The growing connectivity of cyber-physical systems (CPSes) has led to an increased concern over the ability of cyber-attacks to inflict physical damage. Current cybersecurity measures focus on preventing attacks from penetrating control supervisory networks. These reactive techniques, however, are often plagued with vulnerabilities and zero-day exploits. Embedded processors in CPS field devices often possess little security of their own, and are easily exploited once the network is penetrated. In response, researchers at Georgia Tech and Virginia Tech have proposed a Trustworthy Autonomic Interface Guardian Architecture (TAIGA), which monitors communication between the embedded controller and physical process. This autonomic architecture provides the physical process with a last line of defense against cyber-attacks by switching process control to a trusted backup controller if an attack causes a system specification violation. This thesis focuses on classifying the effects of cyberattacks on embedded controllers, evaluating TAIGA’s resilience against these attacks, and determining the applicability of TAIGA to other CPSes. This thesis identifies four possible outcomes of a cyber-attack on a CPS embedded processor. We then evaluate TAIGA’s mechanisms to defend against those attack outcomes, and verify TAIGA satisfies the listed trust requirements. Next, we discuss an implementation and the experimental results of TAIGA on a hazardous cargo transportation robot. Then, by making various modifications to the setup configuration, we are able to explore TAIGA’s ability to provide security and process protection to other CPSes with varying levels of autonomy or distributed components.

A hypothetical causal link between ventilatory regulation of carbon dioxide anddevelopment of syncope during orthostatic challenges is reduction in arterial partialpressure of carbon dioxide and resultant reduction in cerebral blood flow. We performedtwo experiments to investigate the ventilatory sensitivity to carbon dioxide and factorsaffecting cerebral autoregulation (CA). We also studied the nonlinear phase couplingbetween cardio-respiratory parameters before syncope.For experiment one, in 30 healthy adults, we stimulated chemo and baro reflexesby breathing either room-air or room-air with 5 percent carbon dioxide in a pseudorandom binary sequence during supine and 70 degree head up tilt (HUT). Six subjectsdeveloped presyncope during tilt.To determine whether changes in ventilatory control contribute to the observeddecrease in PaCO2 during HUT, we assessed ventilatory dynamic sensitivity to changesin PaCO2 during supine and 70 degrees HUT. The sensitivity of the ventilatory controlsystem to perturbations in end tidal carbon dioxide increased during tilt.To investigate nonlinear phase coupling between cardio-respiratory parametersbefore syncope, bispectra were estimated and compared between presyncopal andnon-presyncopal subjects. Our results indicate that preceding presyncope, nonlinearphase coupling is altered by perturbations to baro and chemo reflexes.To investigate the effects of gender in CA, we selected 10 men and 10age-matched women and used spectral analysis to compare differences in CA betweenmen and women. Our results showed that gender-related differences in CA did exist andgender may need to be considered as a factor in investigating CA.To investigate the influence of induced hypocapnia on CA in absence ofventilatory variability, we performed experiment two in which subjects were randomlyassigned to a Control (under normocapnia) or Treatment (under hypocapnia) group. Bothgroups voluntarily controlled their breathing pattern yet two groups breathed in air withdifferent levels of carbon dioxide. Our results show that changes in mean blood pressureat middle cerebral artery level were less transferred into mean cerebral blood flow in theTreatment group than in the Control group, suggesting better CA under hypocapniarelative to under normocapnia.

Indivíduos diabéticos estão sujeitos a maior mortalidade cardiovascular relacionadas à disfunção autonômica e à neuropatia. Por outro lado o exercício físico é uma ferramenta importante no tratamento do diabetes, melhorando a função autonômica, melhorando a sensibilidade baroreflexa e quimioreflexa além de melhorar a freqüência cardíaca intrínseca. Os benefícios cardiovasculares e metabólicos do exercício físico em indivíduos diabéticos são bem conhecidos, mas uma questão permanece em aberto: Por quanto tempo esse benefícios são mantidos após a cessação do treinamento físico? O objetivo desse estudo foi avaliar o efeito de 3 semanas de destreinamento nas variáveis autonômicas, metabólicas e cardiovasculares após 10 semanas de treinamento em ratos diabéticos por STZ. Os experimentos foram realizados em ratos Wistar, machos, randomicamente divididos em 6 grupos: controle sedentário (CS), controle treinado (CT), controle treinado-destreinado (CD), diabético sedentário (DS), diabético treinado (DT) e diabético treinado-destreinado (DD). A indução do diabetes foi feita por uma injeção IV de STZ (50-60 mg/kg). O exercício físico de moderada intensidade (50 70% da velocidade máxima de corrida) foi realizado durante 10 semanas, seguidas de 3 semanas de destreinamento. Ao final do protocolo, artéria e veia femoral foram canuladas, pressão arterial (PA) e freqüência cardíaca (FC) foram medidas, de forma direta, gravadas e processadas por um sistema de aquisição de dados (Windaq, 2 kHz). A sensibilidade baroreflexa foi calculada pela razão da FC/PAM, depois da infusão de drogas vasoativas. A análise da potência espectral (FFT) foi calculada pela densidade media dos espectros. O diabetes reduziu a PA, a FC e a sensibilidade do barorreflexo (SBR), o que foi revertido pelo exercício físico e mantido após as 3 semanas de destreinamento. Nos animais controles, o exercício físico diminuiu a PA e a FC e melhorou a SBR, alterações estas que retornaram aos valores basais depois do destreinamento. A variância foi menor no DS quando comparada ao CS. O treinamento físico melhorou a variância no grupo DT enquanto que no grupo DD a variância foi similar a do grupo DS. Nenhuma alteração foi observada nos animais controle não diabéticos. No domínio da freqüência, o componente LF (%) e a razão entre LF/HF foi atenuada no grupo DT e no grupo DD, entretanto essa diferença não foi estatisticamente diferente do grupo DS. Nos animais controle, o exercício físico diminuiu a razão entre LF/HF (balance simpatovagal) e o componente LF enquanto que o componente HF esteve aumentado nos dois grupos: CT e DT. Embora a variância do IP tenha retornado aos valores basais após o destreinamento, nos animais diabéticos, observamos uma relação inversa entre o nível de glicose plasmatica e o componente HF (%) da variância da FC. O diabetes induziu redução da freqüência cardíaca intrínseca quando comparada a todos os grupos controle. O exercício físico reverteu essa alteração, e o efeito persistiu após o destreinamento. Nos animais diabéticos, o exercício diminuiu a glicemia e o destreinamento aumentou-a, mas mesmo assim o nível glicemico ainda ficou menor do que os observados nos animais diabéticos sedentários. Os ratos diabéticos treinados apresentaram 84% de sobrevivência durante as 10 semanas de treinamento enquanto que os diabéticos sedentários apresentaram 62%. Na 13 semana de protocolo, os diabéticos destreinados tiveram 80% de sobrevivência e os diabéticos sedentários 53% de sobrevivência. Esses achados indicam que o exercício físico não é apenas uma importante ferramenta no manejo das disfunções metabólicas e cardiovasculares do diabetes, mas mostra também que a algumas dessas melhoras persistem mesmo após 3 semanas de destreinamento, contribuindo para o aumento da taxa de sobrevida dos grupos treinados e destreinados quando comparados com os sedentários. / Diabetic subjects are prone to increased cardiovascular morbidity and mortality related to autonomic dysfunction and neuropathy. On the other hand, exercise is an important therapeutic tool in the treatment of diabetes improving autonomic function, increasing baro (BRS) and chemoreflex sensitivity and also intrinsic heart rate (IHR). Regarding the benefits of exercise training in cardiovascular and metabolic function in diabetic subjects, a question not yet answered is how long are these benefits kept. The aim of this study was to evaluate the effect of 3 weeks of detraining in the autonomic, metabolic and cardiovascular parameters after a previous period of 10 weeks of training in STZ-diabetic rats. Experiments were performed on male Wistar rats randomly assigned to 1 of 6 groups (n=8 each): sedentary controls (CS), trained controls (CT), traineddetrained controls (CD), sedentary diabetic (DS), trained diabetic (DT) and trained-detrained diabetic (DD). Diabetes was induced by STZ (50 mg/kg. ev). Moderate intensity exercise training (50-70% maximal running speed) was performed during 10 weeks, and 3 weeks for detraining. After femoral vessels catheterization, arterial pressure (AP) and heart rate (HR) were directly obtained, recorded and processed by a data acquisition system (Windaq, 2 kHz). BRS was calculated by the ratio HR/MAP, after infusion of vasoactive drugs. Spectral power (FFT) was calculated by means of power spectrum density integration. Diabetes promoted attenuation in AP, HR as well as in BRS, which was reversed by exercise training and maintained after 3 weeks of detraining. In controls, training decreased AP and HR and improved BRS, changes that returned to baseline values after the detraining. Variance was lower in DS versus CS. Exercise training improved variance in DT while in DD it was similar to DS. No changes were observed in non diabetic controls. In frequency domain the LF (%) component and the LF/HF ratio were attenuated in DT and DD groups, but not statistically different to DS. Exercise training in controls decreased the LF/HF ratio (sympathovagal balance) and LF component while HF component was increased in both CT and DT. Although whole variance of HR returned to basal levels after detraining in diabetic animals, there was an inverse relationship between plasma glucose levels and the HF (%) component of HRV. Diabetes induced IHR reduction in relation to all groups of control animals. Exercise training reversed that change, which was kept after detraining. In diabetes, exercise training lowered glycemia while detraining increased it when compared to trained diabetic, but the glucose levels were still lower than in sedentary diabetic. Diabetic trained rats presented 84% while diabetic sedentary 62% of survival rate in 10 weeks of training protocol. In the 13th week of protocol the diabetic detrained animals had 80% and the diabetic sedentary rats had a survival rate of 53%. These findings indicate that exercise training is not only an effective tool in the management of cardiovascular and metabolic diabetic derangements but also that these changes were kept working even after 3 weeks of detraining, contributing to the increase survival rate of the trained and detrained groups in comparison with sedentary ones

A fase da vida adulta entre 35 e 60 anos, também denominada de meia idade, compreende o período em que os principais sistemas biológicos apresentam importantes declínios funcionais. Nas mulheres, especificamente, é a fase marcada pelo climatério que tem como principal evento a ocorrência da menopausa. Esse evento fisiológico de importância hormonal e reprodutiva está associado em muitas mulheres ao expressivo aumento da prevalência de doenças cardiovasculares, muitas vezes associadas e precedidas por prejuízos na função autonômica cardiovascular. Nesse sentido, a avaliação da funcionalidade autonômica cardíaca é muito importante como conduta para estratificação de risco cardiovascular. De fato, a análise da variabilidade da frequência cardíaca (VFC) é muito utilizada, entretanto a metodologia segue um protocolo padrão que não leva em consideração situações fisiológicas importantes, como é o caso da reorganização da modulação autonômica cardíaca após o estresse induzido pelo exercício. Adicionalmente, a literatura tem optado por ferramentas lineares em detrimento das não lineares na avaliação da VFC. Nesse caso, a proposta do presente estudo foi avaliar e comparar a função autonômica cardíaca em mulheres jovens (GJ: 21 a 30 anos) e de meia idade pósmenopausa (GMI: 45 a 60 anos) por meio da análise linear (análise espectral) e não linear (análise simbólica) da variabilidade da frequência cardíaca em três momentos distintos (em repouso na posição supina, durante o tilt teste e durante o período de recuperação pós teste cardiopulmonar submáximo). O GMI apresentou menores valores de VO2pico (24 ± 1.0 vs 39 ± 1.3 ml.kg. min-1) frequência cardíaca basal (71 ± 2 vs 81 ± 2 bpm) e maiores valores da pressão arterial média (91 ± 2 vs 81 ± 1 mmHg) em relação ao GJ. Também apresentou maior modulação simpática e menor modulação vagal da FC na posição supina, entretanto somente evidenciado pela análise linear. Durante o tilt test as respostas do GMI foram menos proeminentes quando comparado com o GJ. Nesse caso, as avaliações linear e não linear apresentaram resultados semelhantes. Por fim, a análise da VFC durante o período de recuperação mostrou que o GMI apresentou recuperação da modulação autonômica vagal mais rápida evidenciada em ambas análises, linear e não linear. Em conclusão, a avaliação da modulação autonômica cardíaca mostrou que em repouso as mulheres jovens apresentam um predomínio do componente autonômico vagal, enquanto as mulheres de meia idade pós-menopausa apresentam um predomínio simpático. Por sua vez, o tilt test mostrou que a resposta autonômica das mulheres jovens é mais intensa, entretanto na reorganização após o exercício físico as mulheres de meia idade apresentaram maior velocidade no reestabelecimento da modulação vagal. As causas são incertas, porém podem ser decorrentes da redução dos hormônios ovarianos, bem como do processo de envelhecimento por estabelecimento de uma menor complexidade nos sistemas fisiológicos envolvidos. / The stage of adulthood between 35 and 60, also known as middle-aged, covers the period in which the main biological systems have important functional decline. In women, specifically, it is the stage marked by climacteric whose main event the occurrence of menopause. This physiological event of hormonal and reproductive importance is associated in many women to the significant increase in the prevalence of cardiovascular disease, often associated and preceded by losses in cardiovascular autonomic function. In this sense, the evaluation of cardiac autonomic functionality is very important as practice for cardiovascular risk stratification. In fact, the analysis of heart rate variability (HRV) is widely used, however the methodology follows a standard protocol that does not take into account important physiological situations, such as the reorganization of cardiac autonomic modulation after exercise-induced stress. Additionally, the literature has opted for linear tools instead of linear no evaluation of HRV. In this case, the purpose of this study was to evaluate and compare the cardiac autonomic function in young women (GJ: 21 to 30 years) and half postmenopausal age (GMI: 45-60 years) through the linear analysis (spectral analysis ) and non-linear (symbolic analysis) of heart rate variability at three different times (at rest in the supine position during the tilt test and during the recovery period after submaximal cardiopulmonary test). The GMI showed lower values of peak VO2 (24 ± 1.0 vs 39 ± 1.3 ml.kg. min-1) basal heart rate (71 ± 2 vs 81 ± 2 bpm) and higher mean arterial pressure (91 ± 2 vs 81 ± 1 mm Hg) compared to GJ. Also showed higher sympathetic modulation and lower vagal modulation of HF in the supine position, however only evidenced by linear analysis. During the tilt test responses GMI were less prominent compared to GJ. In this case, the linear and nonlinear tools showed similar results. Finally, the analysis of HRV after submaximal cardiopulmonary test showed that the GMI recovered faster autonomic modulation, shown in both analyzes, linear and non-linear. In conclusion, the evaluation of cardiac autonomic modulation showed that resting young women have a predominance of vagal autonomic component, while women half postmenopausal age present a sympathetic predominance. In turn, the tilt test showed that the autonomic response of young women is more intense, but the autonomic reorganization after exercise, the middle-aged women have faster the reestablishment of vagal modulation. The reasons are unknown, but may be due to the reduction in ovarian hormones, as well as the aging process by establishing less complex physiological mechanisms.

Communication network technologies have been evolving exponentially in the late decades. These innovations increase the network capabilities and open new horizons to creating novel and original services. The heterogeneity in equipment qualifications increases the level of complexity in the technological advancement. In such environment, service management has become an everyday challenge to service providers. Important efforts have been deployed to innovate in the exploitation of intelligent devices in the home and other private locations. In this Thesis, we propose a framework for self-configuration of devices within Hone Area Networks. We propose a device self-configuration architecture based on IBM Monitor-Analyze-Plan-Execute using Knowledge autonomic control loop. To prove the validity of our system architecture and support its applicability, we developed a prototype system that gives a general control loop implementation for device self-configuration using the CPE WAN Management Protocol. A video streaming scenario is implemented and used to evaluate validity our framework.

Communication network technologies have been evolving exponentially in the late decades. These innovations increase the network capabilities and open new horizons to creating novel and original services. The heterogeneity in equipment qualifications increases the level of complexity in the technological advancement. In such environment, service management has become an everyday challenge to service providers. Important efforts have been deployed to innovate in the exploitation of intelligent devices in the home and other private locations. In this Thesis, we propose a framework for self-configuration of devices within Hone Area Networks. We propose a device self-configuration architecture based on IBM Monitor-Analyze-Plan-Execute using Knowledge autonomic control loop. To prove the validity of our system architecture and support its applicability, we developed a prototype system that gives a general control loop implementation for device self-configuration using the CPE WAN Management Protocol. A video streaming scenario is implemented and used to evaluate validity our framework.

Communication network technologies have been evolving exponentially in the late decades. These innovations increase the network capabilities and open new horizons to creating novel and original services. The heterogeneity in equipment qualifications increases the level of complexity in the technological advancement. In such environment, service management has become an everyday challenge to service providers. Important efforts have been deployed to innovate in the exploitation of intelligent devices in the home and other private locations. In this Thesis, we propose a framework for self-configuration of devices within Hone Area Networks. We propose a device self-configuration architecture based on IBM Monitor-Analyze-Plan-Execute using Knowledge autonomic control loop. To prove the validity of our system architecture and support its applicability, we developed a prototype system that gives a general control loop implementation for device self-configuration using the CPE WAN Management Protocol. A video streaming scenario is implemented and used to evaluate validity our framework.

Indivíduos diabéticos estão sujeitos a maior mortalidade cardiovascular relacionadas à disfunção autonômica e à neuropatia. Por outro lado o exercício físico é uma ferramenta importante no tratamento do diabetes, melhorando a função autonômica, melhorando a sensibilidade baroreflexa e quimioreflexa além de melhorar a freqüência cardíaca intrínseca. Os benefícios cardiovasculares e metabólicos do exercício físico em indivíduos diabéticos são bem conhecidos, mas uma questão permanece em aberto: Por quanto tempo esse benefícios são mantidos após a cessação do treinamento físico? O objetivo desse estudo foi avaliar o efeito de 3 semanas de destreinamento nas variáveis autonômicas, metabólicas e cardiovasculares após 10 semanas de treinamento em ratos diabéticos por STZ. Os experimentos foram realizados em ratos Wistar, machos, randomicamente divididos em 6 grupos: controle sedentário (CS), controle treinado (CT), controle treinado-destreinado (CD), diabético sedentário (DS), diabético treinado (DT) e diabético treinado-destreinado (DD). A indução do diabetes foi feita por uma injeção IV de STZ (50-60 mg/kg). O exercício físico de moderada intensidade (50 70% da velocidade máxima de corrida) foi realizado durante 10 semanas, seguidas de 3 semanas de destreinamento. Ao final do protocolo, artéria e veia femoral foram canuladas, pressão arterial (PA) e freqüência cardíaca (FC) foram medidas, de forma direta, gravadas e processadas por um sistema de aquisição de dados (Windaq, 2 kHz). A sensibilidade baroreflexa foi calculada pela razão da FC/PAM, depois da infusão de drogas vasoativas. A análise da potência espectral (FFT) foi calculada pela densidade media dos espectros. O diabetes reduziu a PA, a FC e a sensibilidade do barorreflexo (SBR), o que foi revertido pelo exercício físico e mantido após as 3 semanas de destreinamento. Nos animais controles, o exercício físico diminuiu a PA e a FC e melhorou a SBR, alterações estas que retornaram aos valores basais depois do destreinamento. A variância foi menor no DS quando comparada ao CS. O treinamento físico melhorou a variância no grupo DT enquanto que no grupo DD a variância foi similar a do grupo DS. Nenhuma alteração foi observada nos animais controle não diabéticos. No domínio da freqüência, o componente LF (%) e a razão entre LF/HF foi atenuada no grupo DT e no grupo DD, entretanto essa diferença não foi estatisticamente diferente do grupo DS. Nos animais controle, o exercício físico diminuiu a razão entre LF/HF (balance simpatovagal) e o componente LF enquanto que o componente HF esteve aumentado nos dois grupos: CT e DT. Embora a variância do IP tenha retornado aos valores basais após o destreinamento, nos animais diabéticos, observamos uma relação inversa entre o nível de glicose plasmatica e o componente HF (%) da variância da FC. O diabetes induziu redução da freqüência cardíaca intrínseca quando comparada a todos os grupos controle. O exercício físico reverteu essa alteração, e o efeito persistiu após o destreinamento. Nos animais diabéticos, o exercício diminuiu a glicemia e o destreinamento aumentou-a, mas mesmo assim o nível glicemico ainda ficou menor do que os observados nos animais diabéticos sedentários. Os ratos diabéticos treinados apresentaram 84% de sobrevivência durante as 10 semanas de treinamento enquanto que os diabéticos sedentários apresentaram 62%. Na 13 semana de protocolo, os diabéticos destreinados tiveram 80% de sobrevivência e os diabéticos sedentários 53% de sobrevivência. Esses achados indicam que o exercício físico não é apenas uma importante ferramenta no manejo das disfunções metabólicas e cardiovasculares do diabetes, mas mostra também que a algumas dessas melhoras persistem mesmo após 3 semanas de destreinamento, contribuindo para o aumento da taxa de sobrevida dos grupos treinados e destreinados quando comparados com os sedentários. / Diabetic subjects are prone to increased cardiovascular morbidity and mortality related to autonomic dysfunction and neuropathy. On the other hand, exercise is an important therapeutic tool in the treatment of diabetes improving autonomic function, increasing baro (BRS) and chemoreflex sensitivity and also intrinsic heart rate (IHR). Regarding the benefits of exercise training in cardiovascular and metabolic function in diabetic subjects, a question not yet answered is how long are these benefits kept. The aim of this study was to evaluate the effect of 3 weeks of detraining in the autonomic, metabolic and cardiovascular parameters after a previous period of 10 weeks of training in STZ-diabetic rats. Experiments were performed on male Wistar rats randomly assigned to 1 of 6 groups (n=8 each): sedentary controls (CS), trained controls (CT), traineddetrained controls (CD), sedentary diabetic (DS), trained diabetic (DT) and trained-detrained diabetic (DD). Diabetes was induced by STZ (50 mg/kg. ev). Moderate intensity exercise training (50-70% maximal running speed) was performed during 10 weeks, and 3 weeks for detraining. After femoral vessels catheterization, arterial pressure (AP) and heart rate (HR) were directly obtained, recorded and processed by a data acquisition system (Windaq, 2 kHz). BRS was calculated by the ratio HR/MAP, after infusion of vasoactive drugs. Spectral power (FFT) was calculated by means of power spectrum density integration. Diabetes promoted attenuation in AP, HR as well as in BRS, which was reversed by exercise training and maintained after 3 weeks of detraining. In controls, training decreased AP and HR and improved BRS, changes that returned to baseline values after the detraining. Variance was lower in DS versus CS. Exercise training improved variance in DT while in DD it was similar to DS. No changes were observed in non diabetic controls. In frequency domain the LF (%) component and the LF/HF ratio were attenuated in DT and DD groups, but not statistically different to DS. Exercise training in controls decreased the LF/HF ratio (sympathovagal balance) and LF component while HF component was increased in both CT and DT. Although whole variance of HR returned to basal levels after detraining in diabetic animals, there was an inverse relationship between plasma glucose levels and the HF (%) component of HRV. Diabetes induced IHR reduction in relation to all groups of control animals. Exercise training reversed that change, which was kept after detraining. In diabetes, exercise training lowered glycemia while detraining increased it when compared to trained diabetic, but the glucose levels were still lower than in sedentary diabetic. Diabetic trained rats presented 84% while diabetic sedentary 62% of survival rate in 10 weeks of training protocol. In the 13th week of protocol the diabetic detrained animals had 80% and the diabetic sedentary rats had a survival rate of 53%. These findings indicate that exercise training is not only an effective tool in the management of cardiovascular and metabolic diabetic derangements but also that these changes were kept working even after 3 weeks of detraining, contributing to the increase survival rate of the trained and detrained groups in comparison with sedentary ones

Submitted by Renata Lopes (renatasil82@gmail.com) on 2016-07-14T20:24:58Z No. of bitstreams: 1 fabioantoniodamascenomarques.pdf: 1072493 bytes, checksum: 5c90ebd0061c6a81a5a657aeb312e3bf (MD5) / Approved for entry into archive by Diamantino Mayra (mayra.diamantino@ufjf.edu.br) on 2016-07-19T15:49:06Z (GMT) No. of bitstreams: 1 fabioantoniodamascenomarques.pdf: 1072493 bytes, checksum: 5c90ebd0061c6a81a5a657aeb312e3bf (MD5) / Made available in DSpace on 2016-07-19T15:49:06Z (GMT). No. of bitstreams: 1 fabioantoniodamascenomarques.pdf: 1072493 bytes, checksum: 5c90ebd0061c6a81a5a657aeb312e3bf (MD5) Previous issue date: 2016-06-15 / Desde o estudo de Robinson, publicado em 1938, em que se utilizou a idade como variável independente, não se conhece outra variável que possa melhorar a predição da freqüência cardíaca máxima (FCMAX). Recentemente, tem se estudado a FC de recuperação (FCREC) após exercício máximo, que tem sido apontada como preditora de mortalidade. Para melhor entender a FCMAX e a FCREC após teste máximo, o presente estudo teve como objetivos: 1)Identificar se a FCMax é influenciada pela variabilidade da freqüência cardíaca (VFC) em repouso, 2) Verificar se a FCREC avaliada por meio de deltas é influenciada pela FCMAX; 3) Sugerir um modelo de avaliação da FCREC relativizado pelos valores de FCMAX. e 4) Propor uma equação que descreva a cinética de recuperação da FCMAX em indivíduos jovens e de meia idade saudáveis. Para alcançar os objetivos propostos, foram realizados dois estudos. Estudo 1 - foram avaliados 63 indivíduos (21 mulheres e 42 homens) de 20 a 30 anos. Foi avaliada a FC e VFC em repouso na posição sentada. Em seguida, os indivíduos realizaram um teste cardiopulmonar máximo em esteira (Protocolo de Bruce). Os indivíduos foram divididos, sexo e pela mediana da FCMAX, em grupo de alta de baixa FCMAX. Os índices de VFC de repouso dos dois grupos foram comparados por teste ―t‖ de Student para grupos independentes (p<0,05). Apenas a banda de baixa freqüência (LF) nas mulheres se mostrou significativamente diferente entre os grupos. Conclui-se que maiores valores de LF nas mulheres em repouso estão associadas à FCMAX mais elevadas. Estudo 2 - foram avaliados 77 indivíduos (24 mulheres e 53 homens) de 18 a 50 anos. Após o teste máximo de Bruce, foi coletada a FC durante 300s após o fim do teste. Foi feita correlação da FCREC em valores absolutos, deltas e percentuais nos tempos 10, 20, 30, 40, 50, 60, 120, 180, 240 e 300 s com a FCMAX atingida no teste. O grupo também foi dividido em grupo e alta e baixa FCMAX pelo valor da mediana do grupo total. As diferenças entre as médias dos grupos foram testadas pelo teste ―t‖ de Student para grupos independentes (p<0,05). Observou-se que os valores absolutos de FCREC se correlacionam com a FCMAX, não sendo assim a melhor estratégia para classificação dessa recuperação. Os deltas de FCREC, na fase rápida (10 a 60 s), não mostraram correlação com a FCMAX. Já, na fase lenta (60 a 300 s), foi evidenciada correlação com a FCMAX. A utilização de valores percentuais da FCMAX se mostrou a única 9 estratégia em que em nenhum momento da FCREC apresentou correlação com a FCMAX. Construíram-se equações de regressão, uma para cada fase da FCREC, para indicar os valores médios de recuperação. / Since Robinson's study, published in 1938, which used age as an independent variable, do not know any other variable that can improve the prediction of maximal heart rate (HRMAX). Recently it has been studied HR recovery (HRR) after maximal exercise, which has been identified as a predictor of mortality. To better understand HRMAX and HRR after maximal test, this study aimed to: 1) Identify if the maximum heart rate is influenced by heart rate variability (HRV) at rest 2) Suggest an assessment model FCREC relativized by the values of HRMAX 3) Test the strategy of the calculation of deltas for the times of 10, 20, 30, 40, 50, 60, 120, 180, 240 and 300 s of recovery, and 4) Propose a strategy for calculating the FCREC by the percentage of recovery, as well as create an equation for predicting% FCREC. To achieve the proposed objectives, two studies were performed. Study 1 - 63 subjects were evaluated (21 women and 42 men) from 20 to 30 years. We evaluated the HR and your variability (HRV) at rest in a sitting position. Then, subjects performed a maximal cardiopulmonary exercise testing on treadmill (Bruce Protocol). The subjects were divided by gender and the median of HRMAX in group high low HRMAX. The indices of (HRV) at rest in both groups were compared by ―t‖ test of Student for independent groups (p <0.05). Only the low frequency band (LF) was significantly different between groups. We conclude that higher values of LF at rest are associated with higher HRMAX. Study 2 - 77 individuals were assessed (24 women and 53 men) from 18 to 50 years. After Bruce protocol, FC was collected for 300s after the test. Correlation of the FCREC in absolute values, deltas and percentages at 10, 20, 30, 40, 50, 60, 120, 180, 240 and 300 s with the HRMAX reached in the test. The group was also divided into groups and high and low HRMAX the median value of total group. The differences between group means were tested by ―t’ test for independent groups (p <0.05). It was observed that the absolute values of FCREC are correlated with HRMAX and thus not the best strategy for classification of that recovery. The deltas of FCREC, the fast phase (10-60 s) showed no correlation with the HRMAX. Already, in the slow phase (60 to 300 s) there was significant correlation with the HRMAX. The use of percentages of HRMAX proved to be the only strategy that at no time was correlated with the HRMAX. We constructed regression equations, one for each phase of the FCREC, to indicate the average values of recovery.

Communication network technologies have been evolving exponentially in the late decades. These innovations increase the network capabilities and open new horizons to creating novel and original services. The heterogeneity in equipment qualifications increases the level of complexity in the technological advancement. In such environment, service management has become an everyday challenge to service providers. Important efforts have been deployed to innovate in the exploitation of intelligent devices in the home and other private locations. In this Thesis, we propose a framework for self-configuration of devices within Hone Area Networks. We propose a device self-configuration architecture based on IBM Monitor-Analyze-Plan-Execute using Knowledge autonomic control loop. To prove the validity of our system architecture and support its applicability, we developed a prototype system that gives a general control loop implementation for device self-configuration using the CPE WAN Management Protocol. A video streaming scenario is implemented and used to evaluate validity our framework.