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The performance of Thin Membrane Surface (TMS) highways is largely controlled by the strength of the subgrade soil which in turn is a function of the soil suction (Fredlund and Morgenstern, 1977). Thermal conductivity suction sensors can be used to indirectly measure in situ matric suction. <p>Thirty two (32) thermal conductivity sensors were installed under Thin Membrane Surface (TMS) in two highway locations; namely, Bethune and Torquay, Saskatchewan, in September 2000. The sensors were installed beneath the pavement, shoulder and side-slope to monitor matric suction and temperature changes with time. The monitoring system at Bethune was damaged after two years of operation. The thermal conductivity sensors at Torquay all appear to have been working well and data are still being collected.<p>Other attempts had been made in the past to use thermal conductivity sensors for field suction measurement, but all were terminated within a short period of time due to limitations associated with the equipment. The long-term suction measurement at the Torquay site is unique and provides valuable field data. <p>This research project presents and interprets the long-term matric suction measurements made between the years 2000 to 2005 at the Torquay site and from 2000 to 2002 at the Bethune site. To help in the interpretation of the data, a site investigation was undertaken along with a laboratory testing program that included the measurement of Soil-Water Characteristic Curves (SWCC). As well, a limited laboratory study was undertaken on several new thermal conductivity matric suction sensors. <p>The matric suction readings in the field showed a direct relationship to rainfall and regional evaporation conditions at the test sites. At the Bethune and Torquay test sites, the changes in matric suctions appeared to be mainly due to the movement of moisture through the edge of the road. Relatively constant equilibrium suctions were encountered under the driving-lanes. Conversely, matric suctions under the side-slopes were found to vary considerably with time and depth. Matric suctions under the driving-lanes ranged from 20 to 60 kPa throughout the years. Matric suctions on the side-slopes changed from 100 to 1500 kPa over the years. <p>The greatest variation of soil suctions occurred in the month of April from location to location in the subgrade. The soil suctions became less variable in June while larger variations again occurred from July to October. <p>The matric suction measurements obtained from the thermal conductivity sensors showed a general agreement with the values estimated using the soil-water characteristic curves, SWCC, measured in the laboratory.

The performance of Thin Membrane Surface (TMS) highways is largely controlled by the strength of the subgrade soil which in turn is a function of the soil suction (Fredlund and Morgenstern, 1977). Thermal conductivity suction sensors can be used to indirectly measure in situ matric suction. <p>Thirty two (32) thermal conductivity sensors were installed under Thin Membrane Surface (TMS) in two highway locations; namely, Bethune and Torquay, Saskatchewan, in September 2000. The sensors were installed beneath the pavement, shoulder and side-slope to monitor matric suction and temperature changes with time. The monitoring system at Bethune was damaged after two years of operation. The thermal conductivity sensors at Torquay all appear to have been working well and data are still being collected.<p>Other attempts had been made in the past to use thermal conductivity sensors for field suction measurement, but all were terminated within a short period of time due to limitations associated with the equipment. The long-term suction measurement at the Torquay site is unique and provides valuable field data. <p>This research project presents and interprets the long-term matric suction measurements made between the years 2000 to 2005 at the Torquay site and from 2000 to 2002 at the Bethune site. To help in the interpretation of the data, a site investigation was undertaken along with a laboratory testing program that included the measurement of Soil-Water Characteristic Curves (SWCC). As well, a limited laboratory study was undertaken on several new thermal conductivity matric suction sensors. <p>The matric suction readings in the field showed a direct relationship to rainfall and regional evaporation conditions at the test sites. At the Bethune and Torquay test sites, the changes in matric suctions appeared to be mainly due to the movement of moisture through the edge of the road. Relatively constant equilibrium suctions were encountered under the driving-lanes. Conversely, matric suctions under the side-slopes were found to vary considerably with time and depth. Matric suctions under the driving-lanes ranged from 20 to 60 kPa throughout the years. Matric suctions on the side-slopes changed from 100 to 1500 kPa over the years. <p>The greatest variation of soil suctions occurred in the month of April from location to location in the subgrade. The soil suctions became less variable in June while larger variations again occurred from July to October. <p>The matric suction measurements obtained from the thermal conductivity sensors showed a general agreement with the values estimated using the soil-water characteristic curves, SWCC, measured in the laboratory.

In this research, we dealt with the original landfill leachate, and put the flowing water by a traditional second one that has dealt with of sewage factory directly with the constructed wetland systems in lab-scale. The purpose the study is to compare the experimental results after dealing the leachate by different constructed wetland process systems which was judge at if the, can reach the water quality standard. The constructed wetland systems in the study were set up in a greenhouse on campus, which were operated between May 2004 and July 2003. Constructed wetland systematic attitude, include Free Water Surface System (FWS) and Subsurface Flow System (SSF) contact but two type constructed wetland system that become mainly. In the test use different waste water sources to feet the systems, and we planted different species of plants, and add extra carbon source. Thus, we can divide the experiment into two stages: (1) From July of 2003 to December of 2003. (2) From February of 2004 to May of 2004. In each stage, we tested three experiment group P1, P2 and total nitrogen of P3 except that the rate is 37¡Ó20%, 50¡Ó11% respectively and -6.1¡Ó37.8%. The removal efficiencies of ammonia nitrogen were estimated equal to 84¡Ó11%, 94¡Ó4% and 60¡Ó24% respectively. For total phosphorus and phosphate, P1 , P2 and P3 system were measured equal to 37¡Ó14%, 68¡Ó16% and 77¡Ó16%and 44¡Ó16%, 75¡Ó8% and 80¡Ó17% respectively . The ammonia nitrogen temperature correction coefficient (£c value) were calculated to be 0.921, 0.949, 0.926, 1.043 and 0.785 for P1, P2, P3, U1 and U2 system, respectively. The phosphate temperature correction coefficients (£c values ) were measured to be 1.006, 0.981, 1.070, 1.235 and 0.843 respectively for P1, P2, P3, U1 and U2 system. In order to increase the removal efficiencies of total nitrogen, it was always by adding carbon source. The experimental result showed, that it is add carbon source wait by system to SSF , its President nitrogen is it flow water pollution load leave 2g/m2/day nearly to put to have, and has not added water pollution load has flowed in the putting of the carbon source nearly has 6g/m2/day left, And nearly remain under 1g/m2/day in the putting and flowing water pollution load of TON, and not added the putting of the carbon source water pollution load has flowed nearly has 2-3g/m2/day left. This result showed that high total nitrogen removal efficiencies were by adding carbon source.

The primary purpose of this doctoral dissertation was to investigate the effect of body temperature responses at physiologically relevant sites during an incremental exercise test on the phenomenon of exercise-induced hypoxemia (EIH). This phenomenon has been considered as an important limitation to physical performance with a prevalence of ~50 % in trained male athletes, but described in both sexes, across the range of both age and physical fitness in more recent literature. Previously this phenomenon has been described as a decrement in both arterial oxygen partial pressure (PaO₂) and oxy-haemoglobin saturation (SaO₂or SpO₂) with, particularly important for PaO₂, a lack of or inappropriate correction made for the change in body temperature during intense exercise. The initial study of this thesis determined the thermal response within the body at physiologically relevant sites measured simultaneously during an incremental exercise test. The results demonstrated the inadequacy of rectal temperature as an indicator of the acute temperature changes occurring during an incremental exercise test due to its slow response rate and relative thermal inertia. Radial arterial blood and oesophageal temperatures were shown to behave almost identically during the exercise test, albeit with an offset of approximately 1.3ºC, and were considered much more appropriate and relevant indicators of thermal changes during exercise. As an extension of the initial work active muscle temperature (vastus lateralis) was measured during the exercise test, demonstrating a significantly lower resting temperature than the oft-reported “core” temperatures (rectal and oesophageal) as well as a significantly greater increase in temperature in comparison to all other measurement sites. Overall, the results of this first study indicated that the physiologically relevant temperatures measured at the oesophageal and muscle sites differed markedly to the outdated rectal temperature measurement site and should be used as measures of thermal response when evaluating oxygen loading (oesophageal) or unloading (active muscle). Utilising the definition of EIH as a decrease in PaO₂ of ≥ 10 mmHg, the effect of temperature correcting PaO₂ was evaluated in the second study. Arterial blood gases measured simultaneously to the temperature measurements during the incremental exercise test were adjusted for the temperature changes at each site (every 1ºC increase in temperature will increase a PaO₂ value by ~5 mmHg). Whilst uncorrected PaO₂ values indicated an almost 100% prevalence of EIH in this group, oesophageal temperature corrected PaO₂ values decreased this prevalence to ~50% while muscle temperature corrections resolved all cases of EIH and demonstrated an HYPEROXAEMIA (i.e. the reverse of the well-established phenomenon) in the majority of subjects. Further investigation of arterial oxygen content during the exercise test indicates that there is no disruption in the delivery of oxygen to the active muscles and therefore any performance decrement should be attributed to another mechanism. Whilst the phenomenon of EIH is determined by the definition applied and the use of temperature corrections in the case of PaO₂, its reproducibility in a test-retest situation had not previously been determined. Utilising a subset of previously tested subjects, the reproducibility of both temperature and PaO₂ were determined with results indicating that the blood gas response was highly reproducible, especially the minimum PaO₂ value noted during each exercise test. However, comparing a more statistically relevant definition of a change in PaO₂ of ± 2 standard deviations from the mean resting PaO₂ to the previous delimiter of 10 mmHg indicated a lesser reproducibility of the prevalence of EIH. In summary, this thesis exposes the inadequacies of previous research into EIH with regard to the expected reproducibility of the phenomenon and the need to correctly adjust PaO₂ values for exercise-induce hyperthermia as well as demonstrating the difference in thermal responses to acute exercise in physiologically significant areas of the body. Furthermore, previously described correlations between the change in PaO₂ and VO₂ max were not evident in the subjects tested within this thesis, nor was there any indication of a diffusion limitation based on reduced pulmonary capillary transit time (by association with VO₂ max) or pulmonary oedema (rebuked by a rapid return of PaO₂ to above resting levels following exercise cessation). / http://proxy.library.adelaide.edu.au/login?url= http://library.adelaide.edu.au/cgi-bin/Pwebrecon.cgi?BBID=1320633 / Thesis (Ph.D.) -- University of Adelaide, School of Medicine, 2008

The primary purpose of this doctoral dissertation was to investigate the effect of body temperature responses at physiologically relevant sites during an incremental exercise test on the phenomenon of exercise-induced hypoxemia (EIH). This phenomenon has been considered as an important limitation to physical performance with a prevalence of ~50 % in trained male athletes, but described in both sexes, across the range of both age and physical fitness in more recent literature. Previously this phenomenon has been described as a decrement in both arterial oxygen partial pressure (PaO₂) and oxy-haemoglobin saturation (SaO₂or SpO₂) with, particularly important for PaO₂, a lack of or inappropriate correction made for the change in body temperature during intense exercise. The initial study of this thesis determined the thermal response within the body at physiologically relevant sites measured simultaneously during an incremental exercise test. The results demonstrated the inadequacy of rectal temperature as an indicator of the acute temperature changes occurring during an incremental exercise test due to its slow response rate and relative thermal inertia. Radial arterial blood and oesophageal temperatures were shown to behave almost identically during the exercise test, albeit with an offset of approximately 1.3ºC, and were considered much more appropriate and relevant indicators of thermal changes during exercise. As an extension of the initial work active muscle temperature (vastus lateralis) was measured during the exercise test, demonstrating a significantly lower resting temperature than the oft-reported “core” temperatures (rectal and oesophageal) as well as a significantly greater increase in temperature in comparison to all other measurement sites. Overall, the results of this first study indicated that the physiologically relevant temperatures measured at the oesophageal and muscle sites differed markedly to the outdated rectal temperature measurement site and should be used as measures of thermal response when evaluating oxygen loading (oesophageal) or unloading (active muscle). Utilising the definition of EIH as a decrease in PaO₂ of ≥ 10 mmHg, the effect of temperature correcting PaO₂ was evaluated in the second study. Arterial blood gases measured simultaneously to the temperature measurements during the incremental exercise test were adjusted for the temperature changes at each site (every 1ºC increase in temperature will increase a PaO₂ value by ~5 mmHg). Whilst uncorrected PaO₂ values indicated an almost 100% prevalence of EIH in this group, oesophageal temperature corrected PaO₂ values decreased this prevalence to ~50% while muscle temperature corrections resolved all cases of EIH and demonstrated an HYPEROXAEMIA (i.e. the reverse of the well-established phenomenon) in the majority of subjects. Further investigation of arterial oxygen content during the exercise test indicates that there is no disruption in the delivery of oxygen to the active muscles and therefore any performance decrement should be attributed to another mechanism. Whilst the phenomenon of EIH is determined by the definition applied and the use of temperature corrections in the case of PaO₂, its reproducibility in a test-retest situation had not previously been determined. Utilising a subset of previously tested subjects, the reproducibility of both temperature and PaO₂ were determined with results indicating that the blood gas response was highly reproducible, especially the minimum PaO₂ value noted during each exercise test. However, comparing a more statistically relevant definition of a change in PaO₂ of ± 2 standard deviations from the mean resting PaO₂ to the previous delimiter of 10 mmHg indicated a lesser reproducibility of the prevalence of EIH. In summary, this thesis exposes the inadequacies of previous research into EIH with regard to the expected reproducibility of the phenomenon and the need to correctly adjust PaO₂ values for exercise-induce hyperthermia as well as demonstrating the difference in thermal responses to acute exercise in physiologically significant areas of the body. Furthermore, previously described correlations between the change in PaO₂ and VO₂ max were not evident in the subjects tested within this thesis, nor was there any indication of a diffusion limitation based on reduced pulmonary capillary transit time (by association with VO₂ max) or pulmonary oedema (rebuked by a rapid return of PaO₂ to above resting levels following exercise cessation). / http://proxy.library.adelaide.edu.au/login?url= http://library.adelaide.edu.au/cgi-bin/Pwebrecon.cgi?BBID=1320633 / Thesis (Ph.D.) -- University of Adelaide, School of Medicine, 2008

This thesis investigates the use of field-programmable gate arrays (FPGAs) to implement a time-to-digital converter (TDC) with on-chip calibration and temperature correction.Using carry-chains on the Xilinx Kintex UltraScale architecture to create a tapped delay line (TDL) has previously been proven to give good time resolution.This project improves the resolution further by using a bit-counter to handle bubbles in the TDL without removing any taps.The bit counter also adds the possibility of using a wave-union approach previously dismissed as unusable on this architecture.The final implementation achieves an RMS resolution of 1.8 ps.