The purpose of this study was to determine which MT techniques current licensed massage therapists practicing sports massage are using to treat delayed onset muscle soreness (DOMS) by conducting an anonymous online survey of practicing sports massage therapists. The secondary purpose was to determine if previous sports massage research protocols match current practices in sports massage to treat DOMS. There were 85 anonymous respondents ranging in age from 24 to 74 years with a mean age of 44.5 years. Participants included 55 female (65.5%) and 29 male (34.5%) therapists. Professional experience ranged from 4 years or less to more than 20 years, and the majority reported having training specific to sports massage. Previous research methods have placed an overwhelming emphasis on effleurage and petrissage in the treatment protocols to test the effects of massage on DOMS. However, practicing therapists rely on many more techniques and modalities when giving sports massage treatments for DOMS. On a scale of 1 to 5 respondents rated the importance of effleurage strokes in their sports massage treatment at 3.3, petrissage strokes averaged 3.4, friction 3.3, and tapotement ranked slightly lower with a weighted average of 2.1. Sixty six percent of respondents reported that they use stretching techniques often or always. Myofascial release techniques are used sometimes or often for 65% of the respondents, but fewer utilize manual lymphatic drainage in their sports massage. Additional techniques besides those mentioned in the survey are used by 64% of the respondents.

No description available.

No description available.

Syfte: Målsättningen med studien är att hos friska kvinnor utvärdera om intensiv tuggning leder till en proprioceptiv allodyni i massetermuskulaturen. Följande hypoteser testades i studien: 30 minuters intensivt tuggande leder till (1) ökad trötthet och smärta, (2) sänkt smärttröskel, (3) ökad vibrotaktil känslighet (4) samt träningsvärk i massetermuskulaturen bilateralt. Material och Metod: 16 friska kvinnor (medelålder 25.8 ± 5.0 år) deltog i studien som bestod av två 60-minuters sessioner med 24-timmars uppföljning. Vid ena sessionen tuggade försökspersonerna på ett grekiskt tuggummi (Masticha Chios) i 5-minuters intervaller under 30 minuter, och vilade vid den andra sessionen. En vibrameter användes för att mäta vibrationströskel, upplevd vibrationsintensitet och upplevd vibrationsobehag. Upplevd trötthet och upplevd smärtintensitet värderades enligt en visuell analog skala 0-100 mm (VAS skala). Smärttröskeln mättes med en algometer som applicerades på massetermuskelns fäste. Två vägs variansanalys användes för att testa utfallsvariabler för upprepade mätningar och Dunnett’s posthoc-test. Resultat: En signifikant skillnad kunde ses mellan tuggning och icke tuggning för vibrationströskeln (P < 0.001); upplevda tröttheten (P < 0.001); smärttröskeln (P < 0.01); upplevda vibrationsobehaget (P < 0.05). Inga signifikanta skillnader observerades för upplevd smärta och upplevd vibrationsobehag. Däremot kunde en signifikant ökning ses över tid för vibrationströskeln och den upplevda tröttheten. Konklusion: Våra fynd indikerar att det ej finns något samband mellan intensiv tuggning och träningsvärk i massetermuskulaturen. Däremot resulterade tuggningen i ökad muskeltrötthet och vibrationsobehag, samt signifikant högre trösklar för smärta och vibration under försökets gång. Proprioceptiv allodyni kunde ej påvisas. / Aims: To evaluate if intense chewing leads to proprioceptive allodynia in the masseter muscle, and test if (1) pain and fatigue are increased after intense chewing, (2) pressure pain thresholds are decreased after intense chewing, (3) vibration thresholds are increased after intense chewing, (5) intense vibrations exacerbate pain after intense chewing. Materials and Methods: 16 healthy female volunteers (mean age 25.8 ± 5 years) participated in two 60-minute sessions, each with 24-hour follow-ups. In the first session, the subjects were instructed to chew on a viscous chewing gum (Masticha Chios). In the other session the participant were assigned a control task where no chewing was performed. Perceived intensity of vibration and perceived discomfort were assessed with a Vibrameter® on a 0-50-100 visual analog scale (VAS). Two 0-100 mm visual analog scales measured pain intensity and perceived fatigue. The pain threshold was measured with an electronic algometer. These variables were measured to assess delayed onset muscle soreness. All measurements were made on the masseter muscles. Two-way analysis of variance for repeated measures and Dunnett’s post hoc test was used to test for significant alterations in the outcome variables. Results: Significant differences were observed between chewing and no-activity for vibration threshold (P<0.001); perceived fatigue (P<0.001); pain threshold (P<0.01) and vibration discomfort (P<0.05). No significant differences were seen for perceived pain and perceived intensity of vibration. A significant increase over time could only be observed for vibration threshold (P<0.01) and perceived fatigue (P<0.05). Conclusion: Our findings suggest that there is no relation between intense chewing and delayed onset muscle soreness in the masseter muscles. Intense chewing evoked moderate levels of fatigue and discomfort, and a hypoalgesia to mechanical stimulation, and reduced vibrotactile sensitivity.

Este estudo compara quatro diferentes intensidades com o volume total similar no exercício supino. Avaliou-se a dor muscular de início tardio (DMIT), atividade de creatina quinase (CK), as concentrações sangüíneas de interleucina (IL)-1<font face=\"symbol\">b, IL-6, fator de necrose tumoral-<font face=\"symbol\">a (TNF-<font face=\"symbol\">a), prostaglandina E2 (PGE2) e o perfil hormonal. A amostra foi composta de soldados do exército brasileiro, divididos em cinco grupos: 50%-1RM, 75%-1RM, 90%-1RM, 110%-1RM e o controle. A DMIT e a atividade plasmática de CK aumentaram significativamente (P<0,05) após a sessão de exercício. A concentração de PGE2 também teve aumento significativo (P<0,05) após a sessão (P<0,05). A concentração plasmática de cortisol após 1h do término do exercício aumentou apenas no grupo 75%-1RM (p < 0,05). Esses resultados sugerem que a intensidade no exercício supino não afeta a magnitude da DMIT, marcadores de lesão muscular, inflamação e na resposta hormonal geral, desde que haja a equalização do volume total, com exceção da concentração plasmática do cortisol, grupo 75%-1RM. / This study compared four different intensities with similar total volume of a bench press exercise for muscle soreness, creatine kinase (CK) activity, interleukin (IL)-1<font face=\"symbol\">b, IL-6, tumor necrosis factor-<font face=\"symbol\">a (TNF-<font face=\"symbol\">a), prostaglandin E2 (PGE2) and hormonal concentrations in the blood. Brazilian Army male soldiers were placed into five groups: 50%-1RM, 75%-1RM, 90%-1RM, and 110%-1RM, and control that did not perform the exercise. Muscle soreness and plasma CK activity increased significantly (p<0.05) after exercise. Serum PGE2 concentration also increased significantly (p<0.05) after exercise. After one hour post exercise cortisol increased in 75%-1RM group, with this response also exceeding the other intensities (p<0.05). These results suggest that the intensity of bench press exercise does not affect the magnitude of muscle soreness and blood markers of muscle damage, inflammation and largely similar hormonal responses, which may be attributed to the equalization of total volume, exception made for the 75%-1RM group for serum cortisol concentration.

Este estudo compara quatro diferentes intensidades com o volume total similar no exercício supino. Avaliou-se a dor muscular de início tardio (DMIT), atividade de creatina quinase (CK), as concentrações sangüíneas de interleucina (IL)-1<font face=\"symbol\">b, IL-6, fator de necrose tumoral-<font face=\"symbol\">a (TNF-<font face=\"symbol\">a), prostaglandina E2 (PGE2) e o perfil hormonal. A amostra foi composta de soldados do exército brasileiro, divididos em cinco grupos: 50%-1RM, 75%-1RM, 90%-1RM, 110%-1RM e o controle. A DMIT e a atividade plasmática de CK aumentaram significativamente (P<0,05) após a sessão de exercício. A concentração de PGE2 também teve aumento significativo (P<0,05) após a sessão (P<0,05). A concentração plasmática de cortisol após 1h do término do exercício aumentou apenas no grupo 75%-1RM (p < 0,05). Esses resultados sugerem que a intensidade no exercício supino não afeta a magnitude da DMIT, marcadores de lesão muscular, inflamação e na resposta hormonal geral, desde que haja a equalização do volume total, com exceção da concentração plasmática do cortisol, grupo 75%-1RM. / This study compared four different intensities with similar total volume of a bench press exercise for muscle soreness, creatine kinase (CK) activity, interleukin (IL)-1<font face=\"symbol\">b, IL-6, tumor necrosis factor-<font face=\"symbol\">a (TNF-<font face=\"symbol\">a), prostaglandin E2 (PGE2) and hormonal concentrations in the blood. Brazilian Army male soldiers were placed into five groups: 50%-1RM, 75%-1RM, 90%-1RM, and 110%-1RM, and control that did not perform the exercise. Muscle soreness and plasma CK activity increased significantly (p<0.05) after exercise. Serum PGE2 concentration also increased significantly (p<0.05) after exercise. After one hour post exercise cortisol increased in 75%-1RM group, with this response also exceeding the other intensities (p<0.05). These results suggest that the intensity of bench press exercise does not affect the magnitude of muscle soreness and blood markers of muscle damage, inflammation and largely similar hormonal responses, which may be attributed to the equalization of total volume, exception made for the 75%-1RM group for serum cortisol concentration.

In this project, the effects of dietary polyphenols on exercise-induced muscle damage and vascular health are examined. Dietary polyphenols exert well-known anti-inflammatory effects; however, how these effects are realized with respect to vascular health and EIMD is relatively unknown. I begin by reviewing the available literature surrounding the impact of three dietary polyphenols (curcumin, catechins, and quercetin) on inflammation associated with EIMD. It is well established that their primary means of anti-inflammation is through alterations of NF-κB and AP-1 transcription activities. Given this, their inclusion into training strategies seems reasonable. Consistent evidence is presented making a case for the anti-inflammatory effects of dietary polyphenols following EIMD. I follow this review up by completing an in-depth study on the consumption of curcumin prior to EIMD. I found curcumin (1000 mg/day) can reduce subjective soreness and decrease inflammation compared to placebo controls. To further understand the effects of dietary polyphenols on health, I investigate the effects of a four-week supplementation period of cocoa (catechins) on vascular. I concluded that atherogenic risk in obese women is reduced after consumption of cocoa. In addition to these experimental projects, I developed two novel methods that can be used to investigate vascular health (EMP concentration) and intracellular protein and mRNA production using flow cytometry.

No description available.

Träningsvärk efter fysisk aktivitet är ett välkänt fenomen. Vad som ligger bakomträningsvärkens effekter såsom smärta och nedsatt rörlighet är mindre välkänt. Träningsvärk, eller ”delayed onset muscle soreness” (DOMS), har i forskningenantagits bero på muskelskada och inflammation i skelettmusklerna efter att ovana ellerexcentriska övningar utförts. Försöken att lindra träningsvärkens effekter har varitmånga, t ex genom antiinflammatoriska läkemedel (NSAID). Syftet med dennasystematiska litteraturstudie var att svara på frågeställningen hur ibuprofen påverkarträningsvärk där hypotesen att ibuprofen skulle ha en dämpande effekt på träningsvärkantogs. Nio artiklar granskades för att besvara hypotesen utan att några slutsatser omibuprofens inverkan på träningsvärk kunde dras då resultaten pekade åt olika håll ochinga tendenser kunde observeras. Den slutsats som kunde dras utifrån litteraturstudienvar att ytterligare forskning på området är behövlig. Om studier inte kan visa att NSAIDdämpar träningsvärk så kan det ifrågasättas om inflammation är orsaken tillträningsvärken. Ytterligare studier på området är viktigt då NSAID är vanligtförekommande som smärtlindrande läkemedel samtidigt som det har biverkningar. Det finns också forskning som tyder på att NSAID kan ha negativ inverkan på de positivaeffekter som är av intresse i träningssammanhang.

Bien qu’il soit recommandé de pratiquer tout au long de sa vie une activité physique régulière pour se maintenir en bonne santé, peu d’études portent sur les modifications des patrons locomoteurs lors de la répétition de séances à 2 ou 3 jours d’intervalle. Lorsqu’elles sont intenses ou inhabituelles, les formes naturelles de locomotion pédestre – dites de type cycle étirement-détente (CED) – se caractérisent par des perturbations structuro-fonctionnelles et proprioceptives qui peuvent perdurer plusieurs jours. Ces déficiences sont autant de sources de risques accrus de blessures lors de la répétition d’une pratique sportive. Ce travail doctoral a pour objectif (i) d’examiner les effets immédiats et retardés d’exercices épuisants de type CED sur les paramètres neuromécaniques de la locomotion humaine en conditions de marche, de course et de sauts et (ii) d’étudier l’influence combinée d’une pratique pieds nus vs. pieds chaussés. Nos résultats ne révèlent pas de modification significative des patrons locomoteurs en phase de récupération immédiate (post-exercice) mais démontrent l’intervention de stratégies compensatrices et/ou protectrices en phase de récupération retardée (2ème jour posteffort). Ces stratégies diffèrent entre les conditions de marche et de course. Nos travaux soulignent également l’importance de discriminer les stratégies adoptées dès la première minute de l’exercice des ajustements ultérieurs apparaissant pendant la phase dite d’optimisation du CED. Par contre, les différences observées entre les conditions de course pieds nus vs. pieds chaussés restent étonnamment similaires avec la fatigue. Le port de chaussures se traduit par une réduction des chocs d’impact mais également par une restriction des mouvements naturels du pied et par une éversion accrue dont il conviendrait d’étudier les conséquences lors de la répétition en état de fatigue d’exercices de plus longue durée. / Although lifelong practice of moderate amounts of regular physical activity is recommended to ensure a healthy living state, little is know about the modifications of the locomotion patterns when repeating exercises every 2 to 3 days. Intense or unaccustomed stretch-shortening cycle (SSC) forms of ground locomotion are characterized, however, by structuro-functional and proprioceptive impairments that may last for a few days. These impairments may then be expected to increase the risk of injury. The present work aimed (i) to examine the acute and delayed SSC fatigue effects on the neuro-mechanical gait characteristics in walking, running and jumping conditions and (ii) to study the combined influence of a barefoot vs. shod gait condition. Our results did not reveal any modification of the locomotion patterns in the acute recovery phase (post-exercise), but demonstrated compensatory and/or protective strategies in the delayed phase (2 days post-exercise). The observed strategies differed significantly in walking and running. Our results emphasized also the need to differentiate the strategies occurring within the first minute of exercise from the subsequent adjustments related to the progressive SSC pattern optimization. Surprisingly, the observed differences between the barefoot and shod running conditions remained quite independent of the fatigue state. The shod running condition was thus found to attenuate ground impact transmission, but it resulted also in restricted natural foot motions and increased eversion that should be worth re-examining when repeating exercises of longer duration.