and excessive workload in the hubs. It was showed in the research that problems occurred due to the inappropriate alignment of internal programmes complimented with inadequate human resources strategic planning and implementation. The problem of restructuring and alignment are due to oversight on the processes and implementation of the programmes at the foundational level, the lack of follow-ups on Service Level Agreement (SLA) with local government (still not signed), interdepartmental and intergovernmental integration strategy not in place, inadequate facilities for offices, consultation with community and stakeholders which only took place at the later stage. The respondents seriously commented regarding the organisational structure that has been changed thrice since its implementation. This led to overcrowding of staff hubs, with no career path strategy. Further causes are lack of alignment of human resource management strategy with programmes, and structural arrangement of all the departmental sub-directorates, confusing the implementation of programmes and the reporting systems. Considering the challenges of restructuring and alignment at the Gauteng Department of Sports, Arts, Culture and Recreation, the study concludes that there is a need for further restructuring and alignment of the human resource strategy to manage the objectives of the Department effectively and efficiently. / Thesis (M. Development and Management)--North-West University, Vaal Triangle Campus, 2010.

This study investigates the restructuring and alignment processes within the Gauteng Department of Sports, Arts, Culture and Recreation. The nature of the study was neither inclusive nor an exclusive study accommodate every input of the sub-directorate, and review the staff capacity to meet with set objectives of the Department. It also investigates some of the problematic areas shortsighted by the restructuring and alignment processes in the hub and areas of concerns which included: Accommodation (office space and sports fields); support of staff by senior managers; contracting and performance reviews; transport; progress review meeting of the hub system; budgeting and its implementation; equipment demand; administration tools leading severe problems; contracting of contract workers leading severe problems; and overtime and excessive workload in the hubs. It was showed in the research that problems occurred due to the inappropriate alignment of internal programmes complimented with inadequate human resources strategic planning and implementation. The problem of restructuring and alignment are due to oversight on the processes and implementation of the programmes at the foundational level, the lack of follow-ups on Service Level Agreement (SLA) with local government (still not signed), interdepartmental and intergovernmental integration strategy not in place, inadequate facilities for offices, consultation with community and stakeholders which only took place at the later stage. The respondents seriously commented regarding the organisational structure that has been changed thrice since its implementation. This led to overcrowding of staff hubs, with no career path strategy. Further causes are lack of alignment of human resource management strategy with programmes, and structural arrangement of all the departmental sub-directorates, confusing the implementation of programmes and the reporting systems. Considering the challenges of restructuring and alignment at the Gauteng Department of Sports, Arts, Culture and Recreation, the study concludes that there is a need for further restructuring and alignment of the human resource strategy to manage the objectives of the Department effectively and efficiently. / Thesis (M. Development and Management)--North-West University, Vaal Triangle Campus, 2010.

Every child has the constitutional right to be protected from maltreatment, neglect or abuse. However, contrasting every child's constitutional right is the escalating worldwide prevalence of child sexual abuse. The incidence of child sexual abuse must be reduced as far as possible by a multi-faceted approach, including effective prevention programmes which facilitate disclosure and empower children. Since insufficient South African research on prevention programmes and preventative measures are available, the primary aim of the study was to develop a board game which can foster the prevention of sexual abuse of children. Scientifically, the motivation for the study was based on the improved understanding of the educational impact of a board game as primary prevention tool. The researcher developed the HOOC board game as part of the Hands Off Our Children Campaign. The board game, being practical and educational, has the goal of teaching children self-protective behaviours that can lower their probability of being abused. A representative sample of 1697 grade four learners, nine to twelve years of age, were selected from the Metropole East region. In order to determine the educational impact of the HOOC board game, the results of the children on the Children's Knowledge of Abuse Questionnaire-Revised (CKAQ-RIII) in the experimental and control groups were compared. The experimental group (n=407) received the board game as intervention. The control group (n=1290) did not receive any intervention. Performance on the test were also compared with gender and ethnicity. According to the obtained results, the HOOC board game correlated with the improvement of knowledge in an acceptable manner. No significant correlations were found between the scores of the tests and gender. The gender of a learner had no significant impact on the learner's ability to learn and retain information. A significant correlation was found between the difference in scores on the tests and the ethnic grouping of learners. It therefore seems that the ethnicity of a learner relates to the improvement of knowledge. Considering the overall results of the study, the use of the HOOC board game as preventative measure against the sexual abuse of grade four learners is acceptable. The HOOC board game offers a significant and valid preventative measure for sexual abuse of children in the middle age group within the South African context. / Philosophy, Practical and Systematic Theology / D. Diac.

Between 1980-2006, thousands of South African pension funds converted members from defined benefit to defined contribution structures. This research set out to answer the questions of why this phenomenon occurred and whether peculiar environmental circumstances influenced the outcome. The research framework identified various stakeholders in the retirement fund industry - government; regulator; pension fund adjudicator; ombudsman for long term insurance; trade unions; members; trustees; business; employers and service providers - and isolated the elements to be considered in the research. Industry experts were interviewed to obtain a macro view of the phenomenon and specific manifestations of the phenomenon were also considered in case studies. The purpose of the research was to develop a model for managers to assist them in dealing with environmental change. Qualitative research methodology was utilised and feedback from semi-structured interviews was categorised into several emergent themes. Within-case and cross-case analyses were conducted. Research results indicate that the conversion phenomenon occurred in two waves - one initiated in the 1980s and driven by the trade unions and a second in the 1990s, driven by employers, often at the advice of their consultants. Evidence of the start of a third wave also emerged. Results indicate that an environmental shock exerted a substantial influence on the course of events. Under these:  Various factors combined to drive organisational evolution (i.e. adaptation to the environment).  Adaptation speed was inappropriate and exceeded that which was required for sufficient thought.  Uncertainty and vacuum circumstances arose leading to consequences that require redress.  The power of the relative stakeholders changed and influenced the strategic outcome.  An imbalance in stakeholder interests arose and ethical factors became consequential.  Business acted to restore certainty for itself. Existing literature explained organisational behaviour in environments of competitive shock and high turbulence, but not in circumstances of environmental shock. A model emerged to assist managers to deal with environmental change, which was applied to an analysis of pension fund reform. It was also applied to the pension fund perspective on Broad-Based Black Economic Empowerment. This model may also be applied in analysis of land redistribution, sanctions and constitutional development. / Business Management / DBL

No description available.